Redox-mediated reciprocal regulation of SERCA and Na+-Ca2+ exchanger contributes to sarcoplasmic reticulum Ca2+ depletion in cardiac myocytes. - Wikidata - wikimedia - TriplyDB

Redox-mediated reciprocal regulation of SERCA and Na+-Ca2+ exchanger contributes to sarcoplasmic reticulum Ca2+ depletion in cardiac myocytes.

Redox-mediated reciprocal regulation of SERCA and Na+-Ca2+ exchanger contributes to sarcoplasmic reticulum Ca2+ depletion in cardiac myocytes.

http://www.wikidata.org/entity/Q33761932

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The Measurement of Reversible Redox Dependent Post-translational Modifications and Their Regulation of Mitochondrial and Skeletal Muscle Function Ca(2+) signaling in the myocardium by (redox) regulation of PKA/CaMKII Abnormal Ca(2+) cycling in failing ventricular myocytes: role of NOS1-mediated nitroso-redox balance Obligatory role of neuronal nitric oxide synthase in the heart's antioxidant adaptation with exercise Ranolazine improves cardiac diastolic dysfunction through modulation of myofilament calcium sensitivity Redox regulation of store-operated Ca2+ entry Role of Ca2+ in the control of H2O2-modulated phosphorylation pathways leading to eNOS activation in cardiac myocytes Preclinical left ventricular diastolic dysfunction in metabolic syndrome.The Na+/Ca2+ exchanger-1 mediates left ventricular dysfunction in mice with chronic intermittent hypoxia.Diesterified nitrone rescues nitroso-redox levels and increases myocyte contraction via increased SR Ca(2+) handling Redox signaling in cardiac myocytes.Dysregulation of intracellular calcium transporters in animal models of sepsis-induced cardiomyopathy ß-blocker timolol prevents arrhythmogenic Ca²⁺ release and normalizes Ca²⁺ and Zn²⁺ dyshomeostasis in hyperglycemic rat heart.Diastolic dysfunction in the diabetic continuum: association with insulin resistance, metabolic syndrome and type 2 diabetes.FXYD proteins reverse inhibition of the Na+-K+ pump mediated by glutathionylation of its beta1 subunit.Growth hormone secretagogues protect mouse cardiomyocytes from in vitro ischemia/reperfusion injury through regulation of intracellular calcium.Cardiac mitochondrial network excitability: insights from computational analysis.Stromal interaction molecule 1 (STIM1) is involved in the regulation of mitochondrial shape and bioenergetics and plays a role in oxidative stress.The plasma membrane channel ORAI1 mediates detrimental calcium influx caused by endogenous oxidative stress.SERCA Cys674 sulphonylation and inhibition of L-type Ca2+ influx contribute to cardiac dysfunction in endotoxemic mice, independent of cGMP synthesis.ATP-binding cassette B10 regulates early steps of heme synthesis.Hydrogen peroxide-mediated SERCA cysteine 674 oxidation contributes to impaired cardiac myocyte relaxation in senescent mouse heart Physiological regulation of cardiac contractility by endogenous reactive oxygen species.Exercise tames the wild side of the Myc network: a hypothesis.Redox control of cardiac excitability.Posttranslational modifications of cardiac ryanodine receptors: Ca(2+) signaling and EC-coupling.Tri-modal regulation of cardiac muscle relaxation; intracellular calcium decline, thin filament deactivation, and cross-bridge cycling kinetics.Oxidative stress in cardiomyocytes contributes to decreased SERCA2a activity in rats with metabolic syndrome.Crosstalk between calcium and reactive oxygen species signaling in cancer.Pathophysiological Fundamentals of Diabetic Cardiomyopathy.The role of Ca2+ in cell death caused by oxidative glutamate toxicity and ferroptosis.Hydrogen peroxide activates store-operated Ca(2+) entry in coronary arteries.Obesity-related cardiomyopathy is an adipocyte-mediated paracrine disease Mechanisms of the beneficial actions of ischemic preconditioning on subcellular remodeling in ischemic-reperfused heart Mitochondrial Ca2+ transport in the endothelium: regulation by ions, redox signalling and mechanical forces.Toll-like receptor 4 induced ryanodine receptor 2 oxidation and sarcoplasmic reticulum Ca2+ leakage promote cardiac contractile dysfunction in sepsis.Impact of Labile Zinc on Heart Function: From Physiology to Pathophysiology.Mitochondrial Bioenergetics During Ischemia and Reperfusion.Cigarette smoke elicits relaxation of renal arteries.Slow calcium waves and redox changes precede mitochondrial permeability transition pore opening in the intact heart during hypoxia and reoxygenation

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P2860

Redox-mediated reciprocal regulation of SERCA and Na+-Ca2+ exchanger contributes to sarcoplasmic reticulum Ca2+ depletion in cardiac myocytes.

http://www.wikidata.org/entity/Q33761932

description

2010 nî lūn-bûn

@nan

2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

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name

Redox-mediated reciprocal regu ...... depletion in cardiac myocytes.

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Redox-mediated reciprocal regu ...... depletion in cardiac myocytes.

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type

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Redox-mediated reciprocal regu ...... depletion in cardiac myocytes.

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Redox-mediated reciprocal regu ...... depletion in cardiac myocytes.

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prefLabel

Redox-mediated reciprocal regu ...... depletion in cardiac myocytes.

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Redox-mediated reciprocal regu ...... depletion in cardiac myocytes.

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J.FREERADBIOMED.2010.01.038

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Free Radical Biology and Medicine

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Redox-mediated reciprocal regu ...... depletion in cardiac myocytes.

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Catherine Communal

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Chee C Lim

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Deborah A Siwik

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Gabriela M Kuster

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Jingmei Zhang

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Mario P Trucillo

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Otmar Pfister

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Richard A Cohen

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Ronglih Liao

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P2860

Fractional SR Ca release is regulated by trigger Ca and SR Ca content in cardiac myocytes

Strain-stimulated hypertrophy in cardiac myocytes is mediated by reactive oxygen species-dependent Ras S-glutathiolation.

Dysregulated sarcoplasmic reticulum calcium release: potential pharmacological target in cardiac disease.

Role of oxidative stress in myocardial hypertrophy and failure.

Calcium cycling in congestive heart failure.

Direction-independent block of bi-directional Na+/Ca2+ exchange current by KB-R7943 in guinea-pig cardiac myocytes.

Is depressed myocyte contractility centrally involved in heart failure?

Enhanced ryanodine receptor-mediated calcium leak determines reduced sarcoplasmic reticulum calcium content in chronic canine heart failure.

Cardiac myocytes Ca2+ and Na+ regulation in normal and failing hearts.

Hydrogen peroxide as an endogenous mediator and exogenous tool in cardiovascular research: issues and considerations.

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9

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48

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Ellen O Weinberg

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2010-02-01T00:00:00Z

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41407734

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20132882

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