GABAergic dysfunction in schizophrenia and mood disorders as reflected by decreased levels of glutamic acid decarboxylase 65 and 67 kDa and Reelin proteins in cerebellum.
about
The GABA B receptor agonist CGP44532 and the positive modulator GS39783 reverse some behavioural changes related to positive syndromes of psychosis in mice.GABAergic interneuron origin of schizophrenia pathophysiology.Histone modifications, DNA methylation, and schizophreniaThe involvement of Reelin in neurodevelopmental disorders.The neurodevelopmental hypothesis of schizophrenia, revisitedIn Sickness and in Health: Perineuronal Nets and Synaptic Plasticity in Psychiatric DisordersActive DNA demethylation in post-mitotic neurons: a reason for optimismTranscriptional regulation of GAD1 GABA synthesis gene in the prefrontal cortex of subjects with schizophreniaThe X-Ray Crystal Structure of Escherichia coli Succinic Semialdehyde Dehydrogenase; Structural Insights into NADP+/Enzyme InteractionsGrowth arrest and DNA-damage-inducible, beta (GADD45b)-mediated DNA demethylation in major psychosisThe GABAβ receptor as a target for antidepressant drug actionDysfunction in GABA signalling mediates autism-like stereotypies and Rett syndrome phenotypesThe Neurobiological Basis for Social Affiliation in Autism Spectrum Disorder and Schizophrenia.Animal models of gene-environment interaction in schizophrenia: A dimensional perspective.Distinct behavioral consequences of short-term and prolonged GABAergic depletion in prefrontal cortex and dorsal hippocampus.Expression of GABAA α2-, β1- and ε-receptors are altered significantly in the lateral cerebellum of subjects with schizophrenia, major depression and bipolar disorderDeficits in emotional learning and memory in an animal model of schizophreniaThe role of fragile X mental retardation protein in major mental disorders.Deficits in GABA(B) receptor system in schizophrenia and mood disorders: a postmortem study.Eye-blink conditioning deficits indicate temporal processing abnormalities in schizophreniaAbnormalities in brain structure and behavior in GSK-3alpha mutant miceThe reelin receptors VLDLR and ApoER2 regulate sensorimotor gating in mice.Vldlr overexpression causes hyperactivity in rats.Altered γ-aminobutyric acid neurotransmission in major depressive disorder: a critical review of the supporting evidence and the influence of serotonergic antidepressantsGAD1 mRNA expression and DNA methylation in prefrontal cortex of subjects with schizophrenia.Increased timing variability in schizophrenia and bipolar disorder.Modulation of GABAergic transmission in development and neurodevelopmental disorders: investigating physiology and pathology to gain therapeutic perspectives.Reduced density of calbindin immunoreactive GABAergic neurons in the occipital cortex in major depression: relevance to neuroimaging studies.Lower number of cerebellar Purkinje neurons in psychosis is associated with reduced reelin expression.Reduced level of glutamic acid decarboxylase-67 kDa in the prefrontal cortex in major depression.mRNA and protein levels for GABAAalpha4, alpha5, beta1 and GABABR1 receptors are altered in brains from subjects with autismPostural control in bipolar disorder: increased sway area and decreased dynamical complexity.Peripubertal stress-induced behavioral changes are associated with altered expression of genes involved in excitation and inhibition in the amygdala.Impaired cerebellar-dependent eyeblink conditioning in first-degree relatives of individuals with schizophrenia.Mapping pathological phenotypes in reelin mutant mice.The molecular pathology of schizophrenia--focus on histone and DNA modificationsUpregulation of TET1 and downregulation of APOBEC3A and APOBEC3C in the parietal cortex of psychotic patients.Neurodevelopment, GABA system dysfunction, and schizophreniaPersonalized medicine in psychiatry: problems and promises.Cognitive dysfunction associated with anti-glutamic acid decarboxylase autoimmunity: a case-control study.
P2860
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P2860
GABAergic dysfunction in schizophrenia and mood disorders as reflected by decreased levels of glutamic acid decarboxylase 65 and 67 kDa and Reelin proteins in cerebellum.
description
2005 nî lūn-bûn
@nan
2005 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
GABAergic dysfunction in schiz ...... Reelin proteins in cerebellum.
@ast
GABAergic dysfunction in schiz ...... Reelin proteins in cerebellum.
@en
GABAergic dysfunction in schiz ...... Reelin proteins in cerebellum.
@nl
type
label
GABAergic dysfunction in schiz ...... Reelin proteins in cerebellum.
@ast
GABAergic dysfunction in schiz ...... Reelin proteins in cerebellum.
@en
GABAergic dysfunction in schiz ...... Reelin proteins in cerebellum.
@nl
prefLabel
GABAergic dysfunction in schiz ...... Reelin proteins in cerebellum.
@ast
GABAergic dysfunction in schiz ...... Reelin proteins in cerebellum.
@en
GABAergic dysfunction in schiz ...... Reelin proteins in cerebellum.
@nl
P2093
P1476
GABAergic dysfunction in schiz ...... Reelin proteins in cerebellum.
@en
P2093
Elisabeth Eagan
Joel M Stary
Julie A Earle
Mohsen Araghi-Niknam
S Hossein Fatemi
P304
P356
10.1016/J.SCHRES.2004.02.017
P407
P577
2005-01-01T00:00:00Z