Clearance of Borrelia burgdorferi may not be required for resistance to experimental lyme arthritis.
about
Relative contributions of innate and acquired host responses to bacterial control and arthritis development in Lyme diseaseActivation of natural killer cells in arthritis-susceptible but not arthritis-resistant mouse strains following Borrelia burgdorferi infection.Experimental lyme arthritis in the absence of interleukin-4 or gamma interferon.Genetic control of experimental lyme arthritis in the absence of specific immunityModulation of cytokine release in ex vivo-stimulated blood from borreliosis patients.The chemokine receptor CXCR2 ligand KC (CXCL1) mediates neutrophil recruitment and is critical for development of experimental Lyme arthritis and carditis.Infection of Interleukin 17 Receptor A-Deficient C3H Mice with Borrelia burgdorferi Does Not Affect Their Development of Lyme Arthritis and Carditis.Antibiotic treatment of Lyme borreliosis: what is the evidence?Gene expression profiling provides insights into the pathways involved in inflammatory arthritis development: murine model of Lyme disease.Adenoviral delivery of interleukin-10 fails to attenuate experimental Lyme diseaseInterval-specific congenic lines reveal quantitative trait Loci with penetrant lyme arthritis phenotypes on chromosomes 5, 11, and 12.T Cells Exacerbate Lyme Borreliosis in TLR2-Deficient Mice.Treatment of mice with the neutrophil-depleting antibody RB6-8C5 results in early development of experimental lyme arthritis via the recruitment of Gr-1- polymorphonuclear leukocyte-like cells.The role of eicosanoids in experimental Lyme arthritis.Noncoding RNAs and chronic inflammation: Micro-managing the fire within.Localized production of IL-10 suppresses early inflammatory cell infiltration and subsequent development of IFN-γ-mediated Lyme arthritisThe nucleotide excision repair system of Borrelia burgdorferi is the sole pathway involved in repair of DNA damage by UV light.IL-10 Deficiency Reveals a Role for TLR2-Dependent Bystander Activation of T Cells in Lyme Arthritis.
P2860
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P2860
Clearance of Borrelia burgdorferi may not be required for resistance to experimental lyme arthritis.
description
1998 nî lūn-bûn
@nan
1998 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Clearance of Borrelia burgdorf ...... o experimental lyme arthritis.
@ast
Clearance of Borrelia burgdorf ...... o experimental lyme arthritis.
@en
type
label
Clearance of Borrelia burgdorf ...... o experimental lyme arthritis.
@ast
Clearance of Borrelia burgdorf ...... o experimental lyme arthritis.
@en
prefLabel
Clearance of Borrelia burgdorf ...... o experimental lyme arthritis.
@ast
Clearance of Borrelia burgdorf ...... o experimental lyme arthritis.
@en
P2860
P1476
Clearance of Borrelia burgdorf ...... o experimental lyme arthritis.
@en
P2093
P2860
P304
P407
P577
1998-05-01T00:00:00Z