Gamma interferon production by cytotoxic T lymphocytes is required for resolution of Chlamydia trachomatis infection.
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The role of intracellular organisms in the pathogenesis of inflammatory arthritisCD8+ T cells recognize an inclusion membrane-associated protein from the vacuolar pathogen Chlamydia trachomatisA MyD88-dependent early IL-17 production protects mice against airway infection with the obligate intracellular pathogen Chlamydia muridarum.Steroids alone or as adjunctive therapy with doxycycline fail to improve oviduct damage in mice infected with Chlamydia muridarum.Acquired immunity to Chlamydia pneumoniae is dependent on gamma interferon in two mouse strains that initially differ in this respect after primary challenge.Immunity to murine Chlamydia trachomatis genital tract reinfection involves B cells and CD4(+) T cells but not CD8(+) T cells.Immunity to murine chlamydial genital infection.Role of interferon-stimulated gene factor 3gamma and beta interferon in HLA class I enhancement in synovial fibroblasts upon infection with Chlamydia trachomatisThe frequency of Chlamydia trachomatis major outer membrane protein-specific CD8+ T lymphocytes in active trachoma is associated with current ocular infection.The major CD8 T cell effector memory subset in the normal and Chlamydia trachomatis-infected human endocervix is low in perforinReactive arthritis or chronic infectious arthritis?The hypothetical protein CT813 is localized in the Chlamydia trachomatis inclusion membrane and is immunogenic in women urogenitally infected with C. trachomatis.A chlamydial type III-secreted effector protein (Tarp) is predominantly recognized by antibodies from humans infected with Chlamydia trachomatis and induces protective immunity against upper genital tract pathologies in mice.The duration of Chlamydia muridarum genital tract infection and associated chronic pathological changes are reduced in IL-17 knockout mice but protection is not increased further by immunization.Protective immunity against mouse upper genital tract pathology correlates with high IFNγ but low IL-17 T cell and anti-secretion protein antibody responses induced by replicating chlamydial organisms in the airway.In Vivo and Ex Vivo Imaging Reveals a Long-Lasting Chlamydial Infection in the Mouse Gastrointestinal Tract following Genital Tract InoculationThe recall response induced by genital challenge with Chlamydia muridarum protects the oviduct from pathology but not from reinfectionCaspase-1 contributes to Chlamydia trachomatis-induced upper urogenital tract inflammatory pathologies without affecting the course of infection.Chlamydia trachomatis-specific human CD8+ T cells show two patterns of antigen recognition.Role of CD8(+)T cells in the host response to ChlamydiaEndogenous processing and presentation of T-cell epitopes from Chlamydia trachomatis with relevance in HLA-B27-associated reactive arthritis.T cell responses in the absence of IFN-gamma exacerbate uterine infection with Chlamydia trachomatis.Salmonella enterica serovar typhimurium exploits Toll-like receptor signaling during the host-pathogen interaction.The infecting dose of Chlamydia muridarum modulates the innate immune response and ascending infection.Chlamydia trachomatis-infected epithelial cells and fibroblasts retain the ability to express surface-presented major histocompatibility complex class I molecules.Sensing the enemy, containing the threat: cell-autonomous immunity to Chlamydia trachomatis.Temporal cytokine gene expression patterns in subjects with trachoma identify distinct conjunctival responses associated with infection.Immunological memory in B-cell-deficient mice conveys long-lasting protection against genital tract infection with Chlamydia trachomatis by rapid recruitment of T cells.Failure to detect HLA-A*6802-restricted CD8+ T cells specific for Chlamydia trachomatis antigens in subjects from trachoma-endemic communities.Depletion of CD8+ cells abolishes memory in acquired immunity against Chlamydia pneumoniae in BALB/c mice.A flow cytometry-based assay to determine the phagocytic activity of both clinical and nonclinical antibody samples against Chlamydia trachomatis.Chlamydia trachomatis-infected cells and uninfected-bystander cells exhibit diametrically opposed responses to interferon gamma.
P2860
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P2860
Gamma interferon production by cytotoxic T lymphocytes is required for resolution of Chlamydia trachomatis infection.
description
1998 nî lūn-bûn
@nan
1998 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Gamma interferon production by ...... lamydia trachomatis infection.
@ast
Gamma interferon production by ...... lamydia trachomatis infection.
@en
type
label
Gamma interferon production by ...... lamydia trachomatis infection.
@ast
Gamma interferon production by ...... lamydia trachomatis infection.
@en
prefLabel
Gamma interferon production by ...... lamydia trachomatis infection.
@ast
Gamma interferon production by ...... lamydia trachomatis infection.
@en
P2093
P2860
P1476
Gamma interferon production by ...... hlamydia trachomatis infection
@en
P2093
C B Wilson
M N Starnbach
P2860
P304
P407
P577
1998-11-01T00:00:00Z