The v-mos and H-ras oncogene expression represses glucocorticoid hormone-dependent transcription from the mouse mammary tumor virus LTR.
about
Mutual transrepression of Fos and the glucocorticoid receptor: involvement of a functional domain in Fos which is absent in FosBAlpha B-crystallin expression in mouse NIH 3T3 fibroblasts: glucocorticoid responsiveness and involvement in thermal protectionPartial reversion of the transformed phenotype in HRAS-transfected tumorigenic cells by transfer of a human gene.Glucocorticoid and cAMP induction mechanisms are differentially affected by the p85gag-mos oncoprotein.Expression of a constitutive form of calcium/calmodulin dependent protein kinase II leads to arrest of the cell cycle in G2Serum- and oncoprotein-mediated induction of a gene with sequence similarity to the gene encoding carcinoembryonic antigenProlactin regulation of beta-casein gene expression and of a cytosolic 120-kd protein in a cloned mouse mammary epithelial cell line.Dexamethasone-dependent inhibition of differentiation of C2 myoblasts bearing steroid-inducible N-ras oncogenes.An activated allele of the c-erbB-2 oncogene impairs kidney and lung function and causes early death of transgenic mice.Mitogen-activated and cyclin-dependent protein kinases selectively and differentially modulate transcriptional enhancement by the glucocorticoid receptorThe dual-specificity CLK kinase induces neuronal differentiation of PC12 cells.Alpha B crystallin accumulation is a specific response to Ha-ras and v-mos oncogene expression in mouse NIH 3T3 fibroblasts.Overexpression of Mos, Ras, Src, and Fos inhibits mouse mammary epithelial cell differentiation.Expression of v-src in embryonic neural retina alters cell adhesion, inhibits histogenesis, and prevents induction of glutamine synthetase.Oncogene N-ras mediates selective inhibition of c-fos induction by nerve growth factor and basic fibroblast growth factor in a PC12 cell lineInduction of a mitogen-responsive gene after expression of the Ha-ras oncogene in NIH 3T3 fibroblastsAlpha B-crystallin is a small heat shock protein.PEA3 is a nuclear target for transcription activation by non-nuclear oncogenesConstruction and characterization of a hybrid mouse mammary tumor virus/murine leukemia virus-based retroviral vector.Two dominant inhibitory mutants of p21ras interfere with insulin-induced gene expression.Possible involvement of normal p21 H-ras in the insulin/insulinlike growth factor 1 signal transduction pathway.Desensitization of the Ca2+-mobilizing system to serum growth factors by Ha-ras and v-mos.The cellular response to induction of the p21 c-Ha-ras oncoprotein includes stimulation of jun gene expression.Reversible abrogation of IL-3 dependence by an inducible H-ras oncogene.Cell-specific inhibitory and stimulatory effects of Fos and Jun on transcription activation by nuclear receptorsCell cycle regulation of glucocorticoid receptor function.
P2860
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P2860
The v-mos and H-ras oncogene expression represses glucocorticoid hormone-dependent transcription from the mouse mammary tumor virus LTR.
description
1986 nî lūn-bûn
@nan
1986 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1986 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1986年の論文
@ja
1986年論文
@yue
1986年論文
@zh-hant
1986年論文
@zh-hk
1986年論文
@zh-mo
1986年論文
@zh-tw
1986年论文
@wuu
name
The v-mos and H-ras oncogene e ...... mouse mammary tumor virus LTR.
@ast
The v-mos and H-ras oncogene e ...... mouse mammary tumor virus LTR.
@en
type
label
The v-mos and H-ras oncogene e ...... mouse mammary tumor virus LTR.
@ast
The v-mos and H-ras oncogene e ...... mouse mammary tumor virus LTR.
@en
prefLabel
The v-mos and H-ras oncogene e ...... mouse mammary tumor virus LTR.
@ast
The v-mos and H-ras oncogene e ...... mouse mammary tumor virus LTR.
@en
P2093
P2860
P1433
P1476
The v-mos and H-ras oncogene e ...... mouse mammary tumor virus LTR
@en
P2093
P2860
P304
P356
10.1002/J.1460-2075.1986.TB04541.X
P407
P577
1986-10-01T00:00:00Z