T helper phenotype and genetic susceptibility in experimental Lyme disease
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Evidence of Borrelia Autoimmunity-Induced Component of Lyme Carditis and ArthritisBabesiosisDominant recognition of a Borrelia burgdorferi outer surface protein A peptide by T helper cells in patients with treatment-resistant Lyme arthritisHuman Coinfection with Borrelia burgdorferi and Babesia microti in the United StatesBorrelia burgdorferi gene expression in vivo and spirochete pathogenicity.Cerebrospinal fluid-infiltrating CD4+ T cells recognize Borrelia burgdorferi lysine-enriched protein domains and central nervous system autoantigens in early lyme encephalitisRelative contributions of innate and acquired host responses to bacterial control and arthritis development in Lyme diseaseInterleukin-4 (IL-4) and IL-13 signaling pathways do not regulate Borrelia burgdorferi-induced arthritis in mice: IgG1 is not required for host control of tissue spirochetes.Distinct characteristics of resistance to Borrelia burgdorferi-induced arthritis in C57BL/6N mice.Modulation of murine Lyme borreliosis by interruption of the B7/CD28 T-cell costimulatory pathway.Clearance of Borrelia burgdorferi may not be required for resistance to experimental lyme arthritis.Activation of natural killer cells in arthritis-susceptible but not arthritis-resistant mouse strains following Borrelia burgdorferi infection.Recombinant vaccinia virus coexpressing the F protein of respiratory syncytial virus (RSV) and interleukin-4 (IL-4) does not inhibit the development of RSV-specific memory cytotoxic T lymphocytes, whereas priming is diminished in the presence of higBorrelia burgdorferi spirochetes induce mast cell activation and cytokine release.Experimental lyme arthritis in the absence of interleukin-4 or gamma interferon.Genetic control of experimental lyme arthritis in the absence of specific immunityEarly induction of gamma interferon and interleukin-10 production in draining lymph nodes from mice infected with Borrelia burgdorferi.Coinfection with Borrelia burgdorferi and the agent of human granulocytic ehrlichiosis alters murine immune responses, pathogen burden, and severity of Lyme arthritis.Gamma interferon is not required for arthritis resistance in the murine Lyme disease modelCD4+ T helper 1 cells facilitate regression of murine Lyme carditisA critical role for type I IFN in arthritis development following Borrelia burgdorferi infection of mice.Cyclooxygenase 2 activity modulates the severity of murine Lyme arthritis.Relationship between immunity to Borrelia burgdorferi outer-surface protein A (OspA) and Lyme arthritisMurine Lyme arthritis development mediated by p38 mitogen-activated protein kinase activity.Borrelia burgdorferi-induced inflammation facilitates spirochete adaptation and variable major protein-like sequence locus recombinationControl of Borrelia burgdorferi-specific CD4+-T-cell effector function by interleukin-12- and T-cell receptor-induced p38 mitogen-activated protein kinase activityInvolvement of CD4+ T lymphocytes in induction of severe destructive Lyme arthritis in inbred LSH hamstersMacrophages exposed to Borrelia burgdorferi induce Lyme arthritis in hamstersDecreased interleukin-4 and increased gamma interferon production by peripheral blood mononuclear cells of patients with Lyme borreliosis.Macrophages and enriched populations of T lymphocytes interact synergistically for the induction of severe, destructive Lyme arthritis.B7-1 and B7-2 monoclonal antibodies modulate the severity of murine Lyme arthritis.T-helper-cell cytokines in the early evolution of murine Lyme arthritisAblation of interleukin-12 exacerbates Lyme arthritis in SCID mice.CD8+ T cells are activated during the early Th1 and Th2 immune responses in a murine Lyme disease model.Phagocytosis of Borrelia burgdorferi and Treponema pallidum potentiates innate immune activation and induces gamma interferon productionAnti-DR5 mAb inhibits proliferation of human fibroblast-like synovial cells and reduces their cytokine secretion in vitro.Adenoviral delivery of interleukin-10 fails to attenuate experimental Lyme diseaseLyme arthritis: current concepts and a change in paradigm.NKT cells prevent chronic joint inflammation after infection with Borrelia burgdorferi.Borreliosis: recent research, diagnosis, and management.
P2860
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P2860
T helper phenotype and genetic susceptibility in experimental Lyme disease
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh
1995年學術文章
@zh-hant
name
T helper phenotype and genetic susceptibility in experimental Lyme disease
@ast
T helper phenotype and genetic susceptibility in experimental Lyme disease
@en
type
label
T helper phenotype and genetic susceptibility in experimental Lyme disease
@ast
T helper phenotype and genetic susceptibility in experimental Lyme disease
@en
prefLabel
T helper phenotype and genetic susceptibility in experimental Lyme disease
@ast
T helper phenotype and genetic susceptibility in experimental Lyme disease
@en
P2860
P356
P1476
T helper phenotype and genetic susceptibility in experimental Lyme disease
@en
P2093
J E Matyniak
S L Reiner
P2860
P304
P356
10.1084/JEM.181.3.1251
P407
P577
1995-03-01T00:00:00Z