Mechanical stress activates the nuclear factor-kappaB pathway in skeletal muscle fibers: a possible role in Duchenne muscular dystrophy.
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LINC complex alterations in DMD and EDMD/CMT fibroblastsOxidative Stress-Mediated Skeletal Muscle Degeneration: Molecules, Mechanisms, and TherapiesCyclooxygenase-2 or tumor necrosis factor-α inhibitors attenuate the mechanotransductive effects of pulsed focused ultrasound to suppress mesenchymal stromal cell homing to healthy and dystrophic muscleThe role of oxidative stress in skeletal muscle injury and regeneration: focus on antioxidant enzymesRedox signaling in skeletal muscle: role of aging and exerciseFibroblast growth factor inducible 14 (Fn14) is required for the expression of myogenic regulatory factors and differentiation of myoblasts into myotubes. Evidence for TWEAK-independent functions of Fn14 during myogenesisIncreased atrial arrhythmia susceptibility induced by intense endurance exercise in mice requires TNFαAnisotropic regulation of Ankrd2 gene expression in skeletal muscle by mechanical stretchMulti-tasking role of the mechanosensing protein Ankrd2 in the signaling network of striated muscle.Use of imaging biomarkers to assess perfusion and glucose metabolism in the skeletal muscle of dystrophic mice.NF-kappaB mediates the transcription of mouse calsarcin-1 gene, but not calsarcin-2, in C2C12 cellsActivation of p53-regulated pro-apoptotic signaling pathways in PrP-mediated myopathyGenomic profiling of messenger RNAs and microRNAs reveals potential mechanisms of TWEAK-induced skeletal muscle wasting in miceA Novel NF-κB Inhibitor, Edasalonexent (CAT-1004), in Development as a Disease-Modifying Treatment for Patients With Duchenne Muscular Dystrophy: Phase 1 Safety, Pharmacokinetics, and Pharmacodynamics in Adult SubjectsTherapeutic targeting of signaling pathways in muscular dystrophy.The TWEAK-Fn14 dyad is involved in age-associated pathological changes in skeletal muscleThe TWEAK-Fn14 system is a critical regulator of denervation-induced skeletal muscle atrophy in mice.Dlk1 is necessary for proper skeletal muscle development and regeneration.Skeletal muscle NADPH oxidase is increased and triggers stretch-induced damage in the mdx mouse.c-Kit modifies the inflammatory status of smooth muscle cellsPharmacological Inhibition of PKCθ Counteracts Muscle Disease in a Mouse Model of Duchenne Muscular Dystrophy.Excessive collagen accumulation in dystrophic (mdx) respiratory musculature is independent of enhanced activation of the NF-kappaB pathway.Selective modulation through the glucocorticoid receptor ameliorates muscle pathology in mdx miceMatrix metalloproteinase inhibitor batimastat alleviates pathology and improves skeletal muscle function in dystrophin-deficient mdx mice.Bone marrow mononuclear cells up-regulate toll-like receptor expression and produce inflammatory mediators in response to cigarette smoke extractTumor necrosis factor-alpha augments matrix metalloproteinase-9 production in skeletal muscle cells through the activation of transforming growth factor-beta-activated kinase 1 (TAK1)-dependent signaling pathwayGenetic ablation of TWEAK augments regeneration and post-injury growth of skeletal muscle in mice.PKC theta ablation improves healing in a mouse model of muscular dystrophy.Targeted ablation of TRAF6 inhibits skeletal muscle wasting in mice.Identification of genes that elicit disuse muscle atrophy via the transcription factors p50 and Bcl-3.Induction of Sirt1 by mechanical stretch of skeletal muscle through the early response factor EGR1 triggers an antioxidative responseNuclear factor-kappa B signaling in skeletal muscle atrophy.The influence of passive stretch and NF-κB inhibitors on the morphology of dystrophic muscle fibers.A multidisciplinary evaluation of the effectiveness of cyclosporine a in dystrophic mdx mice.C26 cancer-induced muscle wasting is IKKβ-dependent and NF-kappaB-independent.Lipid peroxidation inhibition blunts nuclear factor-kappaB activation, reduces skeletal muscle degeneration, and enhances muscle function in mdx mice.Targeted ablation of IKK2 improves skeletal muscle strength, maintains mass, and promotes regenerationOsteopontin-stimulated expression of matrix metalloproteinase-9 causes cardiomyopathy in the mdx model of Duchenne muscular dystrophyRemote activation of the Wnt/β-catenin signalling pathway using functionalised magnetic particles.The dual roles of neutrophils and macrophages in inflammation: a critical balance between tissue damage and repair.
P2860
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P2860
Mechanical stress activates the nuclear factor-kappaB pathway in skeletal muscle fibers: a possible role in Duchenne muscular dystrophy.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Mechanical stress activates th ...... n Duchenne muscular dystrophy.
@en
Mechanical stress activates th ...... n Duchenne muscular dystrophy.
@nl
type
label
Mechanical stress activates th ...... n Duchenne muscular dystrophy.
@en
Mechanical stress activates th ...... n Duchenne muscular dystrophy.
@nl
prefLabel
Mechanical stress activates th ...... n Duchenne muscular dystrophy.
@en
Mechanical stress activates th ...... n Duchenne muscular dystrophy.
@nl
P356
P1433
P1476
Mechanical stress activates th ...... in Duchenne muscular dystrophy
@en
P2093
Aladin M Boriek
P304
P356
10.1096/FJ.02-0542COM
P407
P50
P577
2003-03-01T00:00:00Z