Aged BALB/c mice as a model for increased severity of severe acute respiratory syndrome in elderly humans
about
Design of wide-spectrum inhibitors targeting coronavirus main proteasesTime course and cellular localization of SARS-CoV nucleoprotein and RNA in lungs from fatal cases of SARS.Animal models for SARS and MERS coronavirusesMolecular pathology of emerging coronavirus infectionsLack of innate interferon responses during SARS coronavirus infection in a vaccination and reinfection ferret modelEarly Upregulation of Acute Respiratory Distress Syndrome-Associated Cytokines Promotes Lethal Disease in an Aged-Mouse Model of Severe Acute Respiratory Syndrome Coronavirus InfectionA new mouse-adapted strain of SARS-CoV as a lethal model for evaluating antiviral agents in vitro and in vivoA crucial role of angiotensin converting enzyme 2 (ACE2) in SARS coronavirus-induced lung injurySevere acute respiratory syndrome coronavirus as an agent of emerging and reemerging infectionVaccine efficacy in senescent mice challenged with recombinant SARS-CoV bearing epidemic and zoonotic spike variantsA decade after SARS: strategies for controlling emerging coronavirusesThe role of epidermal growth factor receptor (EGFR) signaling in SARS coronavirus-induced pulmonary fibrosis.Nonhuman primate models for SARS.A mouse-adapted SARS-coronavirus causes disease and mortality in BALB/c mice.Toll-like receptors, chemokine receptors and death receptor ligands responses in SARS coronavirus infected human monocyte derived dendritic cells.SARS-CoV pathogenesis is regulated by a STAT1 dependent but a type I, II and III interferon receptor independent mechanism.Differential downregulation of ACE2 by the spike proteins of severe acute respiratory syndrome coronavirus and human coronavirus NL63.Cellular immune responses to severe acute respiratory syndrome coronavirus (SARS-CoV) infection in senescent BALB/c mice: CD4+ T cells are important in control of SARS-CoV infection.Coronaviruses post-SARS: update on replication and pathogenesis.Allelic Variation in the Toll-Like Receptor Adaptor Protein Ticam2 Contributes to SARS-Coronavirus Pathogenesis in Mice.Lung fibroblasts accelerate wound closure in human alveolar epithelial cells through hepatocyte growth factor/c-Met signaling.Mechanisms of severe acute respiratory syndrome pathogenesis and innate immunomodulation.Coronavirus pathogenesis and the emerging pathogen severe acute respiratory syndrome coronavirus.Severe acute respiratory syndrome coronavirus infection of human ciliated airway epithelia: role of ciliated cells in viral spread in the conducting airways of the lungs.A new model for Hendra virus encephalitis in the mouse.Coronaviruses: an overview of their replication and pathogenesis.An immunosuppressed Syrian golden hamster model for SARS-CoV infection.Emerging respiratory viruses: challenges and vaccine strategiesEpithelial cells lining salivary gland ducts are early target cells of severe acute respiratory syndrome coronavirus infection in the upper respiratory tracts of rhesus macaques.A double-inactivated severe acute respiratory syndrome coronavirus vaccine provides incomplete protection in mice and induces increased eosinophilic proinflammatory pulmonary response upon challengeSevere acute respiratory syndrome coronavirus infection of mice transgenic for the human Angiotensin-converting enzyme 2 virus receptor.Participation of both host and virus factors in induction of severe acute respiratory syndrome (SARS) in F344 rats infected with SARS coronavirus.Molecular determinants of severe acute respiratory syndrome coronavirus pathogenesis and virulence in young and aged mouse models of human disease.New Metrics for Evaluating Viral Respiratory Pathogenesis.Utility of the aged BALB/c mouse model to demonstrate prevention and control strategies for severe acute respiratory syndrome coronavirus (SARS-CoV)Review of bats and SARSSynthetic reconstruction of zoonotic and early human severe acute respiratory syndrome coronavirus isolates that produce fatal disease in aged mice.Rat coronaviruses infect rat alveolar type I epithelial cells and induce expression of CXC chemokines.Mechanisms of zoonotic severe acute respiratory syndrome coronavirus host range expansion in human airway epithelium.Structural basis for potent cross-neutralizing human monoclonal antibody protection against lethal human and zoonotic severe acute respiratory syndrome coronavirus challenge
P2860
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P2860
Aged BALB/c mice as a model for increased severity of severe acute respiratory syndrome in elderly humans
description
2005 nî lūn-bûn
@nan
2005 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Aged BALB/c mice as a model fo ...... ory syndrome in elderly humans
@ast
Aged BALB/c mice as a model fo ...... ory syndrome in elderly humans
@en
type
label
Aged BALB/c mice as a model fo ...... ory syndrome in elderly humans
@ast
Aged BALB/c mice as a model fo ...... ory syndrome in elderly humans
@en
prefLabel
Aged BALB/c mice as a model fo ...... ory syndrome in elderly humans
@ast
Aged BALB/c mice as a model fo ...... ory syndrome in elderly humans
@en
P2860
P50
P1433
P1476
Aged BALB/c mice as a model fo ...... ory syndrome in elderly humans
@en
P2093
Emily Butler
Leatrice Vogel
P2860
P304
P356
10.1128/JVI.79.9.5833-5838.2005
P577
2005-05-01T00:00:00Z