Epithelial cell senescence impairs repair process and exacerbates inflammation after airway injury.
about
Airway epithelial repair in health and disease: Orchestrator or simply a player?Changes in Regenerative Capacity through LifespanSIRT1 as a therapeutic target in inflammaging of the pulmonary diseaseThe Telomere/Telomerase System in Chronic Inflammatory Diseases. Cause or Effect?Cellular senescence in normal and premature lung aging.Aging exacerbates damage and delays repair of alveolar epithelia following influenza viral pneumonia.Delay of airway epithelial wound repair in COPD is associated with airflow obstruction severityAging does not enhance experimental cigarette smoke-induced COPD in the mouseThe pathogenesis of COPD and IPF: distinct horns of the same devil?PARK2-mediated mitophagy is involved in regulation of HBEC senescence in COPD pathogenesis.Involvement of Igf1r in Bronchiolar Epithelial Regeneration: Role during Repair Kinetics after Selective Club Cell Ablation.Cellular and molecular factors in flexor tendon repair and adhesions: a histological and gene expression analysis.Modulation of bacterial pathogenesis by oppressive aging factors: insights into host-pneumococcal interaction strategiesInfluenza virus specific CD8⁺ T cells exacerbate infection following high dose influenza challenge of aged mice.The role of club cell phenoconversion and migration in idiopathic pulmonary fibrosis.Cellular Senescence - its role in cancer and the response to ionizing radiation.Radiation induced pulmonary fibrosis as a model of progressive fibrosis: Contributions of DNA damage, inflammatory response and cellular senescence genes.Sex-Specific Routes To Immune Senescence In Drosophila melanogaster.Aged blood factors decrease cellular responses associated with delayed gingival wound repair.Increased expression of senescence markers in cystic fibrosis airwaysDecreased serum transforming growth factor-β1 concentration with aging is associated with the severity of emphysema in chronic obstructive pulmonary disease.Shelterin Telomere Protection Protein 1 Reduction Causes Telomere Attrition and Cellular Senescence via Sirtuin 1 Deacetylase in Chronic Obstructive Pulmonary Disease.Genetic Ablation of p16(Ink4a) Does Not Protect Against Cellular Senescence in Mouse Models of COPD/Emphysema.Analysis of Cell Turnover in the Bronchiolar Epithelium Through the Normal Aging Process.Cigarette Smoke Induces Human Airway Epithelial Senescence via Growth Differentiation Factor 15 Production.Maternal human telomerase reverse transcriptase variants are associated with preterm labor and preterm premature rupture of membranes.Lung cellular senescence is independent of aging in a mouse model of COPD/emphysema.
P2860
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P2860
Epithelial cell senescence impairs repair process and exacerbates inflammation after airway injury.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Epithelial cell senescence imp ...... lammation after airway injury.
@ast
Epithelial cell senescence imp ...... lammation after airway injury.
@en
type
label
Epithelial cell senescence imp ...... lammation after airway injury.
@ast
Epithelial cell senescence imp ...... lammation after airway injury.
@en
prefLabel
Epithelial cell senescence imp ...... lammation after airway injury.
@ast
Epithelial cell senescence imp ...... lammation after airway injury.
@en
P2093
P2860
P356
P1433
P1476
Epithelial cell senescence imp ...... lammation after airway injury.
@en
P2093
Atsushi Nagai
Kazutetsu Aoshiba
Shigemitsu Onizawa
P2860
P2888
P356
10.1186/1465-9921-12-78
P577
2011-06-10T00:00:00Z
P5875
P6179
1031468272