Regulation of COX-2 expression and IL-6 release by particulate matter in airway epithelial cells - Wikidata - wikimedia - TriplyDB

Regulation of COX-2 expression and IL-6 release by particulate matter in airway epithelial cells

Regulation of COX-2 expression and IL-6 release by particulate matter in airway epithelial cells

http://www.wikidata.org/entity/Q33765074

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Multi-walled carbon nanotubes induce COX-2 and iNOS expression via MAP kinase-dependent and -independent mechanisms in mouse RAW264.7 macrophages Epigenetic Regulation in Particulate Matter-Mediated Cardiopulmonary Toxicities: A Systems Biology Perspective Particulate matter disrupts human lung endothelial barrier integrity via ROS- and p38 MAPK-dependent pathways Inflammation-related effects of diesel engine exhaust particles: studies on lung cells in vitro Temporal-spatial analysis of U.S.-Mexico border environmental fine and coarse PM air sample extract activity in human bronchial epithelial cells Cyclooxygenase-2 and the inflammogenesis of breast cancer.Septin-2 mediates airway epithelial barrier function in physiologic and pathologic conditions.Inhibition of aldose reductase prevents experimental allergic airway inflammation in mice.Urban air pollutants reduce synaptic function of CA1 neurons via an NMDA/NȮ pathway in vitro Diesel exhaust particles induce CYP1A1 and pro-inflammatory responses via differential pathways in human bronchial epithelial cells.Lysophosphatidic acid receptor 1 modulates lipopolysaccharide-induced inflammation in alveolar epithelial cells and murine lungs.Exposure to diesel exhaust upregulates COX-2 expression in ApoE knockout mice.Hydrogen sulfide attenuates particulate matter-induced human lung endothelial barrier disruption via combined reactive oxygen species scavenging and Akt activation Particulate matter air pollution disrupts endothelial cell barrier via calpain-mediated tight junction protein degradation.Lysophosphatidic acid (LPA) and its receptors: role in airway inflammation and remodeling.Proapoptotic Noxa is required for particulate matter-induced cell death and lung inflammation.Lysophosphatidic acid enhances pulmonary epithelial barrier integrity and protects endotoxin-induced epithelial barrier disruption and lung injury.Epithelial cells and airway diseases.Nonylphenol regulates cyclooxygenase-2 expression via Ros-activated NF-κB pathway in sertoli TM4 cells.Mechanisms and treatments for severe, steroid-resistant allergic airway disease and asthma.Dung biomass smoke activates inflammatory signaling pathways in human small airway epithelial cells.Emissions from commercial-grade charbroiling meat operations induce oxidative stress and inflammatory responses in human bronchial epithelial cells Multiple protein kinase pathways mediate amplified IL-6 release by human lung fibroblasts co-exposed to nickel and TLR-2 agonist, MALP-2.Fibroblastic reticular cells of the lymphoid tissues modulate T cell activation threshold during homeostasis via hyperactive cyclooxygenase-2/prostaglandin E2 axis.The effect of curcumin on human bronchial epithelial cells exposed to fine particulate matter: a predictive analysis.Airborne particulate matter increases MUC5AC expression by downregulating Claudin-1 expression in human airway cells.Urban particulate matter triggers lung inflammation via the ROS-MAPK-NF-κB signaling pathway.CCAAT/enhancer binding protein β regulates prostaglandin E synthase expression and prostaglandin E2 production in activated microglial cells.Assessment of genetic diversity in IL-6 and RANTES promoters and their level in Saudi coronary artery disease patients.US EPA particulate matter research centers: summary of research results for 2005–2011 Health Effects Associated with Inhalation of Airborne Arsenic Arising from Mining Operations Monosodium urate crystals reduce osteocyte viability and indirectly promote a shift in osteocyte function towards a proinflammatory and proresorptive state

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P2860

Regulation of COX-2 expression and IL-6 release by particulate matter in airway epithelial cells

http://www.wikidata.org/entity/Q33765074

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2008 nî lūn-bûn

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2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2008 թվականի հուլիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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Regulation of COX-2 expression ...... ter in airway epithelial cells

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Regulation of COX-2 expression ...... ter in airway epithelial cells

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American Journal of Respiratory Cell and Molecular Biology

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Regulation of COX-2 expression ...... ter in airway epithelial cells

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Alison S Geyh

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Donghong He

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Ernst Wm Spannhake

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Irina A Gorshkova

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Joe G N Garcia

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Jonathan M Samet

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Liliana Moreno-Vinasco

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Patrick N Breysse

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Peter V Usatyuk

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Ting Wang

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P2860

Association of fine particulate matter from different sources with daily mortality in six U.S. cities

Seasonal variations in air pollution particle-induced inflammatory mediator release and oxidative stress.

Quinones and aromatic chemical compounds in particulate matter induce mitochondrial dysfunction: implications for ultrafine particle toxicity

Expression of genes for B7-H3 and other T cell ligands by nasal epithelial cells during differentiation and activation

Nickel alterations of TLR2-dependent chemokine profiles in lung fibroblasts are mediated by COX-2

Transcription factors NF-IL6 and NF-kappa B synergistically activate transcription of the inflammatory cytokines, interleukin 6 and interleukin 8

Fine particulate air pollution and mortality in 20 U.S. cities, 1987-1994

Effects of environmental pollutants on airways, allergic inflammation, and the immune response.

Epidemiological evidence for inflammation in cardiovascular disease.

Air pollution in asthma: effect of pollutants on airway inflammation.

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19-30

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1

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40

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