7-Ketocholesterol-induced inflammation signals mostly through the TLR4 receptor both in vitro and in vivo. - Wikidata - wikimedia - TriplyDB

7-Ketocholesterol-induced inflammation signals mostly through the TLR4 receptor both in vitro and in vivo.

7-Ketocholesterol-induced inflammation signals mostly through the TLR4 receptor both in vitro and in vivo.

http://www.wikidata.org/entity/Q33921301

about

The unfolded protein response in retinal vascular diseases: implications and therapeutic potential beyond protein folding 7-Ketocholesterol increases retinal microglial migration, activation, and angiogenicity: a potential pathogenic mechanism underlying age-related macular degeneration Extra-hepatic metabolism of 7-ketocholesterol occurs by esterification to fatty acids via cPLA2α and SOAT1 followed by selective efflux to HDL 7-ketocholesterol induces apoptosis of MC3T3-E1 cells associated with reactive oxygen species generation, endoplasmic reticulum stress and caspase-3/7 dependent pathway Lipids, oxidized lipids, oxidation-specific epitopes, and Age-related Macular Degeneration.The Role of Oxysterols in Human Cancer.Lipidomics reveals accumulation of the oxidized cholesterol in erythrocytes of heart failure patients.

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7-Ketocholesterol-induced inflammation signals mostly through the TLR4 receptor both in vitro and in vivo.

http://www.wikidata.org/entity/Q33921301

description

2014 nî lūn-bûn

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2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2014 թվականի հուլիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Ignacio R Rodriguez

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Jiahn-Dar Huang

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Jung Wha Lee

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P2860

Activation and function of the MAPKs and their substrates, the MAPK-activated protein kinases

The selectivity of protein kinase inhibitors: a further update

A comprehensive pathway map of epidermal growth factor receptor signaling

Oxysterols in the pathogenesis of major chronic diseases

The SYK side of TLR4: signalling mechanisms in response to LPS and minimally oxidized LDL

SOCS, inflammation, and cancer

Targeting toll-like receptors: promising therapeutic strategies for the management of sepsis-associated pathology and infectious diseases

ER stress-induced cell death mechanisms

Modulatory mechanisms controlling the NLRP3 inflammasome in inflammation: recent developments

SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase

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