mRNA and protein levels for GABAAalpha4, alpha5, beta1 and GABABR1 receptors are altered in brains from subjects with autism - Wikidata - wikimedia - TriplyDB

mRNA and protein levels for GABAAalpha4, alpha5, beta1 and GABABR1 receptors are altered in brains from subjects with autism

mRNA and protein levels for GABAAalpha4, alpha5, beta1 and GABABR1 receptors are altered in brains from subjects with autism

http://www.wikidata.org/entity/Q33833647

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The role of cerebellar circuitry alterations in the pathophysiology of autism spectrum disorders Promoting social plasticity in developmental disorders with non-invasive brain stimulation techniques Prenatal stress and inhibitory neuron systems: implications for neuropsychiatric disorders Consensus paper: pathological role of the cerebellum in autism Cytokine dysregulation in autism spectrum disorders (ASD): possible role of the environment Functional regulation of GABAA receptors in nervous system pathologies Changes in plasticity across the lifespan: cause of disease and target for intervention Advancing the understanding of autism disease mechanisms through genetics Use of transcranial magnetic stimulation in autism spectrum disorders The dynamics of autism spectrum disorders: how neurotoxic compounds and neurotransmitters interact Histopathologic characterization of the BTBR mouse model of autistic-like behavior reveals selective changes in neurodevelopmental proteins and adult hippocampal neurogenesis Calbindin Knockout Alters Sex-Specific Regulation of Behavior and Gene Expression in Amygdala and Prefrontal Cortex.GABA receptor subunit distribution and FMRP-mGluR5 signaling abnormalities in the cerebellum of subjects with schizophrenia, mood disorders, and autism.Repetitive behavior profile and supersensitivity to amphetamine in the C58/J mouse model of autism.GABAA receptor subtypes: Therapeutic potential in Down syndrome, affective disorders, schizophrenia, and autism.Diversity of neuronal inhibition: a path to novel treatments for neuropsychiatric disorders.Expression of GABAA α2-, β1- and ε-receptors are altered significantly in the lateral cerebellum of subjects with schizophrenia, major depression and bipolar disorder The role of fragile X mental retardation protein in major mental disorders.Deficits in GABA(B) receptor system in schizophrenia and mood disorders: a postmortem study.Brain plasticity and disease: a matter of inhibition.The BOLD signal and neurovascular coupling in autism Modulation of GABAergic transmission in development and neurodevelopmental disorders: investigating physiology and pathology to gain therapeutic perspectives.GABAA receptor subunit gene polymorphisms predict symptom-based and developmental deficits in Chinese Han children and adolescents with autistic spectrum disorders Genetic analysis of GABRB3 as a candidate gene of autism spectrum disorders Modulation of corticospinal excitability by transcranial magnetic stimulation in children and adolescents with autism spectrum disorder.Brief report: an open-label study of the neurosteroid pregnenolone in adults with autism spectrum disorder Increases in [3H]muscimol and [3H]flumazenil binding in the dorsolateral prefrontal cortex in schizophrenia are linked to α4 and γ2S mRNA levels respectively.Neurodevelopment, GABA system dysfunction, and schizophrenia Genetic controls balancing excitatory and inhibitory synaptogenesis in neurodevelopmental disorder models.Developmental exposure to the organochlorine insecticide endosulfan alters expression of proteins associated with neurotransmission in the frontal cortex Autism: a "critical period" disorder?Metabotropic glutamate receptor 5 upregulation in children with autism is associated with underexpression of both Fragile X mental retardation protein and GABAA receptor beta 3 in adults with autism Trajectory of frequency stability in typical development.Late development of the GABAergic system in the human cerebral cortex and white matter Tonic Inhibitory Control of Dentate Gyrus Granule Cells by α5-Containing GABAA Receptors Reduces Memory Interference Dysregulation of fragile × mental retardation protein and metabotropic glutamate receptor 5 in superior frontal cortex of individuals with autism: a postmortem brain study.Abnormal modulation of corticospinal excitability in adults with Asperger's syndrome.The basolateral amygdala γ-aminobutyric acidergic system in health and disease.α5GABAA receptor deficiency causes autism-like behaviors Synaptic pruning in the female hippocampus is triggered at puberty by extrasynaptic GABAA receptors on dendritic spines

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mRNA and protein levels for GABAAalpha4, alpha5, beta1 and GABABR1 receptors are altered in brains from subjects with autism

http://www.wikidata.org/entity/Q33833647

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

mRNA and protein levels for GA ...... ains from subjects with autism

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mRNA and protein levels for GA ...... ains from subjects with autism

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type

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mRNA and protein levels for GA ...... ains from subjects with autism

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mRNA and protein levels for GA ...... ains from subjects with autism

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prefLabel

mRNA and protein levels for GA ...... ains from subjects with autism

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mRNA and protein levels for GA ...... ains from subjects with autism

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Journal of Autism and Developmental Disorders

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mRNA and protein levels for GA ...... ains from subjects with autism

@en

P2093

Diven H Patel

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Paul D Thuras

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Robert J Rooney

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S Hossein Fatemi

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Teri J Reutiman

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Timothy D Folsom

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P2860

Epigenetic overlap in autism-spectrum neurodevelopmental disorders: MECP2 deficiency causes reduced expression of UBE3A and GABRB3.

Identification of significant association and gene-gene interaction of GABA receptor subunit genes in autism

Investigation of autism and GABA receptor subunit genes in multiple ethnic groups

International Union of Pharmacology. LXX. Subtypes of gamma-aminobutyric acid(A) receptors: classification on the basis of subunit composition, pharmacology, and function. Update

15q11-13 GABAA receptor genes are normally biallelically expressed in brain yet are subject to epigenetic dysregulation in autism-spectrum disorders

Chromosome 15q11-13 duplication syndrome brain reveals epigenetic alterations in gene expression not predicted from copy number.

Pathway-based association analysis of genome-wide screening data suggest that genes associated with the gamma-aminobutyric acid receptor signaling pathway are involved in neuroleptic-induced, treatment-resistant tardive dyskinesia.

Epilepsy in autism.

GABAergic dysfunction in schizophrenia and mood disorders as reflected by decreased levels of glutamic acid decarboxylase 65 and 67 kDa and Reelin proteins in cerebellum.

Decreased GAD67 mRNA levels in cerebellar Purkinje cells in autism: pathophysiological implications.

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