T cell activation-induced mitochondrial hyperpolarization is mediated by Ca2+- and redox-dependent production of nitric oxide.
about
HIV-1 Tat Activates Neuronal Ryanodine Receptors with Rapid Induction of the Unfolded Protein Response and Mitochondrial HyperpolarizationRole of mitochondria in the pheromone- and amiodarone-induced programmed death of yeast.Computational properties of mitochondria in T cell activation and fateBeing right on Q: shaping eukaryotic evolutionPolyglutamine tract-binding protein-1 dysfunction induces cell death of neurons through mitochondrial stressTumour suppressor Fus1 provides a molecular link between inflammatory response and mitochondrial homeostasisTransaldolase is essential for maintenance of the mitochondrial transmembrane potential and fertility of spermatozoa.Systems biology of lupus: mapping the impact of genomic and environmental factors on gene expression signatures, cellular signaling, metabolic pathways, hormonal and cytokine imbalance, and selecting targets for treatment.Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity.Nitric oxide-dependent mitochondrial biogenesis generates Ca2+ signaling profile of lupus T cells.Rapamycin reduces disease activity and normalizes T cell activation-induced calcium fluxing in patients with systemic lupus erythematosus.Oxidative stress in the pathology and treatment of systemic lupus erythematosusHRES-1/Rab4-mediated depletion of Drp1 impairs mitochondrial homeostasis and represents a target for treatment in SLE.T-cell and B-cell signaling biomarkers and treatment targets in lupus.Increased mitochondrial electron transport chain activity at complex I is regulated by N-acetylcysteine in lymphocytes of patients with systemic lupus erythematosusCaspase-3 is transiently activated without cell death during early antigen driven expansion of CD8(+) T cells in vivo.The biology of reactive intermediates in systemic lupus erythematosus.Central role of nitric oxide in the pathogenesis of rheumatoid arthritis and systemic lupus erythematosus.Jacaranone induces apoptosis in melanoma cells via ROS-mediated downregulation of Akt and p38 MAPK activation and displays antitumor activity in vivo.Mechanistic target of rapamycin activation triggers IL-4 production and necrotic death of double-negative T cells in patients with systemic lupus erythematosus.ROS-triggered phosphorylation of complex II by Fgr kinase regulates cellular adaptation to fuel useActivation of mammalian target of rapamycin controls the loss of TCRzeta in lupus T cells through HRES-1/Rab4-regulated lysosomal degradationMitochondrial membrane potential probes and the proton gradient: a practical usage guideThe role of nitric oxide in abnormal T cell signal transduction in systemic lupus erythematosusAdaptive immunity in ankylosing spondylitis: phenotype and functional alterations of T-cells before and during infliximab therapy.Nitric oxide, mitochondrial hyperpolarization, and T cell activation.Targeting T Cell Bioenergetics by Modulating P-Glycoprotein Selectively Depletes Alloreactive T Cells To Prevent Graft-versus-Host Disease.Metabolic regulation of organelle homeostasis in lupus T cellsSustained accumulation of prelamin A and depletion of lamin A/C both cause oxidative stress and mitochondrial dysfunction but induce different cell fateseNOS S-nitrosylates β-actin on Cys374 and regulates PKC-θ at the immune synapse by impairing actin binding to profilin-1.Bioenergetic aspects of apoptosis, necrosis and mitoptosis.Nitrosothiols in the immune system: signaling and protection.Nitric oxide: emerging concepts about its use in cell-based therapies.Endothelial nitric oxide synthase regulates N-Ras activation on the Golgi complex of antigen-stimulated T cellsInduction and stability of human Th17 cells require endogenous NOS2 and cGMP-dependent NO signalingMetabolic control of T cell activation and death in SLE.Prevention of hepatocarcinogenesis and increased susceptibility to acetaminophen-induced liver failure in transaldolase-deficient mice by N-acetylcysteineAssociation of reactive oxygen and nitrogen intermediate and complement levels with apoptosis of peripheral blood mononuclear cells in lupus patientsPharmacotherapy of systemic lupus erythematosus.Fus1/Tusc2 is a novel regulator of mitochondrial calcium handling, Ca2+-coupled mitochondrial processes, and Ca2+-dependent NFAT and NF-κB pathways in CD4+ T cells.
P2860
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P2860
T cell activation-induced mitochondrial hyperpolarization is mediated by Ca2+- and redox-dependent production of nitric oxide.
description
2003 nî lūn-bûn
@nan
2003 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
name
T cell activation-induced mito ...... nt production of nitric oxide.
@ast
T cell activation-induced mito ...... nt production of nitric oxide.
@en
type
label
T cell activation-induced mito ...... nt production of nitric oxide.
@ast
T cell activation-induced mito ...... nt production of nitric oxide.
@en
prefLabel
T cell activation-induced mito ...... nt production of nitric oxide.
@ast
T cell activation-induced mito ...... nt production of nitric oxide.
@en
P2860
P1476
T cell activation-induced mito ...... nt production of nitric oxide.
@en
P2093
Agnes Koncz
Gyorgy Nagy
P2860
P304
P356
10.4049/JIMMUNOL.171.10.5188
P407
P50
P577
2003-11-01T00:00:00Z