Temperature-dependent modulation of Porphyromonas gingivalis lipid A structure and interaction with the innate host defenses.
about
Periodontitis: from microbial immune subversion to systemic inflammationA divergent Pseudomonas aeruginosa palmitoyltransferase essential for cystic fibrosis-specific lipid A.Distinct lipid a moieties contribute to pathogen-induced site-specific vascular inflammation.Tet-Trap, a genetic approach to the identification of bacterial RNA thermometers: application to Pseudomonas aeruginosaPorphyromonas gingivalis lipopolysaccharide weakly activates M1 and M2 polarized mouse macrophages but induces inflammatory cytokines.Porphyromonas gingivalis fimbriae dampen P2X7-dependent interleukin-1β secretion.Beyond the red complex and into more complexity: the polymicrobial synergy and dysbiosis (PSD) model of periodontal disease etiology.Analysis of carbohydrates and glycoconjugates by matrix-assisted laser desorption/ionization mass spectrometry: An update for 2011-2012.Role of the Vibrio cholerae matrix protein Bap1 in cross-resistance to antimicrobial peptides.Oral Microbiome Metabolism: From "Who Are They?" to "What Are They Doing?".Influence of saliva on the oral microbiota.Porphyromonas gingivalis virulence factors involved in subversion of leukocytes and microbial dysbiosisProtective role for TLR4 signaling in atherosclerosis progression as revealed by infection with a common oral pathogen.LptO (PG0027) Is Required for Lipid A 1-Phosphatase Activity in Porphyromonas gingivalis W50.3LPS-binding protein and its interactions with P. gingivalis LPS modulate pro-inflammatory response and Toll-like receptor signaling in human oral keratinocytes.Modification of the 1-Phosphate Group during Biosynthesis of Capnocytophaga canimorsus Lipid A.Environmental stimuli shape biofilm formation and the virulence of periodontal pathogensHeterogeneous Porphyromonas gingivalis LPS modulates immuno-inflammatory response, antioxidant defense and cytoskeletal dynamics in human gingival fibroblasts.Porphyromonas gingivalis lipid A phosphatase activity is critical for colonization and increasing the commensal load in the rabbit ligature model.Breaking bad: manipulation of the host response by Porphyromonas gingivalis.The role of lipopolysaccharide-binding protein in innate immunity: a revisit and its relevance to oral/periodontal health.Defensins and LL-37: a review of function in the gingival epitheliumReviewing and identifying amino acids of human, murine, canine and equine TLR4 / MD-2 receptor complexes conferring endotoxic innate immunity activation by LPS/lipid A, or antagonistic effects by Eritoran, in contrast to species-dependent modulationDifferential Regulation of Mas-Related G Protein-Coupled Receptor X2-Mediated Mast Cell Degranulation by Antimicrobial Host Defense Peptides and Porphyromonas gingivalis Lipopolysaccharide.Variants of Porphyromonas gingivalis lipopolysaccharide alter lipidation of autophagic protein, microtubule-associated protein 1 light chain 3, LC3.Participation of endotoxin in root canal infections: A systematic review and meta-analysisDifferences in survival, virulence and biofilm formation between sialidase-deficient and W83 wild-type Porphyromonas gingivalis strains under stressful environmental conditions.Porphyromonas gingivalis lipopolysaccharide regulates ephrin/Eph signalling in human periodontal ligament fibroblasts.The antibacterial activity of LL-37 against Treponema denticola is dentilisin protease independent and facilitated by the major outer sheath protein virulence factor.Total Lipopolysaccharide from the Human Gut Microbiome Silences Toll-Like Receptor Signaling.Characterization of complex, heterogeneous lipid A samples using HPLC-MS/MS technique II. Structural elucidation of non-phosphorylated lipid A by negative-ion mode tandem mass spectrometry.Aminosugar-based immunomodulator lipid A: synthetic approaches.A D-enantiomer of the antimicrobial peptide GL13K evades antimicrobial resistance in the Gram positive bacteria Enterococcus faecalis and Streptococcus gordonii.Comprehensive analysis of clinical Burkholderia pseudomallei isolates demonstrates conservation of unique lipid A structure and TLR4-dependent innate immune activation.Importance of heterogeneity in Porhyromonas gingivalis lipopolysaccharide lipid A in tissue specific inflammatory signalling.
P2860
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P2860
Temperature-dependent modulation of Porphyromonas gingivalis lipid A structure and interaction with the innate host defenses.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Temperature-dependent modulati ...... with the innate host defenses.
@ast
Temperature-dependent modulati ...... with the innate host defenses.
@en
type
label
Temperature-dependent modulati ...... with the innate host defenses.
@ast
Temperature-dependent modulati ...... with the innate host defenses.
@en
prefLabel
Temperature-dependent modulati ...... with the innate host defenses.
@ast
Temperature-dependent modulati ...... with the innate host defenses.
@en
P2093
P2860
P356
P1476
Temperature-dependent modulati ...... with the innate host defenses
@en
P2093
Deirdre Devine
Edward Tarelli
Minnie Rangarajan
Philip D Marsh
Richard P Darveau
Rimondia S Percival
Stephen R Coats
P2860
P304
P356
10.1128/IAI.00900-10
P407
P577
2011-01-10T00:00:00Z