Apoptotic neutrophils augment the inflammatory response to Mycobacterium tuberculosis infection in human macrophages. - Wikidata - wikimedia - TriplyDB

Apoptotic neutrophils augment the inflammatory response to Mycobacterium tuberculosis infection in human macrophages.

Apoptotic neutrophils augment the inflammatory response to Mycobacterium tuberculosis infection in human macrophages.

http://www.wikidata.org/entity/Q33855500

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Innate Immunity Holding the Flanks until Reinforced by Adaptive Immunity against Mycobacterium tuberculosis Infection Differential transcriptional response in macrophages infected with cell wall deficient versus normal Mycobacterium Tuberculosis.Extracellular traps and macrophages: new roles for the versatile phagocyte.The Numerical Predominance and Large Transcriptome Differences of Neutrophils in Peripheral Blood Together Inevitably Account for a Reported Pulmonary Tuberculosis Signature.Interactions between Type 1 Interferons and the Th17 Response in Tuberculosis: Lessons Learned from Autoimmune Diseases.Reinforcing the Functionality of Mononuclear Phagocyte System to Control Tuberculosis.MyD88 in Mycobacterium tuberculosis infection.

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Apoptotic neutrophils augment the inflammatory response to Mycobacterium tuberculosis infection in human macrophages.

http://www.wikidata.org/entity/Q33855500

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2014 nî lūn-bûn

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2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2014 թվականի հուլիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

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Apoptotic neutrophils augment ...... nfection in human macrophages.

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Apoptotic neutrophils augment ...... nfection in human macrophages.

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Apoptotic neutrophils augment ...... nfection in human macrophages.

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Apoptotic neutrophils augment ...... nfection in human macrophages.

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Apoptotic neutrophils augment ...... nfection in human macrophages.

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Apoptotic neutrophils augment ...... nfection in human macrophages.

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Blanka Andersson

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P2860

Regulation of inflammasome signaling

Alternative activation of macrophages: mechanism and functions

Inhibition of respiration by nitric oxide induces a Mycobacterium tuberculosis dormancy program

Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

Cell death: the significance of apoptosis

Human macrophages infected with a high burden of ESAT-6-expressing M. tuberculosis undergo caspase-1- and cathepsin B-independent necrosis

Tumor necrosis factor alpha stimulates killing of Mycobacterium tuberculosis by human neutrophils.

Hijacking and exploitation of IL-10 by intracellular pathogens.

Development of luminescent Mycobacterium avium subsp. paratuberculosis for rapid screening of vaccine candidates in mice

Mycobacterium tuberculosis and the intimate discourse of a chronic infection.

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e101514

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Alexander Persson

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Mycobacterium tuberculosis

apoptotic process

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