Comprehensive evaluation of the role of EZH2 in the growth, invasion, and aggression of a panel of prostate cancer cell lines
about
The Polycomb complex PRC2 and its mark in lifeHistone H2A.Z deregulation in prostate cancer. Cause or effect?Epigenetic therapy with 3-deazaneplanocin A, an inhibitor of the histone methyltransferase EZH2, inhibits growth of non-small cell lung cancer cells.Genomic deregulation of the E2F/Rb pathway leads to activation of the oncogene EZH2 in small cell lung cancer.Chromatin Memory in the Development of Human CancersMethylator phenotype of malignant germ cell tumours in children identifies strong candidates for chemotherapy resistance.Down-regulation of the histone methyltransferase EZH2 contributes to the epigenetic programming of decidualizing human endometrial stromal cells.Myc enforces overexpression of EZH2 in early prostatic neoplasia via transcriptional and post-transcriptional mechanismsEpigenetic regulation of prostate cancerMethylome-wide Sequencing Detects DNA Hypermethylation Distinguishing Indolent from Aggressive Prostate CancerTargeting EZH2 regulates tumor growth and apoptosis through modulating mitochondria dependent cell-death pathway in HNSCC.Inhibition of EZH2 by chemo- and radiotherapy agents and small molecule inhibitors induces cell death in castration-resistant prostate cancerA rapid ultra HPLC-MS/MS method for the quantitation and pharmacokinetic analysis of 3-deazaneplanocin A in mice.Overexpression of EZH2 is associated with the poor prognosis in osteosarcoma and function analysis indicates a therapeutic potential.The role of microRNAs in the regulation of apoptosis in lung cancer and its application in cancer treatmentSite-Specific Fucosylation Analysis Identifying Glycoproteins Associated with Aggressive Prostate Cancer Cell Lines Using Tandem Affinity Enrichments of Intact Glycopeptides Followed by Mass SpectrometryLncRNA-ANCR down-regulation suppresses invasion and migration of colorectal cancer cells by regulating EZH2 expression.MiR-26a and miR-138 block the G1/S transition by targeting the cell cycle regulating network in prostate cancer cells.Divergent clonal evolution of castration-resistant neuroendocrine prostate cancer.EZH2 dependent H3K27me3 is involved in epigenetic silencing of ID4 in prostate cancer.Future prospects in the diagnosis and management of localized prostate cancer.
P2860
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P2860
Comprehensive evaluation of the role of EZH2 in the growth, invasion, and aggression of a panel of prostate cancer cell lines
description
2010 nî lūn-bûn
@nan
2010 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Comprehensive evaluation of th ...... of prostate cancer cell lines
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Comprehensive evaluation of th ...... of prostate cancer cell lines
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type
label
Comprehensive evaluation of th ...... of prostate cancer cell lines
@ast
Comprehensive evaluation of th ...... of prostate cancer cell lines
@en
prefLabel
Comprehensive evaluation of th ...... of prostate cancer cell lines
@ast
Comprehensive evaluation of th ...... of prostate cancer cell lines
@en
P2093
P2860
P356
P1433
P1476
Comprehensive evaluation of th ...... of prostate cancer cell lines
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P2093
Breanne D W Karanikolas
Marxa L Figueiredo
P2860
P304
P356
10.1002/PROS.21112
P577
2010-05-01T00:00:00Z