The insect homologue of the amyloid precursor protein interacts with the heterotrimeric G protein Go alpha in an identified population of migratory neurons.
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Analysis of Amyloid Precursor Protein Function in Drosophila melanogasterAmyloid β precursor protein as a molecular target for amyloid β--induced neuronal degeneration in Alzheimer's diseaseHow to innervate a simple gut: familiar themes and unique aspects in the formation of the insect enteric nervous system.Amyloid Precursor Proteins Are Dynamically Trafficked and Processed during Neuronal Development.Characterization of the beta amyloid precursor protein-like gene in the central nervous system of the crab Chasmagnathus. Expression during memory consolidationIncreased RhoA prenylation in the loechrig (loe) mutant leads to progressive neurodegenerationReverse signaling by glycosylphosphatidylinositol-linked Manduca ephrin requires a SRC family kinase to restrict neuronal migration in vivo.A translational continuum of model systems for evaluating treatment strategies in Alzheimer's disease: isradipine as a candidate drug.Neurotoxic effects induced by the Drosophila amyloid-beta peptide suggest a conserved toxic function.Eph receptor expression defines midline boundaries for ephrin-positive migratory neurons in the enteric nervous system of Manduca sexta.Amyloid precursor proteins interact with the heterotrimeric G protein Go in the control of neuronal migrationManduca Contactin Regulates Amyloid Precursor Protein-Dependent Neuronal Migration.Neuronal migration during development and the amyloid precursor protein.Role of APP Interactions with Heterotrimeric G Proteins: Physiological Functions and Pathological ConsequencesAmyloid Precursor Protein family as unconventional Go-coupled receptors and the control of neuronal motility.The physiological role of the amyloid precursor protein as an adhesion molecule in the developing nervous system.Impact of a discordant helix on β-amyloid structure, aggregation ability and toxicity.Interactions between the amyloid precursor protein C-terminal domain and G proteins mediate calcium dysregulation and amyloid beta toxicity in Alzheimer's disease.
P2860
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P2860
The insect homologue of the amyloid precursor protein interacts with the heterotrimeric G protein Go alpha in an identified population of migratory neurons.
description
2005 nî lūn-bûn
@nan
2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
The insect homologue of the am ...... pulation of migratory neurons.
@ast
The insect homologue of the am ...... pulation of migratory neurons.
@en
type
label
The insect homologue of the am ...... pulation of migratory neurons.
@ast
The insect homologue of the am ...... pulation of migratory neurons.
@en
prefLabel
The insect homologue of the am ...... pulation of migratory neurons.
@ast
The insect homologue of the am ...... pulation of migratory neurons.
@en
P2093
P2860
P1476
The insect homologue of the am ...... pulation of migratory neurons.
@en
P2093
L M Knittel
M A Snyder
P F Copenhaver
S M Farley
T L Swanson
P2860
P304
P356
10.1016/J.YDBIO.2005.09.029
P407
P577
2005-10-17T00:00:00Z