Blockade of high mobility group box-1 protein attenuates experimental severe acute pancreatitis. - Wikidata - wikimedia - TriplyDB

Blockade of high mobility group box-1 protein attenuates experimental severe acute pancreatitis.

Blockade of high mobility group box-1 protein attenuates experimental severe acute pancreatitis.

http://www.wikidata.org/entity/Q33868755

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Cell death and DAMPs in acute pancreatitis Sterile inflammatory response in acute pancreatitis Inflammasomes in pancreatic physiology and disease Ethyl pyruvate is a novel anti-inflammatory agent to treat multiple inflammatory organ injuries Targeting HMGB1 in inflammation Association between high mobility group box‑1 protein expression and cell death in acute pancreatitis Anti-inflammatory effect of sodium butyrate preconditioning during myocardial ischemia/reperfusion.Novel HMGB1-inhibiting therapeutic agents for experimental sepsis.High mobility group box 1 protein as a potential drug target for infection- and injury-elicited inflammation.Overexpression of receptor for advanced glycation end products and high-mobility group box 1 in human dental pulp inflammation.High-mobility group box 1 released from astrocytes promotes the proliferation of cultured neural stem/progenitor cells.DAMPening inflammation by modulating TLR signalling.Combination of allopurinol and hyperbaric oxygen therapy: a new treatment in experimental acute necrotizing pancreatitis?HMGB1 contributes to kidney ischemia reperfusion injury.HMGB1 in health and disease.High-mobility group box 1 protein and its role in severe acute pancreatitis.TLR9 and the NLRP3 inflammasome link acinar cell death with inflammation in acute pancreatitis.Redox signaling in acute pancreatitis.Glycyrrhizin suppresses the expressions of HMGB1 and relieves the severity of traumatic pancreatitis in rats.TLR4-HMGB1-, MyD88- and TRIF-dependent signaling in mouse intestinal ischemia/reperfusion injury.Spatial and temporal differences of HMGB1 expression in the pancreas of rats with acute pancreatitis HMGB1 is a therapeutic target for sterile inflammation and infection.TLR4-mediated NF-κB signaling pathway mediates HMGB1-induced pancreatic injury in mice with severe acute pancreatitis.Scolopendra subspinipes mutilans protected the cerulein-induced acute pancreatitis by inhibiting high-mobility group box protein-1.Bench-to-bedside review: High-mobility group box 1 and critical illness.Therapeutic potential of HMGB1-targeting agents in sepsis.Potential of the angiotensin receptor blockers (ARBs) telmisartan, irbesartan, and candesartan for inhibiting the HMGB1/RAGE axis in prevention and acute treatment of stroke.Relationship between vascular reactivity and expression of HMGB1 in a rat model of septic aorta.Delayed ethyl pyruvate therapy attenuates experimental severe acute pancreatitis via reduced serum high mobility group box 1 levels in rats.HMGB1 translocation and release mediate cigarette smoke-induced pulmonary inflammation in mice through a TLR4/MyD88-dependent signaling pathway.Intracellular Hmgb1 inhibits inflammatory nucleosome release and limits acute pancreatitis in mice.The RAGE axis in systemic inflammation, acute lung injury and myocardial dysfunction: an important therapeutic target?Cutaneous lupus erythematosus: recent lessons from animal models.Role of Apoptosis in Amplifying Inflammatory Responses in Lung Diseases Sterile inflammation in the liver and pancreas.The spectrum of anti-chromatin/nucleosome autoantibodies: independent and interdependent biomarkers of disease.High-mobility group box-1 in sterile inflammation.Immune-modulating therapy in acute pancreatitis: fact or fiction.Circulating nucleosomes as predictive markers of severe acute pancreatitis.Blockade of high-mobility group box 1 attenuates intestinal mucosal barrier dysfunction in experimental acute pancreatitis.

P2860

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P2860

Blockade of high mobility group box-1 protein attenuates experimental severe acute pancreatitis.

http://www.wikidata.org/entity/Q33868755

description

2006 nî lūn-bûn

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2006 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2006 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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Blockade of high mobility grou ...... tal severe acute pancreatitis.

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Blockade of high mobility grou ...... tal severe acute pancreatitis.

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Blockade of high mobility grou ...... tal severe acute pancreatitis.

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prefLabel

Blockade of high mobility grou ...... tal severe acute pancreatitis.

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Blockade of high mobility grou ...... tal severe acute pancreatitis.

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P2093

Hidehiro Sawa

http://www.w3.org/2001/XMLSchema#string

Makoto Shinzeki

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Takahiro Nakajima

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Takashi Ueda

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Takeo Yasuda

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Yoshifumi Takeyama

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Yoshikazu Kuroda

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P2860

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

A human homologue of the Drosophila Toll protein signals activation of adaptive immunity

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

HMG-1 as a late mediator of endotoxin lethality in mice

Mini-review: The nuclear protein HMGB1 as a proinflammatory mediator

The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

High mobility group 1 protein (HMG-1) stimulates proinflammatory cytokine synthesis in human monocytes

Regulation of DNA-dependent activities by the functional motifs of the high-mobility-group chromosomal proteins.

Reversing established sepsis with antagonists of endogenous high-mobility group box 1.

High mobility group box 1 (HMGB1).

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