On the identification of potential regulatory variants within genome wide association candidate SNP sets.
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Strategies for Integrated Analysis of Genetic, Epigenetic, and Gene Expression Variation in Cancer: Addressing the ChallengesNIH working group report-using genomic information to guide weight management: From universal to precision treatmentaffy2sv: an R package to pre-process Affymetrix CytoScan HD and 750K arrays for SNP, CNV, inversion and mosaicism callingUsing GWAS to identify novel therapeutic targets for osteoporosis.Integration of VDR genome wide binding and GWAS genetic variation data reveals co-occurrence of VDR and NF-κB binding that is linked to immune phenotypes.Polymorphisms of ESR1, UGT1A1, HCN1, MAP3K1 and CYP2B6 are associated with the prognosis of hormone receptor-positive early breast cancer.Role of non-coding sequence variants in cancer.Psoriasis drug development and GWAS interpretation through in silico analysis of transcription factor binding sitesIn-silico identification and functional validation of allele-dependent AR enhancers.Identification and annotation of conserved promoters and macrophage-expressed genes in the pig genomeObesity-related known and candidate SNP markers can significantly change affinity of TATA-binding protein for human gene promoters.In-silico QTL mapping of postpubertal mammary ductal development in the mouse uncovers potential human breast cancer risk loci.Candidate SNP markers of aggressiveness-related complications and comorbidities of genetic diseases are predicted by a significant change in the affinity of TATA-binding protein for human gene promoters.Genetics of Common Endocrine Disease: The Present and the FutureCandidate SNP Markers of Gender-Biased Autoimmune Complications of Monogenic Diseases Are Predicted by a Significant Change in the Affinity of TATA-Binding Protein for Human Gene PromotersCandidate SNP Markers of Chronopathologies Are Predicted by a Significant Change in the Affinity of TATA-Binding Protein for Human Gene Promoters.Kruppel-like factor4 regulates PRDM1 expression through binding to an autoimmune risk allele.Genome-wide association studies and epigenome-wide association studies go together in cancer control.Genome-wide association analysis on normal hearing function identifies PCDH20 and SLC28A3 as candidates for hearing function and loss.Neuregulin signaling pathway in smoking behavior.How to Use SNP_TATA_Comparator to Find a Significant Change in Gene Expression Caused by the Regulatory SNP of This Gene's Promoter via a Change in Affinity of the TATA-Binding Protein for This Promoter.Differential Expression of Hypertensive Phenotypes in BXD Mouse Strains in Response to Angiotensin II.Identification of potential regulatory mutations using multi-omics analysis and haplotyping of lung adenocarcinoma cell lines.Candidate SNP markers of reproductive potential are predicted by a significant change in the affinity of TATA-binding protein for human gene promoters.
P2860
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P2860
On the identification of potential regulatory variants within genome wide association candidate SNP sets.
description
2014 nî lūn-bûn
@nan
2014 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2014年の論文
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2014年学术文章
@wuu
2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
@yue
name
On the identification of poten ...... ssociation candidate SNP sets.
@ast
On the identification of poten ...... ssociation candidate SNP sets.
@en
type
label
On the identification of poten ...... ssociation candidate SNP sets.
@ast
On the identification of poten ...... ssociation candidate SNP sets.
@en
prefLabel
On the identification of poten ...... ssociation candidate SNP sets.
@ast
On the identification of poten ...... ssociation candidate SNP sets.
@en
P2860
P356
P1433
P1476
On the identification of poten ...... ssociation candidate SNP sets.
@en
P2093
Chao A Hsiung
I-Shou Chang
P2860
P2888
P356
10.1186/1755-8794-7-34
P577
2014-06-11T00:00:00Z
P5875
P6179
1035899473