Senescent cells as a source of inflammatory factors for tumor progression.
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Androgen deprivation-induced senescence promotes outgrowth of androgen-refractory prostate cancer cellsSenescence in Human Mesenchymal Stem Cells: Functional Changes and Implications in Stem Cell-Based TherapyEvaluating biomarkers to model cancer risk post cosmic ray exposureThe Growing Complexity of Cancer Cell Response to DNA-Damaging Agents: Caspase 3 Mediates Cell Death or Survival?Role of Natural Radiosensitizers and Cancer Cell Radioresistance: An UpdateTherapeutic Implications for Overcoming Radiation Resistance in Cancer TherapyParainflammation, chronic inflammation, and age-related macular degenerationThe Impact of Immunosenescence on Pulmonary DiseaseThe quiescent cellular state is Arf/p53-dependent and associated with H2AX downregulation and genome stabilityTranscriptional integration of mitochondrial biogenesisTherapeutic targeting of replicative immortalityModulation of inflammation by autophagy: Consequences for human diseaseAutophagy in thyroid cancer: present knowledge and future perspectivesRace, Ethnicity, Psychosocial Factors, and Telomere Length in a Multicenter SettingDefining the toxicology of agingTelomeres and telomerase in head and neck squamous cell carcinoma: from pathogenesis to clinical implications.Changes in the expression of the Toll-like receptor system in the aging rat kidneysSubtelomeric CTCF and cohesin binding site organization using improved subtelomere assemblies and a novel annotation pipeline.Simple biologically informed inflammatory index of two serum cytokines predicts 10 year all-cause mortality in older adults.Oncogenic senescence: a multi-functional perspective.Host mediated inflammatory influence on glioblastoma multiforme recurrence following high-dose ionizing radiation.Gene polymorphisms of cellular senescence marker p21 and disease progression in non-alcohol-related fatty liver diseaseDeep sequencing reveals novel microRNAs and regulation of microRNA expression during cell senescence.Palbociclib: an evidence-based review of its potential in the treatment of breast cancerExploiting tumor cell senescence in anticancer therapyGlucocorticoids suppress selected components of the senescence-associated secretory phenotype.Aging, cellular senescence, and cancerLong-distance inflammatory and genotoxic impact of cancer in vivo.Astrocyte senescence as a component of Alzheimer's disease.Tackling the tumor microenvironment: what challenge does it pose to anticancer therapies?Ageing induced vascular smooth muscle cell senescence in atherosclerosis.Oxidative stress damage as a detrimental factor in preterm birth pathologySimvastatin suppresses breast cancer cell proliferation induced by senescent cellsReciprocal interaction between carcinoma-associated fibroblasts and squamous carcinoma cells through interleukin-1α induces cancer progressionPinX1 the tail on the chromosomeSenescent cardiac fibroblast is critical for cardiac fibrosis after myocardial infarction.p38MAPK is a novel DNA damage response-independent regulator of the senescence-associated secretory phenotype.Epithelial cell senescence impairs repair process and exacerbates inflammation after airway injury.A p53/ARF-dependent anticancer barrier activates senescence and blocks tumorigenesis without impacting apoptosisIncreased levels of circulating interleukin 6, interleukin 8, C-reactive protein, and risk of lung cancer
P2860
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P2860
Senescent cells as a source of inflammatory factors for tumor progression.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Senescent cells as a source of inflammatory factors for tumor progression.
@ast
Senescent cells as a source of inflammatory factors for tumor progression.
@en
type
label
Senescent cells as a source of inflammatory factors for tumor progression.
@ast
Senescent cells as a source of inflammatory factors for tumor progression.
@en
prefLabel
Senescent cells as a source of inflammatory factors for tumor progression.
@ast
Senescent cells as a source of inflammatory factors for tumor progression.
@en
P2093
P2860
P921
P1476
Senescent cells as a source of inflammatory factors for tumor progression.
@en
P2093
Albert R Davalos
Jean-Philippe Coppe
Judith Campisi
Pierre-Yves Desprez
P2860
P2888
P304
P356
10.1007/S10555-010-9220-9
P577
2010-06-01T00:00:00Z
P5875
P6179
1021410177