Microbiota innate stimulation is a prerequisite for T cell spontaneous proliferation and induction of experimental colitis - Wikidata - wikimedia - TriplyDB

Microbiota innate stimulation is a prerequisite for T cell spontaneous proliferation and induction of experimental colitis

Microbiota innate stimulation is a prerequisite for T cell spontaneous proliferation and induction of experimental colitis

http://www.wikidata.org/entity/Q33902387

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Increased prevalence of Methanosphaera stadtmanae in inflammatory bowel diseases Gut microbiota-host interactions and juvenile idiopathic arthritis Luminal Conversion and Immunoregulation by Probiotics The Altered Schaedler Flora: Continued Applications of a Defined Murine Microbial Community Tyro3, Axl, and Mertk receptor signaling in inflammatory bowel disease and colitis-associated cancer The role of gut microbiota in immune homeostasis and autoimmunity Effector and memory T cell responses to commensal bacteria Transient inhibition of ROR-γt therapeutically limits intestinal inflammation by reducing TH17 cells and preserving group 3 innate lymphoid cells Primary Progressive Multiple Sclerosis: Putting Together the Puzzle T cell-derived protein S engages TAM receptor signaling in dendritic cells to control the magnitude of the immune response.Intestinal microbiota link lymphopenia to murine autoimmunity via PD-1+CXCR5-/dim B-helper T cell induction Pharmacological intervention studies using mouse models of the inflammatory bowel diseases: translating preclinical data into new drug therapies.Microbiota activation and regulation of innate and adaptive immunity.Molecular control of steady-state dendritic cell maturation and immune homeostasis.Systemic autoimmunity and lymphoproliferation are associated with excess IL-7 and inhibited by IL-7Rα blockade.Interleukin-7 links T lymphocyte and intestinal epithelial cell homeostasis.Regulation of Toll-like receptor 5 gene expression and function on mucosal dendritic cells.SOCS3 deficiency promotes M1 macrophage polarization and inflammation.The oncoprotein and transcriptional regulator Bcl-3 governs plasticity and pathogenicity of autoimmune T cells The gut microbiota and mucosal T cells.Microbiota: dual-faceted player in experimental colitis.Regulatory T cells and immune tolerance in the intestine.Microbiota regulation of inflammatory bowel disease and colorectal cancer.Regulation of innate and adaptive immunity by the commensal microbiota.Maternal methyl-donor supplementation induces prolonged murine offspring colitis susceptibility in association with mucosal epigenetic and microbiomic changes.Downregulation of microRNA-107 in intestinal CD11c(+) myeloid cells in response to microbiota and proinflammatory cytokines increases IL-23p19 expression.Gut microbiota, immunity, and disease: a complex relationship.Size does not matter: commensal microorganisms forge tumor-promoting inflammation and anti-tumor immunity.mTORC1 Regulates Flagellin-Induced Inflammatory Response in Macrophages Harnessing regulatory T cells for the treatment of inflammatory bowel disease.Both exogenous commensal and endogenous self antigens stimulate T cell proliferation under lymphopenic conditions Th17 cells induce colitis and promote Th1 cell responses through IL-17 induction of innate IL-12 and IL-23 production Unexpected Regulatory Role of CCR9 in Regulatory T Cell Development.The Importance of IL-6 in the Development of LAT-Mediated Autoimmunity.The role of the gastrointestinal tract and microbiota on uremic toxins and chronic kidney disease development.TGF-β signaling to T cells inhibits autoimmunity during lymphopenia-driven proliferation Host-Protozoan Interactions Protect from Mucosal Infections through Activation of the Inflammasome.Lineage targeted MHC-II transgenic mice demonstrate the role of dendritic cells in bacterial-driven colitis.HIV-1 is not a major driver of increased plasma IL-6 levels in chronic HIV-1 disease.Role of the gut microbiota in the development and function of lymphoid cells.

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P2860

Microbiota innate stimulation is a prerequisite for T cell spontaneous proliferation and induction of experimental colitis

http://www.wikidata.org/entity/Q33902387

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Microbiota innate stimulation ...... uction of experimental colitis

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Microbiota innate stimulation ...... uction of experimental colitis

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type

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Microbiota innate stimulation ...... uction of experimental colitis

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Microbiota innate stimulation ...... uction of experimental colitis

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Microbiota innate stimulation ...... uction of experimental colitis

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Microbiota innate stimulation ...... uction of experimental colitis

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Journal of Experimental Medicine

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Microbiota innate stimulation ...... uction of experimental colitis

@en

P2093

Charles O Elson

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Lanfang Wang

http://www.w3.org/2001/XMLSchema#string

Ting Feng

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Trenton R Schoeb

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P2860

Monoclonal Anti–Interleukin 23 Reverses Active Colitis in a T Cell–Mediated Model in Mice

Mechanisms of Disease: pathogenesis of Crohn's disease and ulcerative colitis

Resident enteric bacteria are necessary for development of spontaneous colitis and immune system activation in interleukin-10-deficient mice

Global mapping of H3K4me3 and H3K27me3 reveals specificity and plasticity in lineage fate determination of differentiating CD4+ T cells

Interleukin-23 drives innate and T cell-mediated intestinal inflammation

Pathogen recognition and innate immunity

Molecular-phylogenetic characterization of microbial community imbalances in human inflammatory bowel diseases

Nucleotide binding oligomerization domain 2 deficiency leads to dysregulated TLR2 signaling and induction of antigen-specific colitis

Unravelling the pathogenesis of inflammatory bowel disease

A microbial symbiosis factor prevents intestinal inflammatory disease

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1321-1332

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10.1084/JEM.20092253

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6

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2882839

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