Loss and gain of chromosome 5 controls growth of Candida albicans on sorbose due to dispersed redundant negative regulators
about
Genomic Plasticity of the Human Fungal Pathogen Candida albicansEnvironmental induction of white-opaque switching in Candida albicans.Neocentromeres form efficiently at multiple possible loci in Candida albicans.Regulation of white and opaque cell-type formation in Candida albicans by Rtt109 and Hst3Molecular genetic techniques for gene manipulation in Candida albicans.Adaptation of Candida albicans to growth on sorbose via monosomy of chromosome 5 accompanied by duplication of another chromosome carrying a gene responsible for sorbose utilizationRapid mechanisms for generating genome diversity: whole ploidy shifts, aneuploidy, and loss of heterozygosityDynamic large-scale chromosomal rearrangements fuel rapid adaptation in yeast populations.Candida albicans strain maintenance, replacement, and microvariation demonstrated by multilocus sequence typing.Transcriptional regulatory circuitries in the human pathogen Candida albicans involving sense--antisense interactions.Molecular phylogenetics of Candida albicans.Chromosome 5 monosomy of Candida albicans controls susceptibility to various toxic agents, including major antifungalsAnalysis of Repair Mechanisms following an Induced Double-Strand Break Uncovers Recessive Deleterious Alleles in the Candida albicans Diploid Genome.Widespread occurrence of chromosomal aneuploidy following the routine production of Candida albicans mutants.Human fungal pathogen Candida albicans in the postgenomic era: an overview.Ploidy Variation in Fungi: Polyploidy, Aneuploidy, and Genome EvolutionChromosome 5 of Human Pathogen Candida albicans Carries Multiple Genes for Negative Control of Caspofungin and Anidulafungin Susceptibility.Loss and fragmentation of chromosome 5 are major events linked to the adaptation of rad52-DeltaDelta strains of Candida albicans to sorboseLoss of heterozygosity at an unlinked genomic locus is responsible for the phenotype of a Candida albicans sap4Δ sap5Δ sap6Δ mutant.Aneuploid chromosomes are highly unstable during DNA transformation of Candida albicans.NuA4 histone acetyltransferase activity is required for H4 acetylation on a dosage-compensated monosomic chromosome that confers resistance to fungal toxins.An isochromosome confers drug resistance in vivo by amplification of two genes, ERG11 and TAC1.The zinc cluster transcription factor Tac1p regulates PDR16 expression in Candida albicans.Transcriptional Regulation on Aneuploid Chromosomes in Divers Candida albicans Mutants.
P2860
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P2860
Loss and gain of chromosome 5 controls growth of Candida albicans on sorbose due to dispersed redundant negative regulators
description
2005 nî lūn-bûn
@nan
2005 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Loss and gain of chromosome 5 ...... redundant negative regulators
@ast
Loss and gain of chromosome 5 ...... redundant negative regulators
@en
type
label
Loss and gain of chromosome 5 ...... redundant negative regulators
@ast
Loss and gain of chromosome 5 ...... redundant negative regulators
@en
prefLabel
Loss and gain of chromosome 5 ...... redundant negative regulators
@ast
Loss and gain of chromosome 5 ...... redundant negative regulators
@en
P2093
P2860
P356
P1476
Loss and gain of chromosome 5 ...... redundant negative regulators
@en
P2093
Ausaf Ahmad
Elena Rustchenko
Jay R Greenberg
M Anaul Kabir
Ying-Kai Wang
P2860
P304
12147-12152
P356
10.1073/PNAS.0505625102
P407
P577
2005-08-11T00:00:00Z