Activation of MAPK pathways due to DUSP4 loss promotes cancer stem cell-like phenotypes in basal-like breast cancer - Wikidata - wikimedia - TriplyDB

Activation of MAPK pathways due to DUSP4 loss promotes cancer stem cell-like phenotypes in basal-like breast cancer

Activation of MAPK pathways due to DUSP4 loss promotes cancer stem cell-like phenotypes in basal-like breast cancer

http://www.wikidata.org/entity/Q33875120

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Anti-Cancer Stem-like Cell Compounds in Clinical Development - An Overview and Critical Appraisal Midbody: from cellular junk to regulator of cell polarity and cell fate Melanoma-specific MHC-II expression represents a tumour-autonomous phenotype and predicts response to anti-PD-1/PD-L1 therapy.Sanguinarine inhibits growth and invasion of gastric cancer cells via regulation of the DUSP4/ERK pathway.Triple-negative breast cancer: challenges and opportunities of a heterogeneous disease.YB-1 transforms human mammary epithelial cells through chromatin remodeling leading to the development of basal-like breast cancer Protein kinase C-δ inactivation inhibits the proliferation and survival of cancer stem cells in culture and in vivo DUSP4 deficiency caused by promoter hypermethylation drives JNK signaling and tumor cell survival in diffuse large B cell lymphoma Molecular signatures of sanguinarine in human pancreatic cancer cells: A large scale label-free comparative proteomics approach.Role of a polyphenol-enriched preparation on chemoprevention of mammary carcinoma through cancer stem cells and inflammatory pathways modulation Tumor Cell-Derived Periostin Regulates Cytokines That Maintain Breast Cancer Stem Cells.RAS/MAPK Activation Is Associated with Reduced Tumor-Infiltrating Lymphocytes in Triple-Negative Breast Cancer: Therapeutic Cooperation Between MEK and PD-1/PD-L1 Immune Checkpoint Inhibitors DUSP4/MKP2 overexpression is associated with BRAF(V600E) mutation and aggressive behavior of papillary thyroid cancer.Analysis of phosphatases in ER-negative breast cancers identifies DUSP4 as a critical regulator of growth and invasion Statins affect ETS1-overexpressing triple-negative breast cancer cells by restoring DUSP4 deficiency Global Prioritizing Disease Candidate lncRNAs via a Multi-level Composite Network.Role of TGF-β in breast cancer bone metastases.Triple-negative breast cancers with amplification of JAK2 at the 9p24 locus demonstrate JAK2-specific dependence.Metalloprotease-disintegrin ADAM12 actively promotes the stem cell-like phenotype in claudin-low breast cancer Molecular profiling of the residual disease of triple-negative breast cancers after neoadjuvant chemotherapy identifies actionable therapeutic targets.FAM83D associates with high tumor recurrence after liver transplantation involving expansion of CD44+ carcinoma stem cells.MicroRNA-Based Therapeutic Strategies for Targeting Mutant and Wild Type RAS in Cancer In silico evaluation of DNA Damage Inducible Transcript 4 gene (DDIT4) as prognostic biomarker in several malignancies.Altered purinergic receptor-Ca²⁺ signaling associated with hypoxia-induced epithelial-mesenchymal transition in breast cancer cells.Approaches for targeting cancer stem cells drug resistance.Molecular Pathways: Targeting Protein Tyrosine Phosphatases in Cancer.Agonist immunotherapy restores T cell function following MEK inhibition improving efficacy in breast cancer.High Myc expression and transcription activity underlies intra-tumoral heterogeneity in triple-negative breast cancer.Metalloprotease-dependent activation of EGFR modulates CD44+/CD24- populations in triple negative breast cancer cells through the MEK/ERK pathway.Tracing Information Flow from Erk to Target Gene Induction Reveals Mechanisms of Dynamic and Combinatorial Control.Mineral trioxide aggregate enhances the osteogenic capacity of periodontal ligament stem cells via NF-κB and MAPK signaling pathways.Evidence for embryonic stem-like signature and epithelial-mesenchymal transition features in the spheroid cells derived from lung adenocarcinoma.Hypoxia-regulated gene network in drug resistance and cancer progression.Ribosome display and selection of single-chain variable fragments effectively inhibit growth and progression of microspheres in vitro and in vivo.Melanoma response to anti-PD-L1 immunotherapy requires JAK1 signaling, but not JAK2.MEK inhibitors for the treatment of NRAS mutant melanoma CTCF maintains regulatory homeostasis of cancer pathways Therapeutic landscape in mutational triple negative breast cancer Identification of Differentially Expressed Non-coding RNA in Porcine Alveolar Macrophages from Tongcheng and Large White Pigs Responded to PRRSV Synthesis and biological evaluation of dithiocarbamate esters of parthenolide as potential anti-acute myelogenous leukaemia agents

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Activation of MAPK pathways due to DUSP4 loss promotes cancer stem cell-like phenotypes in basal-like breast cancer

http://www.wikidata.org/entity/Q33875120

description

2013 nî lūn-bûn

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2013 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի օգոստոսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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Activation of MAPK pathways du ...... es in basal-like breast cancer

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Activation of MAPK pathways du ...... es in basal-like breast cancer

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Activation of MAPK pathways du ...... es in basal-like breast cancer

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Activation of MAPK pathways du ...... es in basal-like breast cancer

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0008-5472.CAN-13-1385

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Cancer Research

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Activation of MAPK pathways du ...... es in basal-like breast cancer

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Carlos L Arteaga

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Henry Gómez

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Luis J Schwarz

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Neil E Bhola

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Phillip Owens

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Rebecca S Cook

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Richard Kurupi

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Todd W Miller

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P2860

Oncogenic Ras and B-Raf proteins positively regulate death receptor 5 expression through co-activation of ERK and JNK signaling

Comprehensive molecular portraits of human breast tumours

CXCR1 blockade selectively targets human breast cancer stem cells in vitro and in xenografts

Aldehyde dehydrogenase 1 is a marker for normal and malignant human colonic stem cells (SC) and tracks SC overpopulation during colon tumorigenesis

A collection of breast cancer cell lines for the study of functionally distinct cancer subtypes

The epithelial-mesenchymal transition generates cells with properties of stem cells

Tumor self-seeding by circulating cancer cells

IL-6 triggers malignant features in mammospheres from human ductal breast carcinoma and normal mammary gland

Prospective identification of tumorigenic breast cancer cells

The MAPPER database: a multi-genome catalog of putative transcription factor binding sites

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20

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2013-08-21T00:00:00Z

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23966295

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4090144

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