The cellular response to induction of the p21 c-Ha-ras oncoprotein includes stimulation of jun gene expression.
about
RREB-1, a novel zinc finger protein, is involved in the differentiation response to Ras in human medullary thyroid carcinomasActivation of extracellular signal-regulated kinase, ERK2, by p21ras oncoproteinTranscriptional activation of a conserved sequence element by ras requires a nuclear factor distinct from c-fos or c-junTranscription factor Sp3 antagonizes activation of the ornithine decarboxylase promoter by Sp1.PML is induced by oncogenic ras and promotes premature senescenceLack of a role for Jun kinase and AP-1 in Fas-induced apoptosis.Alpha B crystallin accumulation is a specific response to Ha-ras and v-mos oncogene expression in mouse NIH 3T3 fibroblasts.Regulation of collagen I gene expression by rasc-Ha-ras down regulates the alpha-fetoprotein gene but not the albumin gene in human hepatoma cells.Initiation of transcription from the minute virus of mice P4 promoter is stimulated in rat cells expressing a c-Ha-ras oncogene.Transformation and transactivation suppressor activity of the c-Jun leucine zipper fused to a bacterial repressor.Induction of p38δ expression plays an essential role in oncogenic ras-induced senescencePU.1 and partners: regulation of haematopoietic stem cell fate in normal and malignant haematopoiesis.RAR-specific agonist/antagonists which dissociate transactivation and AP1 transrepression inhibit anchorage-independent cell proliferation.MHC class I gene expression is negatively regulated by the proto-oncogene, c-jun.PEA3 is a nuclear target for transcription activation by non-nuclear oncogenesOrnithine decarboxylase- and ras-induced cell transformations: reversal by protein tyrosine kinase inhibitors and role of pp130CAS.Stepwise transformation of rat embryo fibroblasts: c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization.Transcription factor PEA3 participates in the induction of urokinase plasminogen activator transcription in murine keratinocytes stimulated with epidermal growth factor or phorbol-ester.Differential effects of the adenovirus E1A oncogene on members of the AP-1 transcription factor family.Regulation of the junB gene by v-src.Polyamines are essential for cell transformation by pp60v-src: delineation of molecular events relevant for the transformed phenotypep21ras and protein kinase C function in distinct and interdependent signaling pathways in C3H 10T1/2 fibroblasts.Activation of the neu tyrosine kinase induces the fos/jun transcription factor complex, the glucose transporter and ornithine decarboxylase.Depletion of c-myc with specific antisense sequences reverses the transformed phenotype in ras oncogene-transformed NIH 3T3 cells.Identification of a novel interleukin-6 response element containing an Ets-binding site and a CRE-like site in the junB promoter.c-Ha-rasVal 12 oncogene-transformed NIH-3T3 fibroblasts display more decondensed nucleosomal organization than normal fibroblasts.
P2860
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P2860
The cellular response to induction of the p21 c-Ha-ras oncoprotein includes stimulation of jun gene expression.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
1989年论文
@zh
1989年论文
@zh-cn
name
The cellular response to induc ...... lation of jun gene expression.
@en
type
label
The cellular response to induc ...... lation of jun gene expression.
@en
prefLabel
The cellular response to induc ...... lation of jun gene expression.
@en
P2093
P2860
P1433
P1476
The cellular response to induc ...... lation of jun gene expression.
@en
P2093
P2860
P304
P407
P577
1989-03-01T00:00:00Z