Excessive collagen accumulation in dystrophic (mdx) respiratory musculature is independent of enhanced activation of the NF-kappaB pathway.
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Increased serum and musculotendinous fibrogenic proteins following persistent low-grade inflammation in a rat model of long-term upper extremity overuseApplication of fluorescence two-dimensional difference in-gel electrophoresis as a proteomic biomarker discovery tool in muscular dystrophy researchPharmacologic management of Duchenne muscular dystrophy: target identification and preclinical trialsPericytes: multitasking cells in the regeneration of injured, diseased, and aged skeletal muscle.Dystropathology increases energy expenditure and protein turnover in the mdx mouse model of duchenne muscular dystrophyTransforming growth factor-beta 1 produced by vascular smooth muscle cells predicts fibrosis in the gastrocnemius of patients with peripheral artery disease.Proteomics reveals drastic increase of extracellular matrix proteins collagen and dermatopontin in the aged mdx diaphragm model of Duchenne muscular dystrophyMyosteatosis and myofibrosis: relationship with aging, inflammation and insulin resistance.The multiple faces of valosin-containing protein-associated diseases: inclusion body myopathy with Paget's disease of bone, frontotemporal dementia, and amyotrophic lateral sclerosis.Aquaporin 4 Expression in the mdx Mouse DiaphragmPathoproteomic profiling of the skeletal muscle matrisome in dystrophinopathy associated myofibrosis.Label-free mass spectrometric analysis of the mdx-4cv diaphragm identifies the matricellular protein periostin as a potential factor involved in dystrophinopathy-related fibrosis.Involvement of the nuclear factor-κB pathway in the adhesion of neutrophils to renal tubular cells after injury induced by neonatal postasphyxial serum.Electrical impedance myography for the in vivo and ex vivo assessment of muscular dystrophy (mdx) mouse muscle.The effect of specific IKKβ inhibitors on the cytosolic expression of IκB-α and the nuclear expression of p65 in dystrophic (MDX) muscleTempol Supplementation Restores Diaphragm Force and Metabolic Enzyme Activities in mdx Mice.Inhibition of the angiotensin-converting enzyme decreases skeletal muscle fibrosis in dystrophic mice by a diminution in the expression and activity of connective tissue growth factor (CTGF/CCN-2).Postnatal Hyperplasic Effects of ActRIIB Blockade in a Severely Dystrophic Muscle.
P2860
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P2860
Excessive collagen accumulation in dystrophic (mdx) respiratory musculature is independent of enhanced activation of the NF-kappaB pathway.
description
2010 nî lūn-bûn
@nan
2010 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Excessive collagen accumulatio ...... tion of the NF-kappaB pathway.
@ast
Excessive collagen accumulatio ...... tion of the NF-kappaB pathway.
@en
type
label
Excessive collagen accumulatio ...... tion of the NF-kappaB pathway.
@ast
Excessive collagen accumulatio ...... tion of the NF-kappaB pathway.
@en
prefLabel
Excessive collagen accumulatio ...... tion of the NF-kappaB pathway.
@ast
Excessive collagen accumulatio ...... tion of the NF-kappaB pathway.
@en
P2093
P2860
P1476
Excessive collagen accumulatio ...... tion of the NF-kappaB pathway.
@en
P2093
C G Carlson
C Stefanski
G Malnassy
K Bruemmer
K M Graham
P2860
P356
10.1016/J.JNS.2010.04.007
P577
2010-05-13T00:00:00Z