Is depressed myocyte contractility centrally involved in heart failure?
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Abnormal Ca(2+) cycling in failing ventricular myocytes: role of NOS1-mediated nitroso-redox balanceS100A1 in human heart failure: lack of recovery following left ventricular assist device supportTargeting S100A1 in heart failureAltered intracellular Ca2+ handling in heart failure.Role of RyR2 phosphorylation in heart failure and arrhythmias: protein kinase A-mediated hyperphosphorylation of the ryanodine receptor at serine 2808 does not alter cardiac contractility or cause heart failure and arrhythmiasRedox-mediated reciprocal regulation of SERCA and Na+-Ca2+ exchanger contributes to sarcoplasmic reticulum Ca2+ depletion in cardiac myocytes.Systems approach to understanding electromechanical activity in the human heart: a national heart, lung, and blood institute workshop summary.Abnormal intrastore calcium signaling in chronic heart failure.Investigating the role of uncoupling of troponin I phosphorylation from changes in myofibrillar Ca(2+)-sensitivity in the pathogenesis of cardiomyopathy.{beta}1-Adrenergic receptor activation induces mouse cardiac myocyte death through both L-type calcium channel-dependent and -independent pathways.Transient receptor potential channels contribute to pathological structural and functional remodeling after myocardial infarctionElevated cytosolic Na+ increases mitochondrial formation of reactive oxygen species in failing cardiac myocytes.Expression of a transgene encoding mutant p193/CUL7 preserves cardiac function and limits infarct expansion after myocardial infarctionNitroxyl improves cellular heart function by directly enhancing cardiac sarcoplasmic reticulum Ca2+ cycling.Nitroxyl enhances myocyte Ca2+ transients by exclusively targeting SR Ca2+-cycling.Compromised myocardial energetics in hypertrophied mouse hearts diminish the beneficial effect of overexpressing SERCA2a.The relationship between arrhythmogenesis and impaired contractility in heart failure: role of altered ryanodine receptor function.Positive inotropic effects of low dATP/ATP ratios on mechanics and kinetics of porcine cardiac muscleHistologically measured cardiomyocyte hypertrophy correlates with body height as strongly as with body mass indexTreadmill exercise training prevents myocardial mechanical dysfunction induced by androgenic-anabolic steroid treatment in ratsMolecular determinants of altered contractility in heart failure.Comparative effects of urocortins and stresscopin on cardiac myocyte contractilityCardiac arrhythmia mechanisms in rats with heart failure induced by pulmonary hypertension.Enhanced ryanodine receptor-mediated calcium leak determines reduced sarcoplasmic reticulum calcium content in chronic canine heart failure.Ultrastructural remodelling of Ca(2+) signalling apparatus in failing heart cells.Two subpopulations of mitochondria in the aging rat heart display heterogenous levels of oxidative stress.Mir-24 regulates junctophilin-2 expression in cardiomyocytes.In vivo suppression of microRNA-24 prevents the transition toward decompensated hypertrophy in aortic-constricted miceSarcolipin and phospholamban as regulators of cardiac sarcoplasmic reticulum Ca2+ ATPase.Mesenchymal stem cell transplantation improves regional cardiac remodeling following ovine infarctionExcitation-contraction coupling and mitochondrial energetics.Cellular basis of burn-induced cardiac dysfunction and prevention by mesenteric lymph duct ligation.Increased intracellular [dATP] enhances cardiac contraction in embryonic chick cardiomyocytes.Troponin phosphorylation and myofilament Ca2+-sensitivity in heart failure: increased or decreased?Delivery of nano-objects to functional sub-domains of healthy and failing cardiac myocytes.Analysis of Skeletal Muscle Torque Capacity and Circulating Ceramides in Patients with Advanced Heart Failure.HNO enhances SERCA2a activity and cardiomyocyte function by promoting redox-dependent phospholamban oligomerizationMyocardial protective effect of tezosentan, an endothelin receptor antagonist, for ischemia-reperfusion injury in experimental heart failure models.Inositol 1, 4, 5-trisphosphate receptors and human left ventricular myocytesOxysterols in heart failure.
P2860
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P2860
Is depressed myocyte contractility centrally involved in heart failure?
description
2003 nî lūn-bûn
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2003 թուականի Մարտին հրատարակուած գիտական յօդուած
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2003 թվականի մարտին հրատարակված գիտական հոդված
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2003年の論文
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2003年論文
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2003年論文
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2003年論文
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2003年論文
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2003年論文
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2003年论文
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name
Is depressed myocyte contractility centrally involved in heart failure?
@ast
Is depressed myocyte contractility centrally involved in heart failure?
@en
type
label
Is depressed myocyte contractility centrally involved in heart failure?
@ast
Is depressed myocyte contractility centrally involved in heart failure?
@en
prefLabel
Is depressed myocyte contractility centrally involved in heart failure?
@ast
Is depressed myocyte contractility centrally involved in heart failure?
@en
P1433
P1476
Is depressed myocyte contractility centrally involved in heart failure?
@en
P2093
Kenneth B Margulies
Steven R Houser
P304
P356
10.1161/01.RES.0000060027.40275.A6
P577
2003-03-01T00:00:00Z