Role of heat shock factor-1 activation in the doxorubicin-induced heart failure in mice.
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Heat shock protein 25-enriched plasma transfusion preconditions the heart against doxorubicin-induced dilated cardiomyopathy in mice.Presence of Cx43 in extracellular vesicles reduces the cardiotoxicity of the anti-tumour therapeutic approach with doxorubicinIdentification of protein targets underlying dietary nitrate-induced protection against doxorubicin cardiotoxicityReduced in vivo high-energy phosphates precede adriamycin-induced cardiac dysfunction.Forced expression of heat shock protein 27 (Hsp27) reverses P-glycoprotein (ABCB1)-mediated drug efflux and MDR1 gene expression in Adriamycin-resistant human breast cancer cellsProteostasis and REDOX state in the heart.Heat shock factor-1 knockout induces multidrug resistance gene, MDR1b, and enhances P-glycoprotein (ABCB1)-based drug extrusion in the heart.Small heat shock proteins in redox metabolism: implications for cardiovascular diseases.Reductive stress linked to small HSPs, G6PD, and Nrf2 pathways in heart diseaseCancer therapy-induced cardiac toxicity in early breast cancer: addressing the unresolved issues.Heat shock factor-1 knockout enhances cholesterol 7α-hydroxylase (CYP7A1) and multidrug transporter (MDR1) gene expressions to attenuate atherosclerosis.Cardiovascular disease following hematopoietic stem cell transplantation: Pathogenesis, detection, and the cardioprotective role of aerobic trainingDoxorubicin attenuates CHIP-guarded HSF1 nuclear translocation and protein stability to trigger IGF-IIR-dependent cardiomyocyte death.Contribution of small heat shock proteins to muscle development and function.Anthracycline-Induced Cardiomyopathy in Adults.Hsp70 regulates the doxorubicin-mediated heart failure in Hsp70-transgenic mice.Doxorubicin-induced chronic dilated cardiomyopathy-the apoptosis hypothesis revisited.Heat-shock factor 1 both positively and negatively affects cellular clonogenic growth depending on p53 status.Signalling mechanisms underlying doxorubicin and Nox2 NADPH oxidase-induced cardiomyopathy: involvement of mitofusin-2.Mitochondrial ROS-induced ERK1/2 activation and HSF2-mediated AT1 R upregulation are required for doxorubicin-induced cardiotoxicity.HSF1 phosphorylation by ERK/GSK3 suppresses RNF126 to sustain IGF-IIR expression for hypertension-induced cardiomyocyte hypertrophy.Propofol Protects Rat Cardiomyocytes from Anthracycline-Induced Apoptosis by Regulating MicroRNA-181a In Vitro and In Vivo.
P2860
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P2860
Role of heat shock factor-1 activation in the doxorubicin-induced heart failure in mice.
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2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Role of heat shock factor-1 activation in the doxorubicin-induced heart failure in mice.
@ast
Role of heat shock factor-1 activation in the doxorubicin-induced heart failure in mice.
@en
type
label
Role of heat shock factor-1 activation in the doxorubicin-induced heart failure in mice.
@ast
Role of heat shock factor-1 activation in the doxorubicin-induced heart failure in mice.
@en
prefLabel
Role of heat shock factor-1 activation in the doxorubicin-induced heart failure in mice.
@ast
Role of heat shock factor-1 activation in the doxorubicin-induced heart failure in mice.
@en
P2093
P2860
P1476
Role of heat shock factor-1 activation in the doxorubicin-induced heart failure in mice.
@en
P2093
Govindasamy Ilangovan
Jay L Zweier
Kaushik Vedam
Lawrence J Druhan
Mahmood Khan
Nicanor I Moldovan
Yoshinori Nishijima
P2860
P304
P356
10.1152/AJPHEART.01047.2009
P577
2010-04-02T00:00:00Z