Environmental induction of white-opaque switching in Candida albicans. - Wikidata - wikimedia - TriplyDB

Environmental induction of white-opaque switching in Candida albicans.

Environmental induction of white-opaque switching in Candida albicans.

http://www.wikidata.org/entity/Q33343437

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Genomic Plasticity of the Human Fungal Pathogen Candida albicans Candida albicans the chameleon: transitions and interactions between multiple phenotypic states confer phenotypic plasticity Regulation of phenotypic transitions in the fungal pathogen Candida albicans The role of phenotypic switching in the basic biology and pathogenesis of Candida albicans The cryptic sexual strategies of human fungal pathogens Transcriptional Control of Drug Resistance, Virulence and Immune System Evasion in Pathogenic Fungi: A Cross-Species Comparison The transcription factor Ndt80 does not contribute to Mrr1-, Tac1-, and Upc2-mediated fluconazole resistance in Candida albicans The evolution of sex: a perspective from the fungal kingdom Phenotypic Profiling Reveals that Candida albicans Opaque Cells Represent a Metabolically Specialized Cell State Compared to Default White Cells White-opaque switching of Candida albicans allows immune evasion in an environment-dependent fashion Modeling the transcriptional regulatory network that controls the early hypoxic response in Candida albicans An A643T mutation in the transcription factor Upc2p causes constitutive ERG11 upregulation and increased fluconazole resistance in Candida albicans Yeasts in the gut: from commensals to infectious agents.White-opaque switching in Candida albicans.The transcriptomes of two heritable cell types illuminate the circuit governing their differentiation To switch or not to switch?: Phenotypic switching is sensitive to multiple inputs in a pathogenic fungus.Regulation of white and opaque cell-type formation in Candida albicans by Rtt109 and Hst3 Coming of age--sexual reproduction in Candida species.Evolution of eukaryotic microbial pathogens via covert sexual reproduction Regulation of white-opaque switching in Candida albicans.Rapid mechanisms for generating genome diversity: whole ploidy shifts, aneuploidy, and loss of heterozygosity Factors supporting cysteine tolerance and sulfite production in Candida albicans MTL-independent phenotypic switching in Candida tropicalis and a dual role for Wor1 in regulating switching and filamentation.Temporal anatomy of an epigenetic switch in cell programming: the white-opaque transition of C. albicans Candida albicans Czf1 and Efg1 coordinate the response to farnesol during quorum sensing, white-opaque thermal dimorphism, and cell death.Regulation of efflux pump expression and drug resistance by the transcription factors Mrr1, Upc2, and Cap1 in Candida albicans.Stress-induced phenotypic switching in Candida albicans Activation of the Cph1-dependent MAP kinase signaling pathway induces white-opaque switching in Candida albicans Self-induction of a/a or alpha/alpha biofilms in Candida albicans is a pheromone-based paracrine system requiring switching Global changes in gene expression associated with phenotypic switching of wild yeast Regulation of filamentation in the human fungal pathogen Candida tropicalis.Parasexual Ploidy Reduction Drives Population Heterogeneity Through Random and Transient Aneuploidy in Candida albicans Does stress induce (para)sex? Implications for Candida albicans evolution The WOR1 5' untranslated region regulates white-opaque switching in Candida albicans by reducing translational efficiency The three clades of the telomere-associated TLO gene family of Candida albicans have different splicing, localization, and expression features.Roles of different peptide transporters in nutrient acquisition in Candida albicans Nucleosome assembly factors CAF-1 and HIR modulate epigenetic switching frequencies in an H3K56 acetylation-associated manner in Candida albicans.Deletion of a Yci1 Domain Protein of Candida albicans Allows Homothallic Mating in MTL Heterozygous Cells.Phenotypic Plasticity Regulates Candida albicans Interactions and Virulence in the Vertebrate Host Bcr1 plays a central role in the regulation of opaque cell filamentation in Candida albicans

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Environmental induction of white-opaque switching in Candida albicans.

http://www.wikidata.org/entity/Q33343437

description

2008 nî lūn-bûn

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2008 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2008 թվականի հունիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Environmental induction of white-opaque switching in Candida albicans.

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Environmental induction of white-opaque switching in Candida albicans.

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type

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Environmental induction of white-opaque switching in Candida albicans.

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Environmental induction of white-opaque switching in Candida albicans.

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Environmental induction of white-opaque switching in Candida albicans.

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Environmental induction of white-opaque switching in Candida albicans.

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Environmental induction of white-opaque switching in Candida albicans.

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Bernardo Ramírez-Zavala

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Joachim Morschhäuser

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Knut Ohlsen

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Oliver Reuss

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Yang-Nim Park

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P2860

Interlocking transcriptional feedback loops control white-opaque switching in Candida albicans

Opaque cells signal white cells to form biofilms in Candida albicans

Epigenetic properties of white-opaque switching in Candida albicans are based on a self-sustaining transcriptional feedback loop

A role for sterol levels in oxygen sensing in Saccharomyces cerevisiae.

Regulatory networks controlling Candida albicans morphogenesis.

Loss and gain of chromosome 5 controls growth of Candida albicans on sorbose due to dispersed redundant negative regulators

Rfg1, a protein related to the Saccharomyces cerevisiae hypoxic regulator Rox1, controls filamentous growth and virulence in Candida albicans

Specific chromosome alterations in fluconazole-resistant mutants of Candida albicans.

Misexpression of the opaque-phase-specific gene PEP1 (SAP1) in the white phase of Candida albicans confers increased virulence in a mouse model of cutaneous infection.

Effects of low concentrations of zinc on the growth and dimorphism of Candida albicans: evidence for zinc-resistant and -sensitive pathways for mycelium formation

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