Use of cytoplasmic hybrid cell lines for elucidating the role of mitochondrial dysfunction in Alzheimer's disease and Parkinson's disease.
about
Mitochondrial gene therapy augments mitochondrial physiology in a Parkinson's disease cell model.Nuclear genes involved in mitochondria-to-nucleus communication in breast cancer cellsBioenergetic dysfunction and inflammation in Alzheimer's disease: a possible connectionCombotherapy and current concepts as well as future strategies for the treatment of Alzheimer's diseaseCytoplasmic hybrid (cybrid) cell lines as a practical model for mitochondriopathiesMelatonin in Alzheimer's disease and other neurodegenerative disordersThe aetiology of idiopathic Parkinson's diseaseThe cybrid model of sporadic Parkinson's disease.Pathogenesis of Alzheimer's diseaseCytochrome c oxidase deficiency in neurons decreases both oxidative stress and amyloid formation in a mouse model of Alzheimer's diseaseStem cell-based models and therapies for neurodegenerative diseasesMitochondrial mechanisms of estrogen neuroprotection.Lipid- and receptor-binding regions of apolipoprotein E4 fragments act in concert to cause mitochondrial dysfunction and neurotoxicitySystemic mitochondrial dysfunction and the etiology of Alzheimer's disease and down syndrome dementia.Cell and animal models of mtDNA biology: progress and prospects.Apolipoprotein E: structure and function in lipid metabolism, neurobiology, and Alzheimer's diseases.Mitochondrial DNA variants can mediate methylation status of inflammation, angiogenesis and signaling genes.Mitochondria and cell bioenergetics: increasingly recognized components and a possible etiologic cause of Alzheimer's disease.Novel mechanisms for estrogen-induced neuroprotection.The etiopathogenesis of Parkinson disease and suggestions for future research. Part II.Estrogen actions on mitochondria--physiological and pathological implications.Energy matters: mitochondrial proteomics for biomedicine.Platelet mitochondrial function: from regulation of thrombosis to biomarker of disease.Elevated caspase 3 activity and cytosolic cytochrome c in NT2 cybrids containing amyotrophic lateral sclerosis subject mtDNA.A cybrid cell model for the assessment of the link between mitochondrial deficits and sporadic Parkinson's disease.Mitochondria, Cybrids, Aging, and Alzheimer's Disease.Parkinson syndrome as a manifestation of mitochondriopathy.Mitochondrial gene replacement in human pluripotent stem cell-derived neural progenitors.Roles of apolipoprotein E4 (ApoE4) in the pathogenesis of Alzheimer's disease: lessons from ApoE mouse models.Mitochondria and Mitochondrial Cascades in Alzheimer's Disease.Bilobalide protects mitochondrial function in ovariectomized rats by up-regulation of mRNA and protein expression of cytochrome c oxidase subunit I.
P2860
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P2860
Use of cytoplasmic hybrid cell lines for elucidating the role of mitochondrial dysfunction in Alzheimer's disease and Parkinson's disease.
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Use of cytoplasmic hybrid cell ...... sease and Parkinson's disease.
@ast
Use of cytoplasmic hybrid cell ...... sease and Parkinson's disease.
@en
type
label
Use of cytoplasmic hybrid cell ...... sease and Parkinson's disease.
@ast
Use of cytoplasmic hybrid cell ...... sease and Parkinson's disease.
@en
prefLabel
Use of cytoplasmic hybrid cell ...... sease and Parkinson's disease.
@ast
Use of cytoplasmic hybrid cell ...... sease and Parkinson's disease.
@en
P2093
P2860
P1476
Use of cytoplasmic hybrid cell ...... sease and Parkinson's disease.
@en
P2093
P2860
P304
P356
10.1111/J.1749-6632.1999.TB07825.X
P407
P433
1 OXIDATIVE/ENE
P577
1999-01-01T00:00:00Z