Disruption of sarcolemmal ATP-sensitive potassium channel activity impairs the cardiac response to systolic overload.
about
PGC-1 proteins and heart failureKATP channels and cardiovascular disease: suddenly a syndromeDystrophin is required for the normal function of the cardio-protective K(ATP) channel in cardiomyocytesEffects of hypertension and exercise on cardiac proteome remodellingMaintaining PGC-1α expression following pressure overload-induced cardiac hypertrophy preserves angiogenesis but not contractile or mitochondrial function.Oxidative stress regulates left ventricular PDE5 expression in the failing heartEndoplasmic reticulum stress sensor protein kinase R-like endoplasmic reticulum kinase (PERK) protects against pressure overload-induced heart failure and lung remodeling.Preconditioning by isoflurane elicits mitochondrial protective mechanisms independent of sarcolemmal KATP channel in mouse cardiomyocytes.Exercise-induced expression of cardiac ATP-sensitive potassium channels promotes action potential shortening and energy conservation.Muscle KATP channels: recent insights to energy sensing and myoprotectionAMP activated protein kinase-α2 regulates expression of estrogen-related receptor-α, a metabolic transcription factor related to heart failure development.Measuring and evaluating the role of ATP-sensitive K+ channels in cardiac muscle.KATP Channels in the Cardiovascular System.Induced overexpression of Na(+)/Ca(2+) exchanger does not aggravate myocardial dysfunction induced by transverse aortic constriction.The role of ATP-sensitive potassium channels in cellular function and protection in the cardiovascular systemCardiac sarcolemmal K(ATP) channels: Latest twists in a questing tale!Adenosine Triphosphate-Sensitive Potassium Currents in Heart Disease and Cardioprotection.Metabolic remodelling in hypertrophied and failing myocardium: a review.Selenium attenuates adriamycin-induced cardiac dysfunction via restoring expression of ATP-sensitive potassium channels in rats.PGC-1 alpha regulates expression of myocardial mitochondrial antioxidants and myocardial oxidative stress after chronic systolic overload.Peroxisome proliferator-activated receptor-γ coactivator 1 α1 induces a cardiac excitation-contraction coupling phenotype without metabolic remodelling.
P2860
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P2860
Disruption of sarcolemmal ATP-sensitive potassium channel activity impairs the cardiac response to systolic overload.
description
2008 nî lūn-bûn
@nan
2008 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Disruption of sarcolemmal ATP- ...... response to systolic overload.
@ast
Disruption of sarcolemmal ATP- ...... response to systolic overload.
@en
type
label
Disruption of sarcolemmal ATP- ...... response to systolic overload.
@ast
Disruption of sarcolemmal ATP- ...... response to systolic overload.
@en
prefLabel
Disruption of sarcolemmal ATP- ...... response to systolic overload.
@ast
Disruption of sarcolemmal ATP- ...... response to systolic overload.
@en
P2093
P2860
P1433
P1476
Disruption of sarcolemmal ATP- ...... response to systolic overload
@en
P2093
Colin G Nichols
Robert J Bache
Thomas P Flagg
Yimin Huang
Ying Zhang
P2860
P304
P356
10.1161/CIRCRESAHA.107.170795
P577
2008-09-18T00:00:00Z