Inherent abnormalities in energy metabolism in Alzheimer disease. Interaction with cerebrovascular compromise.
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Insulin in central nervous system: more than just a peripheral hormoneAlzheimer's pathogenesis and its link to the mitochondrionDefects in mitochondrial dynamics and metabolomic signatures of evolving energetic stress in mouse models of familial Alzheimer's diseaseTargeting the prodromal stage of Alzheimer's disease: bioenergetic and mitochondrial opportunitiesOxidatively modified calmodulin binds to the plasma membrane Ca-ATPase in a nonproductive and conformationally disordered complex.2-Deoxy-D-glucose treatment induces ketogenesis, sustains mitochondrial function, and reduces pathology in female mouse model of Alzheimer's diseaseImpaired balance of mitochondrial fission and fusion in Alzheimer's diseaseKetogenic diet improves motor performance but not cognition in two mouse models of Alzheimer's pathologyThe KATP channel activator diazoxide ameliorates amyloid-β and tau pathologies and improves memory in the 3xTgAD mouse model of Alzheimer's disease.What have novel imaging techniques revealed about metabolism in the aging brain?Neuroprotective Effects of Icariin on Brain Metabolism, Mitochondrial Functions, and Cognition in Triple-Transgenic Alzheimer's Disease Mice.Crucial role of calbindin-D28k in the pathogenesis of Alzheimer's disease mouse model.Age-dependent modulation of synaptic plasticity and insulin mimetic effect of lipoic acid on a mouse model of Alzheimer's disease.Estrogen regulation of glucose metabolism and mitochondrial function: therapeutic implications for prevention of Alzheimer's disease.Amyloid-β peptide binds to cytochrome C oxidase subunit 1.Brain metabolism and brain disease: is metabolic deficiency the proximate cause of Alzheimer dementia?Mitochondria, Amyloid β, and Alzheimer's DiseaseUpregulation of PGC-1α expression by Alzheimer's disease-associated pathway: presenilin 1/amyloid precursor protein (APP)/intracellular domain of APPMedroxyprogesterone acetate antagonizes estrogen up-regulation of brain mitochondrial functionOvarian hormone loss induces bioenergetic deficits and mitochondrial β-amyloidDecline in mitochondrial bioenergetics and shift to ketogenic profile in brain during reproductive senescenceCerebrometabolic abnormalities in Alzheimer's disease.Roles of amyloid beta-peptide-associated oxidative stress and brain protein modifications in the pathogenesis of Alzheimer's disease and mild cognitive impairment.Swedish Alzheimer mutation induces mitochondrial dysfunction mediated by HSP60 mislocalization of amyloid precursor protein (APP) and beta-amyloid.New evidence of mitochondria dysfunction in the female Alzheimer's disease brain: deficiency of estrogen receptor-β.White Matter Lipids as a Ketogenic Fuel Supply in Aging Female Brain: Implications for Alzheimer's Disease.The Protective Effect of Icariin on Mitochondrial Transport and Distribution in Primary Hippocampal Neurons from 3× Tg-AD Mice.Dynamin-like protein 1 reduction underlies mitochondrial morphology and distribution abnormalities in fibroblasts from sporadic Alzheimer's disease patients.Activation of c-Jun-N-terminal kinase and decline of mitochondrial pyruvate dehydrogenase activity during brain aging.Mitochondrial bioenergetic deficit precedes Alzheimer's pathology in female mouse model of Alzheimer's disease.Estrogen regulation of mitochondrial bioenergetics: implications for prevention of Alzheimer's disease.JNK: a stress-activated protein kinase therapeutic strategies and involvement in Alzheimer's and various neurodegenerative abnormalities.Astrocytes in Alzheimer's disease.The pyruvate dehydrogenase complex as a therapeutic target for age-related diseases.Mitochondrial function in ageing: coordination with signalling and transcriptional pathways.Translational potential of astrocytes in brain disorders.Nutritional interventions for Alzheimer's prevention: a clinical precision medicine approach.Energy metabolism and inflammation in brain aging and Alzheimer's disease.Bioenergetic Impairment in Animal and Cellular Models of Alzheimer's Disease: PARP-1 Inhibition Rescues Metabolic Dysfunctions.Metabolic Stress and Disorders Related to Alterations in Mitochondrial Fission or Fusion.
P2860
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P2860
Inherent abnormalities in energy metabolism in Alzheimer disease. Interaction with cerebrovascular compromise.
description
2000 nî lūn-bûn
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2000 թուականի Ապրիլին հրատարակուած գիտական յօդուած
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2000 թվականի ապրիլին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
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2000年論文
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2000年論文
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2000年論文
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2000年論文
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2000年论文
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name
Inherent abnormalities in ener ...... th cerebrovascular compromise.
@ast
Inherent abnormalities in ener ...... th cerebrovascular compromise.
@en
type
label
Inherent abnormalities in ener ...... th cerebrovascular compromise.
@ast
Inherent abnormalities in ener ...... th cerebrovascular compromise.
@en
prefLabel
Inherent abnormalities in ener ...... th cerebrovascular compromise.
@ast
Inherent abnormalities in ener ...... th cerebrovascular compromise.
@en
P2093
P2860
P1476
Inherent abnormalities in ener ...... th cerebrovascular compromise.
@en
P2093
P2860
P304
P356
10.1111/J.1749-6632.2000.TB06370.X
P407
P577
2000-04-01T00:00:00Z