Friedreich's ataxia (GAA)n•(TTC)n repeats strongly stimulate mitotic crossovers in Saccharomyces cerevisae.
about
Mechanisms and regulation of mitotic recombination in Saccharomyces cerevisiaeThe balancing act of DNA repeat expansionsThe hidden side of unstable DNA repeats: Mutagenesis at a distanceGenome-wide high-resolution mapping of chromosome fragile sites in Saccharomyces cerevisiae.High-resolution mapping of two types of spontaneous mitotic gene conversion events in Saccharomyces cerevisiaeHigh-resolution mapping of spontaneous mitotic recombination hotspots on the 1.1 Mb arm of yeast chromosome IVA 5', 8-cyclo-2'-deoxypurine lesion induces trinucleotide repeat deletion via a unique lesion bypass by DNA polymerase βFragile DNA motifs trigger mutagenesis at distant chromosomal loci in saccharomyces cerevisiae.Fragile site instability in Saccharomyces cerevisiae causes loss of heterozygosity by mitotic crossovers and break-induced replication.Stress alters rates and types of loss of heterozygosity in Candida albicans.Topoisomerase I plays a critical role in suppressing genome instability at a highly transcribed G-quadruplex-forming sequence.Involvement of a citrus meiotic recombination TTC-repeat motif in the formation of gross deletions generated by ionizing radiation and MULE activation.Repeat instability during DNA repair: Insights from model systemsHigh-resolution genome-wide analysis of irradiated (UV and γ-rays) diploid yeast cells reveals a high frequency of genomic loss of heterozygosity (LOH) events.Trinucleotide repeat deletion via a unique hairpin bypass by DNA polymerase β and alternate flap cleavage by flap endonuclease 1.Gene copy-number variation in haploid and diploid strains of the yeast Saccharomyces cerevisiaeNonrandom distribution of interhomolog recombination events induced by breakage of a dicentric chromosome in Saccharomyces cerevisiae.Base excision repair of oxidative DNA damage coupled with removal of a CAG repeat hairpin attenuates trinucleotide repeat expansion.Role of recombination and replication fork restart in repeat instability.Genome-wide screen identifies pathways that govern GAA/TTC repeat fragility and expansions in dividing and nondividing yeast cells.Replacement of the myotonic dystrophy type 1 CTG repeat with 'non-CTG repeat' insertions in specific tissues.Genomic deletions and point mutations induced in Saccharomyces cerevisiae by the trinucleotide repeats (GAA·TTC) associated with Friedreich's ataxiaFriedreich's ataxia-associated GAA repeats induce replication-fork reversal and unusual molecular junctions.The interplay between G-quadruplex and Transcription.Genetic Control of Genomic Alterations Induced in Yeast by Interstitial Telomeric Sequences.
P2860
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P2860
Friedreich's ataxia (GAA)n•(TTC)n repeats strongly stimulate mitotic crossovers in Saccharomyces cerevisae.
description
2011 nî lūn-bûn
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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Friedreich's ataxia (GAA)n•(TT ...... rs in Saccharomyces cerevisae.
@ast
Friedreich's ataxia (GAA)n•(TT ...... rs in Saccharomyces cerevisae.
@en
type
label
Friedreich's ataxia (GAA)n•(TT ...... rs in Saccharomyces cerevisae.
@ast
Friedreich's ataxia (GAA)n•(TT ...... rs in Saccharomyces cerevisae.
@en
prefLabel
Friedreich's ataxia (GAA)n•(TT ...... rs in Saccharomyces cerevisae.
@ast
Friedreich's ataxia (GAA)n•(TT ...... rs in Saccharomyces cerevisae.
@en
P2093
P2860
P1433
P1476
Friedreich's ataxia (GAA)n•(TT ...... ers in Saccharomyces cerevisae
@en
P2093
Hyun-Min Kim
Kirill S Lobachev
Margaret Dominska
Patricia W Greenwell
Thomas D Petes
Vidhya Narayanan
P2860
P304
P356
10.1371/JOURNAL.PGEN.1001270
P577
2011-01-13T00:00:00Z