T lymphocytes mediate hypertension and kidney damage in Dahl salt-sensitive rats.
about
Infiltrating immune cells in the kidney in salt-sensitive hypertension and renal injuryCrosstalk between the unfolded protein response and NF-κB-mediated inflammation in the progression of chronic kidney diseaseUse of ultrasound to assess renal reperfusion and P-selectin expression following unilateral renal ischemiaProton channels and renal hypertensive injury: a key piece of the Dahl salt-sensitive rat puzzle?Cholinergic agonists reduce blood pressure in a mouse model of systemic lupus erythematosus.Dual opposing roles of adaptive immunity in hypertension.Characterization of biological pathways associated with a 1.37 Mbp genomic region protective of hypertension in Dahl S rats.Sex differences in T-lymphocyte tissue infiltration and development of angiotensin II hypertension.Role of Rho GDP dissociation inhibitor α in control of epithelial sodium channel (ENaC)-mediated sodium reabsorptionThe immunological basis of hypertensionHigh dietary protein exacerbates hypertension and renal damage in Dahl SS rats by increasing infiltrating immune cells in the kidney.Sex differences in T cells in hypertension.Infiltrating T lymphocytes in the kidney increase oxidative stress and participate in the development of hypertension and renal diseaseKlotho gene deficiency causes salt-sensitive hypertension via monocyte chemotactic protein-1/CC chemokine receptor 2-mediated inflammation.Cannabidiol as an emergent therapeutic strategy for lessening the impact of inflammation on oxidative stress.Reactive oxygen species as important determinants of medullary flow, sodium excretion, and hypertensionRenal inflammation, autoimmunity and salt-sensitive hypertension.Autoimmunity: an underlying factor in the pathogenesis of hypertension.Preventing autoimmunity protects against the development of hypertension and renal injury.Differential phenotypes of tissue-infiltrating T cells during angiotensin II-induced hypertension in mice.Blood pressure in a hypertensive mouse model of SLE is not salt-sensitive.Mutation of SH2B3 (LNK), a genome-wide association study candidate for hypertension, attenuates Dahl salt-sensitive hypertension via inflammatory modulation.ETA activation mediates angiotensin II-induced infiltration of renal cortical T cells.Association between circulating specific leukocyte types and incident chronic kidney disease: the Atherosclerosis Risk in Communities (ARIC) study.Increased expression of NAD(P)H oxidase subunit p67(phox) in the renal medulla contributes to excess oxidative stress and salt-sensitive hypertension.The Dahl salt-sensitive rat is a spontaneous model of superimposed preeclampsia.T lymphocytes and vascular inflammation contribute to stress-dependent hypertension.Distinct effects on long-term function of injured and contralateral kidneys following unilateral renal ischemia-reperfusionA novel role for type 1 angiotensin receptors on T lymphocytes to limit target organ damage in hypertension.Th-17 cell activation in response to high salt following acute kidney injury is associated with progressive fibrosis and attenuated by AT-1R antagonism.Relationship Between Different Subpopulations of Circulating CD4+ T-lymphocytes and Microvascular Structural Alterations in Humans.Immune Mechanisms Linking Obesity and Preeclampsia.Endothelial Gata5 transcription factor regulates blood pressure.Direct activation of ENaC by angiotensin II: recent advances and new insights.Immunosuppression preserves renal autoregulatory function and microvascular P2X(1) receptor reactivity in ANG II-hypertensive ratsGenetic mutation of recombination activating gene 1 in Dahl salt-sensitive rats attenuates hypertension and renal damageCD70 Exacerbates Blood Pressure Elevation and Renal Damage in Response to Repeated Hypertensive StimuliNeuroimmune communication in hypertension and obesity: a new therapeutic angle?Oxidative stress promotes hypertension and albuminuria during the autoimmune disease systemic lupus erythematosus.Increased proliferative cells in the medullary thick ascending limb of the loop of Henle in the Dahl salt-sensitive rat
P2860
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P2860
T lymphocytes mediate hypertension and kidney damage in Dahl salt-sensitive rats.
description
2010 nî lūn-bûn
@nan
2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
T lymphocytes mediate hypertension and kidney damage in Dahl salt-sensitive rats.
@ast
T lymphocytes mediate hypertension and kidney damage in Dahl salt-sensitive rats.
@en
type
label
T lymphocytes mediate hypertension and kidney damage in Dahl salt-sensitive rats.
@ast
T lymphocytes mediate hypertension and kidney damage in Dahl salt-sensitive rats.
@en
prefLabel
T lymphocytes mediate hypertension and kidney damage in Dahl salt-sensitive rats.
@ast
T lymphocytes mediate hypertension and kidney damage in Dahl salt-sensitive rats.
@en
P2093
P2860
P1476
T lymphocytes mediate hypertension and kidney damage in Dahl salt-sensitive rats.
@en
P2093
Carmen De Miguel
David L Mattson
Hayley Lund
Satarupa Das
P2860
P304
P356
10.1152/AJPREGU.00298.2009
P577
2010-02-10T00:00:00Z