Mucosal immunity to herpes simplex virus type 2 infection in the mouse vagina is impaired by in vivo depletion of T lymphocytes.
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Immunoglobulin G, plasma cells, and lymphocytes in the murine vagina after vaginal or parenteral immunization with attenuated herpes simplex virus type 2Neutrophils aid in protection of the vaginal mucosae of immune mice against challenge with herpes simplex virus type 2.Nasal immunization of mice with human papillomavirus type 16 (HPV-16) virus-like particles or with the HPV-16 L1 gene elicits specific cytotoxic T lymphocytes in vaginal draining lymph nodesHerpes simplex virus and the chemokines that mediate the inflammation.Interleukin-12 (IL-12) and IL-18 are important in innate defense against genital herpes simplex virus type 2 infection in mice but are not required for the development of acquired gamma interferon-mediated protective immunity.RNA-dependent protein kinase is required for alpha-1 interferon transgene-induced resistance to genital herpes simplex virus type 2.Recent progress in herpes simplex virus immunobiology and vaccine researchAnalysis of vaginal cell populations during experimental vaginal candidiasis.Mechanism of reduced T-cell effector functions and class-switched antibody responses to herpes simplex virus type 2 in the absence of B7 costimulationLoss of the type I interferon pathway increases vulnerability of mice to genital herpes simplex virus 2 infection.A mucosal vaccination approach for herpes simplex virus type 2.Vaginal memory T cells induced by intranasal vaccination are critical for protective T cell recruitment and prevention of genital HSV-2 disease.Immunization with herpes simplex virus 2 (HSV-2) genes plus inactivated HSV-2 is highly protective against acute and recurrent HSV-2 diseaseHSV-2 regulates monocyte inflammatory response via the Fas/FasL pathwayLocal delivery of CpG oligodeoxynucleotides induces rapid changes in the genital mucosa and inhibits replication, but not entry, of herpes simplex virus type 2.The Role of Plasmacytoid Dendritic Cells in Innate and Adaptive Immune Responses against Alpha Herpes Virus InfectionsSusceptibility of CCR5-deficient mice to genital herpes simplex virus type 2 is linked to NK cell mobilizationAbrogation of attenuated lentivirus-induced protection in rhesus macaques by administration of depo-provera before intravaginal challenge with simian immunodeficiency virus mac239Glycoprotein G of herpes simplex virus 2 as a novel vaccine antigen for immunity to genital and neurological disease.Comparison of the host immune response to herpes simplex virus 1 (HSV-1) and HSV-2 at two different mucosal sites.CXCL9 and CXCL10 expression are critical for control of genital herpes simplex virus type 2 infection through mobilization of HSV-specific CTL and NK cells to the nervous systemReplication-defective virus vaccine-induced protection of mice from genital herpes simplex virus 2 requires CD4 T cellsEffector CD4+ T-cell involvement in clearance of infectious herpes simplex virus type 1 from sensory ganglia and spinal cordsChemokines and Chemokine Receptors Critical to Host Resistance following Genital Herpes Simplex Virus Type 2 (HSV-2) InfectionDendritic cells and B cells maximize mucosal Th1 memory response to herpes simplex virusOverexpression of interleukin-15 compromises CD4-dependent adaptive immune responses against herpes simplex virus 2Codelivery of CCR7 ligands as molecular adjuvants enhances the protective immune response against herpes simplex virus type 1.Mucosal innate and adaptive immune responses against herpes simplex virus type 2 in a humanized mouse model.Intraepithelial gammadelta T cells may bridge a gap between innate immunity and acquired immunity to herpes simplex virus type 2B7 costimulation plays an important role in protection from herpes simplex virus type 2-mediated pathology.Immunity to vaginal herpes simplex virus-2 infection in B-cell knockout mice.Interferon-gamma up-regulates intercellular adhesion molecule-1 and vascular cell adhesion molecule-1 and recruits lymphocytes into the vagina of immune mice challenged with herpes simplex virus-2.Immune responses and protection against vaginal infection after nasal or vaginal immunization with attenuated herpes simplex virus type-2.
P2860
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P2860
Mucosal immunity to herpes simplex virus type 2 infection in the mouse vagina is impaired by in vivo depletion of T lymphocytes.
description
1998 nî lūn-bûn
@nan
1998 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Mucosal immunity to herpes sim ...... vo depletion of T lymphocytes.
@ast
Mucosal immunity to herpes sim ...... vo depletion of T lymphocytes.
@en
type
label
Mucosal immunity to herpes sim ...... vo depletion of T lymphocytes.
@ast
Mucosal immunity to herpes sim ...... vo depletion of T lymphocytes.
@en
prefLabel
Mucosal immunity to herpes sim ...... vo depletion of T lymphocytes.
@ast
Mucosal immunity to herpes sim ...... vo depletion of T lymphocytes.
@en
P2860
P1433
P1476
Mucosal immunity to herpes sim ...... ivo depletion of T lymphocytes
@en
P2093
P2860
P304
P577
1998-04-01T00:00:00Z