HMGB1 contributes to kidney ischemia reperfusion injury. - Wikidata - wikimedia - TriplyDB

HMGB1 contributes to kidney ischemia reperfusion injury.

HMGB1 contributes to kidney ischemia reperfusion injury.

http://www.wikidata.org/entity/Q34457038

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The role of high mobility group box 1 (HMGB1) in the pathogenesis of kidney diseases Guidance cue netrin-1 and the regulation of inflammation in acute and chronic kidney disease Oxidative stress-mediated HMGB1 biology Novel Mechanisms of Herbal Therapies for Inhibiting HMGB1 Secretion or Action How the Innate Immune System Senses Trouble and Causes Trouble The renal mononuclear phagocytic system Macrophage in chronic kidney disease The HMGB1-RAGE Inflammatory Pathway: Implications for Brain Injury-Induced Pulmonary Dysfunction Dipeptidyl peptidase IV inhibitor protects against renal interstitial fibrosis in a mouse model of ureteral obstruction Therapeutic inflammatory monocyte modulation using immune-modifying microparticles Oxidant Mechanisms in Renal Injury and Disease The spleen contributes importantly to myocardial infarct exacerbation during post-ischemic reperfusion in mice via signaling between cardiac HMGB1 and splenic RAGE.Melatonin promoted renal regeneration in folic acid-induced acute kidney injury via inhibiting nucleocytoplasmic translocation of HMGB1 in tubular epithelial cells.TLR4 activation promotes podocyte injury and interstitial fibrosis in diabetic nephropathy.Molecular mechanism and therapeutic modulation of high mobility group box 1 release and action: an updated review.Biglycan-triggered TLR-2- and TLR-4-signaling exacerbates the pathophysiology of ischemic acute kidney injury.The role of SDF-1-CXCR4/CXCR7 axis in the therapeutic effects of hypoxia-preconditioned mesenchymal stem cells for renal ischemia/reperfusion injury Placental growth factor, pregnancy-associated plasma protein-A, soluble receptor for advanced glycation end products, extracellular newly identified receptor for receptor for advanced glycation end products binding protein and high mobility group bo Proximal tubule epithelial cell specific ablation of the spermidine/spermine N1-acetyltransferase gene reduces the severity of renal ischemia/reperfusion injury HMGB1 in health and disease.HMGB1 exacerbates renal tubulointerstitial fibrosis through facilitating M1 macrophage phenotype at the early stage of obstructive injury.The alarmin concept applied to human renal transplantation: evidence for a differential implication of HMGB1 and IL-33 Differential activation of Toll-like receptor-mediated apoptosis induced by hypoxia Cisplatin induces primary necrosis through poly(ADP-ribose) polymerase 1 activation in kidney proximal tubular cells The high-mobility group box-1 nuclear factor mediates retinal injury after ischemia reperfusion.Circulating Mitochondrial DAMPs Are Not Effective Inducers of Proteinuria and Kidney Injury in Rodents Serum-stabilized naked caspase-3 siRNA protects autotransplant kidneys in a porcine model.An update on the role of the inflammasomes in the pathogenesis of kidney diseases.Toll-like receptor 4 promotes tubular inflammation in diabetic nephropathy.HCaRG accelerates tubular repair after ischemic kidney injury Interaction between uric acid and HMGB1 translocation and release from endothelial cells.Innate immunity in donor procurement.HMGB1 promotes the activation of NLRP3 and caspase-8 inflammasomes via NF-κB pathway in acute glaucoma.HMGB1 is activated in type 2 diabetes mellitus patients and in mesangial cells in response to high glucose An ongoing search for potential targets and therapies for lethal sepsis Early interleukin 6 production by leukocytes during ischemic acute kidney injury is regulated by TLR4.Complement activation and toll-like receptor-2 signaling contribute to cytokine production after renal ischemia/reperfusion.Absence of MyD88 signaling induces donor-specific kidney allograft tolerance HMGB1 in renal ischemic injury.Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosis

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HMGB1 contributes to kidney ischemia reperfusion injury.

http://www.wikidata.org/entity/Q34457038

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

HMGB1 contributes to kidney ischemia reperfusion injury.

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HMGB1 contributes to kidney ischemia reperfusion injury.

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HMGB1 contributes to kidney ischemia reperfusion injury.

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type

label

HMGB1 contributes to kidney ischemia reperfusion injury.

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HMGB1 contributes to kidney ischemia reperfusion injury.

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HMGB1 contributes to kidney ischemia reperfusion injury.

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prefLabel

HMGB1 contributes to kidney ischemia reperfusion injury.

@ast

HMGB1 contributes to kidney ischemia reperfusion injury.

@en

HMGB1 contributes to kidney ischemia reperfusion injury.

@nl

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P1433

Journal of the American Society of Nephrology

P1476

HMGB1 contributes to kidney ischemia reperfusion injury.

@en

P2093

Huiling Wu

http://www.w3.org/2001/XMLSchema#string

Jin Ma

http://www.w3.org/2001/XMLSchema#string

Kate R Wyburn

http://www.w3.org/2001/XMLSchema#string

Peng Wang

http://www.w3.org/2001/XMLSchema#string

Steven J Chadban

http://www.w3.org/2001/XMLSchema#string

Theresa M Corpuz

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Usha Panchapakesan

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P2860

Signaling danger: toll-like receptors and their potential roles in kidney disease

A human homologue of the Drosophila Toll protein signals activation of adaptive immunity

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

HMG-1 as a late mediator of endotoxin lethality in mice

The danger model: a renewed sense of self

High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal

Decoding the patterns of self and nonself by the innate immune system

Toll-like receptor 9-dependent activation by DNA-containing immune complexes is mediated by HMGB1 and RAGE

Toll-like receptor-4 coordinates the innate immune response of the kidney to renal ischemia/reperfusion injury.

The N-terminal domain of thrombomodulin sequesters high-mobility group-B1 protein, a novel antiinflammatory mechanism.

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1878-1890

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10.1681/ASN.2009101048

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11

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21

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2010-09-16T00:00:00Z

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20847143

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reperfusion injury

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3014003

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