The exon junction complex component Magoh controls brain size by regulating neural stem cell division. - Wikidata - wikimedia - TriplyDB

The exon junction complex component Magoh controls brain size by regulating neural stem cell division.

The exon junction complex component Magoh controls brain size by regulating neural stem cell division.

http://www.wikidata.org/entity/Q33814636

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Proteins associated with the exon junction complex also control the alternative splicing of apoptotic regulators The cellular EJC interactome reveals higher-order mRNP structure and an EJC-SR protein nexus Two mammalian MAGOH genes contribute to exon junction complex composition and nonsense-mediated decay Microcephaly with simplified gyration, epilepsy, and infantile diabetes linked to inappropriate apoptosis of neural progenitors Current Evidence for Developmental, Structural, and Functional Brain Defects following Prenatal Radiation Exposure Spindle orientation in mammalian cerebral cortical development Post-transcriptional regulation in corticogenesis: how RNA-binding proteins help build the brain Nonsense-mediated mRNA decay: inter-individual variability and human disease The Clothes Make the mRNA: Past and Present Trends in mRNP Fashion Adult Deletion of SRF Increases Epileptogenesis and Decreases Activity-Induced Gene Expression.ZIKA virus elicits P53 activation and genotoxic stress in human neural progenitors similar to mutations involved in severe forms of genetic microcephaly and p53 CDC174, a novel component of the exon junction complex whose mutation underlies a syndrome of hypotonia and psychomotor developmental delay p21-Activated kinases 1 and 3 control brain size through coordinating neuronal complexity and synaptic properties Human brain evolution: harnessing the genomics (r)evolution to link genes, cognition, and behavior Convergence and divergence between the transcriptional responses to Zika virus infection and prenatal irradiation The exon junction complex in neural development and neurodevelopmental disease.The closely related RNA helicases, UAP56 and URH49, preferentially form distinct mRNA export machineries and coordinately regulate mitotic progression Prolonged Mitosis of Neural Progenitors Alters Cell Fate in the Developing Brain.Bmp7 maintains undifferentiated kidney progenitor population and determines nephron numbers at birth Contribution of copy number variants involving nonsense-mediated mRNA decay pathway genes to neuro-developmental disorders.Function and pathological implications of exon junction complex factor Y14 The RNA-binding protein MARF1 promotes cortical neurogenesis through its RNase activity domain In vivo fate analysis reveals the multipotent and self-renewal features of embryonic AspM expressing cells.Exon junction complex subunits are required to splice Drosophila MAP kinase, a large heterochromatic gene Modifier genes and the plasticity of genetic networks in mice.Live imaging of mitosis in the developing mouse embryonic cortex.The exon junction complex controls transposable element activity by ensuring faithful splicing of the piwi transcript.Temporally defined neocortical translation and polysome assembly are determined by the RNA-binding protein Hu antigen R.The RNAissance family: SR proteins as multifaceted regulators of gene expression Transcriptome profiling of UPF3B/NMD-deficient lymphoblastoid cells from patients with various forms of intellectual disability.Identification of a microRNA that activates gene expression by repressing nonsense-mediated RNA decay.Rbm8a haploinsufficiency disrupts embryonic cortical development resulting in microcephaly.A critical role of RBM8a in proliferation and differentiation of embryonic neural progenitors.A conserved microRNA/NMD regulatory circuit controls gene expression.Haploinsufficiency for Core Exon Junction Complex Components Disrupts Embryonic Neurogenesis and Causes p53-Mediated Microcephaly Aspm sustains postnatal cerebellar neurogenesis and medulloblastoma growth in mice.Mitotic spindle (DIS)orientation and DISease: cause or consequence?Maternal dietary intake of choline in mice regulates development of the cerebral cortex in the offspring.The EJC component Magoh regulates proliferation and expansion of neural crest-derived melanocytes.Phosphorylation status of human RNA-binding protein 8A in cells and its inhibitory regulation by Magoh

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The exon junction complex component Magoh controls brain size by regulating neural stem cell division.

http://www.wikidata.org/entity/Q33814636

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

The exon junction complex comp ...... ing neural stem cell division.

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The exon junction complex comp ...... ing neural stem cell division.

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type

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The exon junction complex comp ...... ing neural stem cell division.

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The exon junction complex comp ...... ing neural stem cell division.

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prefLabel

The exon junction complex comp ...... ing neural stem cell division.

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The exon junction complex comp ...... ing neural stem cell division.

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Nature Neuroscience

P1476

The exon junction complex comp ...... ting neural stem cell division

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P2093

Anthony J Burnetti

http://www.w3.org/2001/XMLSchema#string

Christopher A Walsh

http://www.w3.org/2001/XMLSchema#string

Debra L Silver

http://www.w3.org/2001/XMLSchema#string

Denise M Larson

http://www.w3.org/2001/XMLSchema#string

Kyungjae Myung

http://www.w3.org/2001/XMLSchema#string

Nicholas Gaiano

http://www.w3.org/2001/XMLSchema#string

Tarran J Pierfelice

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Multicolor Spectral Karyotyping of Human Chromosomes

Identification of microcephalin, a protein implicated in determining the size of the human brain

A role for the lissencephaly gene LIS1 in mitosis and cytoplasmic dynein function

PYM binds the cytoplasmic exon-junction complex and ribosomes to enhance translation of spliced mRNAs

The human RNA surveillance factor UPF1 is required for S phase progression and genome stability

An eIF4AIII-containing complex required for mRNA localization and nonsense-mediated mRNA decay

Magoh, a human homolog of Drosophila mago nashi protein, is a component of the splicing-dependent exon-exon junction complex.

A centrosomal mechanism involving CDK5RAP2 and CENPJ controls brain size

GenePaint.org: an atlas of gene expression patterns in the mouse embryo

Splicing enhances translation in mammalian cells: an additional function of the exon junction complex

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551-558

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scholarly article

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10.1038/NN.2527

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P433

5

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13

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William J Pavan

Karisa C. Schreck

Dawn E Watkins-Chow

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2010-04-04T00:00:00Z

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42975993

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20364144

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2860667

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