Inflammatory infiltration of the trigeminal ganglion after herpes simplex virus type 1 corneal infection
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Spontaneous reactivation of herpes simplex virus type 1 in latently infected murine sensory gangliaSelective retention of herpes simplex virus-specific T cells in latently infected human trigeminal gangliaBovine Herpes Virus 1 (BHV-1) and Herpes Simplex Virus Type 1 (HSV-1) Promote Survival of Latently Infected Sensory Neurons, in Part by Inhibiting ApoptosisThe molecular basis of herpes simplex virus latencyPathogenesis of herpes stromal keratitis--a focus on corneal neovascularizationKeeping it in check: chronic viral infection and antiviral immunity in the brainGenital Herpes: Insights into Sexually Transmitted Infectious DiseaseImmune Escape via a Transient Gene Expression Program Enables Productive Replication of a Latent Pathogen.Repeated social stress enhances the innate immune response to a primary HSV-1 infection in the cornea and trigeminal ganglia of Balb/c miceReactivation of herpes simplex virus type 1 in the mouse trigeminal ganglion: an in vivo study of virus antigen and cytokines.An important role for major histocompatibility complex class I-restricted T cells, and a limited role for gamma interferon, in protection of mice against lethal herpes simplex virus infection.Towards an understanding of the herpes simplex virus type 1 latency-reactivation cycleHerpes simplex type I (HSV-1) infection of the nervous system: is an immune response a good thing?Distinctive roles for 2',5'-oligoadenylate synthetases and double-stranded RNA-dependent protein kinase R in the in vivo antiviral effect of an adenoviral vector expressing murine IFN-beta.Interferon-beta suppresses herpes simplex virus type 1 replication in trigeminal ganglion cells through an RNase L-dependent pathwayThe locus encompassing the latency-associated transcript of herpes simplex virus type 1 interferes with and delays interferon expression in productively infected neuroblastoma cells and trigeminal Ganglia of acutely infected mice.Vaccine protection against simian immunodeficiency virus by recombinant strains of herpes simplex virus.Role of the individual interferon systems and specific immunity in mice in controlling systemic dissemination of attenuated pseudorabies virus infection.Herpes simplex virus and the chemokines that mediate the inflammation.Protective T-cell-based immunity induced in neonatal mice by a single replicative cycle of herpes simplex virusGamma interferon can prevent herpes simplex virus type 1 reactivation from latency in sensory neurons.Herpes simplex virus-specific memory CD8+ T cells are selectively activated and retained in latently infected sensory gangliaHerpes simplex virus type 1 latency-associated transcript expression protects trigeminal ganglion neurons from apoptosis.Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.Reversible nerve damage and corneal pathology in murine herpes simplex stromal keratitis.Gamma interferon can block herpes simplex virus type 1 reactivation from latency, even in the presence of late gene expressionOrgan-specific roles for transcription factor NF-kappaB in reovirus-induced apoptosis and disease.Long-term presence of virus-specific plasma cells in sensory ganglia and spinal cord following intravaginal inoculation of herpes simplex virus type 2.Expression of herpes simplex virus 1-encoded microRNAs in human trigeminal ganglia and their relation to local T-cell infiltrates.Characterization of the host immune response in human Ganglia after herpes zoster.Latent infection with herpes simplex virus is associated with ongoing CD8+ T-cell stimulation by parenchymal cells within sensory gangliaInflation and long-term maintenance of CD8 T cells responding to a latent herpesvirus depend upon establishment of latency and presence of viral antigens.Herpes keratitis.The antiviral efficacy of the murine alpha-1 interferon transgene against ocular herpes simplex virus type 1 requires the presence of CD4(+), alpha/beta T-cell receptor-positive T lymphocytes with the capacity to produce gamma interferonAlpha/Beta interferon and gamma interferon synergize to inhibit the replication of herpes simplex virus type 1.OAS and PKR are not required for the antiviral effect of Ad:IFN-gamma against acute HSV-1 in primary trigeminal ganglia cultures.Virus-specific immune memory at peripheral sites of herpes simplex virus type 2 (HSV-2) infection in guinea pigs.Herpes simplex virus type 1 and bovine herpesvirus 1 latency.Chronic progressive deficits in neuron size, density and number in the trigeminal ganglia of mice latently infected with herpes simplex virus.Establishment of HSV1 latency in immunodeficient mice facilitates efficient in vivo reactivation.
P2860
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P2860
Inflammatory infiltration of the trigeminal ganglion after herpes simplex virus type 1 corneal infection
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Inflammatory infiltration of t ...... virus type 1 corneal infection
@ast
Inflammatory infiltration of t ...... virus type 1 corneal infection
@en
type
label
Inflammatory infiltration of t ...... virus type 1 corneal infection
@ast
Inflammatory infiltration of t ...... virus type 1 corneal infection
@en
prefLabel
Inflammatory infiltration of t ...... virus type 1 corneal infection
@ast
Inflammatory infiltration of t ...... virus type 1 corneal infection
@en
P2093
P2860
P1433
P1476
Inflammatory infiltration of t ...... virus type 1 corneal infection
@en
P2093
P2860
P304
P407
P577
1996-01-01T00:00:00Z