Somatic hypermutation at A/T-rich oligonucleotide substrates shows different strand polarities in Ung-deficient or -proficient backgrounds.
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Proximity to AGCT sequences dictates MMR-independent versus MMR-dependent mechanisms for AID-induced mutation via UNG2.Refining the Neuberger model: Uracil processing by activated B cells.Overlapping hotspots in CDRs are critical sites for V region diversificationNon-canonical actions of mismatch repairThe dual nature of mismatch repair as antimutator and mutator: for better or for worse.Pms2 and uracil-DNA glycosylases act jointly in the mismatch repair pathway to generate Ig gene mutations at A-T base pairs.SAMHD1 enhances immunoglobulin hypermutation by promoting transversion mutation.
P2860
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P2860
Somatic hypermutation at A/T-rich oligonucleotide substrates shows different strand polarities in Ung-deficient or -proficient backgrounds.
description
2014 nî lūn-bûn
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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի ապրիլին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
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name
Somatic hypermutation at A/T-r ...... nt or -proficient backgrounds.
@ast
Somatic hypermutation at A/T-r ...... nt or -proficient backgrounds.
@en
type
label
Somatic hypermutation at A/T-r ...... nt or -proficient backgrounds.
@ast
Somatic hypermutation at A/T-r ...... nt or -proficient backgrounds.
@en
prefLabel
Somatic hypermutation at A/T-r ...... nt or -proficient backgrounds.
@ast
Somatic hypermutation at A/T-r ...... nt or -proficient backgrounds.
@en
P2093
P2860
P356
P1476
Somatic hypermutation at A/T-r ...... ent or -proficient backgrounds
@en
P2093
Alexandre Alcais
Claude-Agnès Reynaud
Frédéric Delbos
Giulia Girelli Zubani
Jean-Claude Weill
Marija Zivojnovic
Sébastien Storck
P2860
P304
P356
10.1128/MCB.01452-13
P407
P577
2014-04-07T00:00:00Z