Nicotinic acid activates the capsaicin receptor TRPV1: Potential mechanism for cutaneous flushing. - Wikidata - wikimedia - TriplyDB

Nicotinic acid activates the capsaicin receptor TRPV1: Potential mechanism for cutaneous flushing.

Nicotinic acid activates the capsaicin receptor TRPV1: Potential mechanism for cutaneous flushing.

http://www.wikidata.org/entity/Q33778078

about

Nicotinamide is an endogenous agonist for a C. elegans TRPV OSM-9 and OCR-4 channel.Nicotinic acid is a common regulator of heat-sensing TRPV1-4 ion channels Nicotinic acid inhibits glioma invasion by facilitating Snail1 degradation Niacin as antidyslipidemic drug.Definition of a tolerable upper intake level of niacin: a systematic review and meta-analysis of the dose-dependent effects of nicotinamide and nicotinic acid supplementation.Measuring niacin-associated skin toxicity (NASTy) stigmata along with symptoms to aid development of niacin mimetics.Nicotinic acid impairs assembly of leading edge in glioma cells.TRPV1 channels are involved in niacin-induced cutaneous vasodilation in mice.Niacin and olive oil promote skewing to the M2 phenotype in bone marrow-derived macrophages of mice with metabolic syndrome.

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Nicotinic acid activates the capsaicin receptor TRPV1: Potential mechanism for cutaneous flushing.

http://www.wikidata.org/entity/Q33778078

description

2014 nî lūn-bûn

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2014 թուականի Մարտին հրատարակուած գիտական յօդուած

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2014 թվականի մարտին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Nicotinic acid activates the c ...... hanism for cutaneous flushing.

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Nicotinic acid activates the c ...... hanism for cutaneous flushing.

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Nicotinic acid activates the c ...... hanism for cutaneous flushing.

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Nicotinic acid activates the c ...... hanism for cutaneous flushing.

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Arteriosclerosis, Thrombosis, and Vascular Biology

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Nicotinic acid activates the c ...... chanism for cutaneous flushing

@en

P2093

Anping Cai

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Bo Hyun Lee

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Heather Clifton

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Jie Zheng

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Rongrong Mao

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Saul Schaefer

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Yunfang Jia

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P2860

The capsaicin receptor: a heat-activated ion channel in the pain pathway

GPR109A (PUMA-G/HM74A) mediates nicotinic acid-induced flushing

Nicotinic acid- and monomethyl fumarate-induced flushing involves GPR109A expressed by keratinocytes and COX-2-dependent prostanoid formation in mice

beta-Arrestin1 mediates nicotinic acid-induced flushing, but not its antilipolytic effect, in mice

TRPV1 structures in distinct conformations reveal activation mechanisms

Molecular identification of high and low affinity receptors for nicotinic acid

PUMA-G and HM74 are receptors for nicotinic acid and mediate its anti-lipolytic effect

Aspirin reduces cutaneous flushing after administration of an optimized extended-release niacin formulation

Suppression of niacin-induced vasodilation with an antagonist to prostaglandin D2 receptor subtype 1

Nicotinic acid: pharmacological effects and mechanisms of action

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1272-1280

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scholarly article

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