Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice. - Wikidata - wikimedia - TriplyDB

Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.

Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.

http://www.wikidata.org/entity/Q33894781

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Herpes simplex virus and varicella zoster virus, the house guests who never leave Targeted DNA mutagenesis for the cure of chronic viral infections Spontaneous reactivation of herpes simplex virus type 1 in latently infected murine sensory ganglia Selective retention of herpes simplex virus-specific T cells in latently infected human trigeminal ganglia Evidence that spontaneous reactivation of herpes virus does not occur in mice Herpes Simplex Virus Type 1 and Other Pathogens are Key Causative Factors in Sporadic Alzheimer's Disease Potential function of miRNAs in herpetic stromal keratitis Bovine Herpes Virus 1 (BHV-1) and Herpes Simplex Virus Type 1 (HSV-1) Promote Survival of Latently Infected Sensory Neurons, in Part by Inhibiting Apoptosis The molecular basis of herpes simplex virus latency Ocular herpes simplex virus: how are latency, reactivation, recurrent disease and therapy interrelated?Local CD4 and CD8 T-cell reactivity to HSV-1 antigens documents broad viral protein expression and immune competence in latently infected human trigeminal ganglia Transient reversal of episome silencing precedes VP16-dependent transcription during reactivation of latent HSV-1 in neurons Characterization of the IFN-gamma T-cell responses to immediate early antigens in humans with genital herpes Presence of HSV-1 immediate early genes and clonally expanded T-cells with a memory effector phenotype in human trigeminal ganglia.Role of nuclear factor Y in stress-induced activation of the herpes simplex virus type 1 ICP0 promoter The stability of herpes simplex virus 1 ICP0 early after infection is defined by the RING finger and the UL13 protein kinase.Towards an understanding of the herpes simplex virus type 1 latency-reactivation cycle Herpes simplex type I (HSV-1) infection of the nervous system: is an immune response a good thing?Epigenetic regulation of latent HSV-1 gene expression.Interferon-beta suppresses herpes simplex virus type 1 replication in trigeminal ganglion cells through an RNase L-dependent pathway Small non-coding RNAs encoded within the herpes simplex virus type 1 latency associated transcript (LAT) cooperate with the retinoic acid inducible gene I (RIG-I) to induce beta-interferon promoter activity and promote cell survival.Public TCR use by herpes simplex virus-2-specific human CD8 CTLs.Herpes simplex virus-specific memory CD8+ T cells are selectively activated and retained in latently infected sensory ganglia The murine intravaginal HSV-2 challenge model for investigation of DNA vaccines.Herpes simplex virus type 1 latency-associated transcript expression protects trigeminal ganglion neurons from apoptosis.A historical analysis of herpes simplex virus promoter activation in vivo reveals distinct populations of latently infected neurones Gamma interferon can block herpes simplex virus type 1 reactivation from latency, even in the presence of late gene expression Long-term presence of virus-specific plasma cells in sensory ganglia and spinal cord following intravaginal inoculation of herpes simplex virus type 2.Lytic gene expression is frequent in HSV-1 latent infection and correlates with the engagement of a cell-intrinsic transcriptional response Expression of herpes simplex virus 1-encoded microRNAs in human trigeminal ganglia and their relation to local T-cell infiltrates.Delaying the expression of herpes simplex virus type 1 glycoprotein B (gB) to a true late gene alters neurovirulence and inhibits the gB-CD8+ T-cell response in the trigeminal ganglion Latent infection with herpes simplex virus is associated with ongoing CD8+ T-cell stimulation by parenchymal cells within sensory ganglia Entry of herpes simplex virus type 1 (HSV-1) into the distal axons of trigeminal neurons favors the onset of nonproductive, silent infection Immunization with different viral antigens alters the pattern of T cell exhaustion and latency in herpes simplex virus type 1-infected mice.Herpes keratitis.Neither LAT nor open reading frame P mutations increase expression of spliced or intron-containing ICP0 transcripts in mouse ganglia latently infected with herpes simplex virus Alpha/Beta interferon and gamma interferon synergize to inhibit the replication of herpes simplex virus type 1.Noninvasive bioluminescence imaging of herpes simplex virus type 1 infection and therapy in living mice.Update on herpes virus infections of the nervous system.The early expression of glycoprotein B from herpes simplex virus can be detected by antigen-specific CD8+ T cells.

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Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.

http://www.wikidata.org/entity/Q33894781

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2002 nî lūn-bûn

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2002 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2002 թվականի հունվարին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.

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Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.

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type

label

Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.

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Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.

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Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.

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Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice

@en

P2093

Aaron R Ellison

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Cynthia C Voytek

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Lawrence T Feldman

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Li Yang

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Todd P Margolis

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P2860

Rapid in vivo reactivation of herpes simplex virus in latently infected murine ganglionic neurons after transient hyperthermia.

Evidence that neurons harbor latent herpes simplex virus.

RNA from an immediate early region of the type 1 herpes simplex virus genome is present in the trigeminal ganglia of latently infected mice

Quantification of transcripts from the ICP4 and thymidine kinase genes in mouse ganglia latently infected with herpes simplex virus

Gamma interferon expression during acute and latent nervous system infection by herpes simplex virus type 1.

Inflammatory infiltration of the trigeminal ganglion after herpes simplex virus type 1 corneal infection

Varicella-zoster virus (VZV) transcription during latency in human ganglia: detection of transcripts mapping to genes 21, 29, 62, and 63 in a cDNA library enriched for VZV RNA

Gene expression during reactivation of herpes simplex virus type 1 from latency in the peripheral nervous system is different from that during lytic infection of tissue cultures

A viral function represses accumulation of transcripts from productive-cycle genes in mouse ganglia latently infected with herpes simplex virus.

Aberrant intracellular localization of Varicella-Zoster virus regulatory proteins during latency

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978-983

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10.1073/PNAS.022301899

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2

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99

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Philip R Krause

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2002-01-02T00:00:00Z

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11583638

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11773630

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117416

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