Anti-high mobility group box-1 monoclonal antibody protects the blood-brain barrier from ischemia-induced disruption in rats.
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Pathobiology of tobacco smoking and neurovascular disorders: untied strings and alternative productsThe Yin and Yang of innate immunity in strokeBiomarkers and molecular probes for cell death imaging and targeted therapeuticsHigh-mobility group box 1: an amplifier of stem and progenitor cell activity after strokePGE2 receptor agonist misoprostol protects brain against intracerebral hemorrhage in miceCerebroprotection by the neuronal PGE2 receptor EP2 after intracerebral hemorrhage in middle-aged miceAnti-high mobility group box-1 (HMGB1) antibody attenuates delayed cerebral vasospasm and brain injury after subarachnoid hemorrhage in ratsTherapeutic effects of anti-HMGB1 monoclonal antibody on pilocarpine-induced status epilepticus in mice.Neuropathic pain in rats with a partial sciatic nerve ligation is alleviated by intravenous injection of monoclonal antibody to high mobility group box-1Pathophysiology of endotoxin tolerance: mechanisms and clinical consequences.Toll-like receptors and ischemic brain injury.Surgery upregulates high mobility group box-1 and disrupts the blood-brain barrier causing cognitive dysfunction in aged rats.Altered protein networks and cellular pathways in severe west nile disease in mice.Purinergic control of vascular tone in the retina.Prolonged neuroinflammation after lipopolysaccharide exposure in aged rats.Anti-inflammatory role of fetuin-A in injury and infection.Acetylcholinesterase loosens the brain's cholinergic anti-inflammatory response and promotes epileptogenesis.Identification of a functional interaction of HMGB1 with Receptor for Advanced Glycation End-products in a model of neuropathic pain.Anti-IL-6 neutralizing antibody modulates blood-brain barrier function in the ovine fetus.Does brain inflammation mediate pathological outcomes in epilepsy?HMGB1 and thrombin mediate the blood-brain barrier dysfunction acting as biomarkers of neuroinflammation and progression to neurodegeneration in Alzheimer's disease.Potential of the angiotensin receptor blockers (ARBs) telmisartan, irbesartan, and candesartan for inhibiting the HMGB1/RAGE axis in prevention and acute treatment of stroke.HDAC4/5-HMGB1 signalling mediated by NADPH oxidase activity contributes to cerebral ischaemia/reperfusion injury.Inhibiting HMGB1 Reduces Cerebral Ischemia Reperfusion Injury in Diabetic MiceEthanol extracts from Portulaca oleracea L. attenuated ischemia/reperfusion induced rat neural injury through inhibition of HMGB1 induced inflammation.Toll-like receptor signaling adapter proteins govern spread of neuropathic pain and recovery following nerve injury in male mice.Toll-like receptor signaling regulates cisplatin-induced mechanical allodynia in miceOne-Compound-Multi-Target: Combination Prospect of Natural Compounds with Thrombolytic Therapy in Acute Ischemic Stroke.IL-17 contributed to the neuropathic pain following peripheral nerve injury by promoting astrocyte proliferation and secretion of proinflammatory cytokines.Post-ischemic inflammation in the brain.Herbal medicines for ischemic stroke: combating inflammation as therapeutic targets.Designing drugs that encourage spinal cord injury healing.Post-ischemic inflammation regulates neural damage and protection.The role of IL-17 in CNS diseases.Enriched housing promotes post-stroke functional recovery through astrocytic HMGB1-IL-6-mediated angiogenesis.Role of high-mobility group box-1 protein in disruption of vascular barriers and regulation of leukocyte-endothelial interactions.Unconventional Pathways of Secretion Contribute to Inflammation.Novel Regenerative Therapies Based on Regionally Induced Multipotent Stem Cells in Post-Stroke Brains: Their Origin, Characterization, and Perspective.HMGB1 Translocation After Ischemia in the Ovine Fetal Brain.HMGB1/anti-HMGB1 antibodies define a molecular signature of early stages of HIV-Associated Neurocognitive Isorders (HAND).
P2860
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P2860
Anti-high mobility group box-1 monoclonal antibody protects the blood-brain barrier from ischemia-induced disruption in rats.
description
2011 nî lūn-bûn
@nan
2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Anti-high mobility group box-1 ...... ia-induced disruption in rats.
@ast
Anti-high mobility group box-1 ...... ia-induced disruption in rats.
@en
type
label
Anti-high mobility group box-1 ...... ia-induced disruption in rats.
@ast
Anti-high mobility group box-1 ...... ia-induced disruption in rats.
@en
prefLabel
Anti-high mobility group box-1 ...... ia-induced disruption in rats.
@ast
Anti-high mobility group box-1 ...... ia-induced disruption in rats.
@en
P2093
P1433
P1476
Anti-high mobility group box-1 ...... mia-induced disruption in rats
@en
P2093
Aiji Ohtsuka
Hidenori Wake
Jiyong Zhang
Masahiro Nishibori
Shuji Mori
Tadashi Yoshino
Tomoko Maruo
P304
P356
10.1161/STROKEAHA.110.598334
P407
P577
2011-04-07T00:00:00Z