Increased expression of NAD(P)H oxidase subunit p67(phox) in the renal medulla contributes to excess oxidative stress and salt-sensitive hypertension.
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Infiltrating immune cells in the kidney in salt-sensitive hypertension and renal injuryHow the kidney is impacted by the perinatal maternal environment to develop hypertensionTargeted disruption of Adamts16 gene in a rat genetic model of hypertensionDr Lewis Kitchener Dahl, the Dahl rats, and the "inconvenient truth" about the genetics of hypertensionGeneration of Esr1-knockout rats using zinc finger nuclease-mediated genome editing.Proton channels and renal hypertensive injury: a key piece of the Dahl salt-sensitive rat puzzle?Ultrastructure of mitochondria and the endoplasmic reticulum in renal tubules of Dahl salt-sensitive rats.Characterization of biological pathways associated with a 1.37 Mbp genomic region protective of hypertension in Dahl S rats.High-salt diet blunts renal autoregulation by a reactive oxygen species-dependent mechanism.Effects of Reactive Oxygen Species on Tubular Transport along the NephronMutation of Plekha7 attenuates salt-sensitive hypertension in the rat.Reactive oxygen species as important determinants of medullary flow, sodium excretion, and hypertensionBase-resolution maps of 5-methylcytosine and 5-hydroxymethylcytosine in Dahl S rats: effect of salt and genomic sequenceHV1 acts as a sodium sensor and promotes superoxide production in medullary thick ascending limb of Dahl salt-sensitive ratsNull mutation of the nicotinamide adenine dinucleotide phosphate-oxidase subunit p67phox protects the Dahl-S rat from salt-induced reductions in medullary blood flow and glomerular filtration rateAngiotensin II-induced hypertension blunts thick ascending limb NO production by reducing NO synthase 3 expression and enhancing threonine 495 phosphorylation.Evidence for a link between gut microbiota and hypertension in the Dahl rat.Antithrombin III/SerpinC1 insufficiency exacerbates renal ischemia/reperfusion injury.Effects of p67phox on the mitochondrial oxidative state in the kidney of Dahl salt-sensitive rats: optical fluorescence 3-D cryoimagingNADPH oxidases: an overview from structure to innate immunity-associated pathologies.Mitochondrial proteomic analysis reveals deficiencies in oxygen utilization in medullary thick ascending limb of Henle in the Dahl salt-sensitive rat.Acupuncture elicits neuroprotective effect by inhibiting NAPDH oxidase-mediated reactive oxygen species production in cerebral ischaemiaMolecular mechanisms of experimental salt-sensitive hypertension.Evidence of the Importance of Nox4 in Production of Hypertension in Dahl Salt-Sensitive Rats.Matrix metalloproteinase 12 modulates high-fat-diet induced glomerular fibrogenesis and inflammation in a mouse model of obesityRenal dopamine and angiotensin II receptor signaling in age-related hypertension.Construction of two novel reciprocal conplastic rat strains and characterization of cardiac mitochondria.Rat Breeding Parameters According to Floor Space Available in Cage.Pappa2 is linked to salt-sensitive hypertension in Dahl S rats.Renal Tumor Necrosis Factor α Contributes to Hypertension in Dahl Salt-Sensitive Rats.Comparison of 2 rat breeding schemes using conventional caging.Differential gene expression in granulosa cells from polycystic ovary syndrome patients with and without insulin resistance: identification of susceptibility gene sets through network analysisCharacteristics of long non-coding RNAs in the Brown Norway rat and alterations in the Dahl salt-sensitive rat.Deficiency of renal cortical EGF increases ENaC activity and contributes to salt-sensitive hypertension.Epidermal growth factors in the kidney and relationship to hypertension.Detection of endogenous substances with enzymatic microelectrode biosensors in the kidneyIncreased proliferative cells in the medullary thick ascending limb of the loop of Henle in the Dahl salt-sensitive ratProgression of glomerular filtration rate reduction determined in conscious Dahl salt-sensitive hypertensive rats.Rats are the smart choice: Rationale for a renewed focus on rats in behavioral genetics.Enoyl coenzyme a hydratase domain-containing 2, a potential novel regulator of myocardial ischemia injury.
P2860
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P2860
Increased expression of NAD(P)H oxidase subunit p67(phox) in the renal medulla contributes to excess oxidative stress and salt-sensitive hypertension.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Increased expression of NAD(P) ...... d salt-sensitive hypertension.
@ast
Increased expression of NAD(P) ...... d salt-sensitive hypertension.
@en
type
label
Increased expression of NAD(P) ...... d salt-sensitive hypertension.
@ast
Increased expression of NAD(P) ...... d salt-sensitive hypertension.
@en
prefLabel
Increased expression of NAD(P) ...... d salt-sensitive hypertension.
@ast
Increased expression of NAD(P) ...... d salt-sensitive hypertension.
@en
P2093
P2860
P1433
P1476
Increased expression of NAD(P) ...... d salt-sensitive hypertension.
@en
P2093
Allen W Cowley
Aron M Geurts
David L Mattson
Jozef Lazar
Mingyu Liang
Paul M O'Connor
Terry Kurth
P2860
P304
P356
10.1016/J.CMET.2012.01.003
P577
2012-02-01T00:00:00Z