Physiological induction and reversal of focus formation and tumorigenicity in NIH 3T3 cells. - Wikidata - wikimedia - TriplyDB

Physiological induction and reversal of focus formation and tumorigenicity in NIH 3T3 cells.

Physiological induction and reversal of focus formation and tumorigenicity in NIH 3T3 cells.

http://www.wikidata.org/entity/Q33927878

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Activation of 5-HT1A receptors expressed in NIH-3T3 cells induces focus formation and potentiates EGF effect on DNA synthesis Incipient and overt stages of neoplastic transformation Degrees and kinds of selection in spontaneous neoplastic transformation: an operational analysis Transformation of BALB/c-3T3 cells: I. Investigation of experimental parameters that influence detection of spontaneous transformation.Cellular epigenetics: topochronology of progressive "spontaneous" transformation of cells under growth constraint.Selective nature of phorbol 12-myristate 13-acetate-induced neoplastic transformation in NIH 3T3 cells.Experimental control of neoplastic progression in cell populations: Foulds' rules revisited.A critical test of the role of population density in producing transformation.A novel canine kidney cell line model for the evaluation of neoplastic development: karyotype evolution associated with spontaneous immortalization and tumorigenicity.Clonal dynamics of progressive neoplastic transformation Cell-cell contact interactions conditionally determine suppression and selection of the neoplastic phenotype.Automatic enumeration and characterization of heterogeneous clonal progression in cell transformation.Cellular epigenetics: effects of passage history on competence of cells for "spontaneous" transformation.Adaptive evolution of degrees and kinds of neoplastic transformation in cell culture Promotion and selection by serum growth factors drive field cancerization, which is anticipated in vivo by type 2 diabetes and obesity Coculturing diverse clonal populations prevents the early-stage neoplastic progression that occurs in the separate clones Sensitivity of transformation to small differences in population density during serial passage of NIH 3T3 cells.The cellular ecology of progressive neoplastic transformation: a clonal analysis.Irreversibility of cellular aging and neoplastic transformation: a clonal analysis.High rate of diversification and reversal among subclones of neoplastically transformed NIH 3T3 clones.Growth in high serum concentrations leads to rapid deadaptation of cells previously adapted to growth in an extremely low concentration of serum.CDK-inhibitor independent cell cycle progression in an experimental haematopoietic stem cell leukaemia despite unaltered Rb-phosphorylation The nature of cancer: morphogenesis and progressive (self)-disorganization in neoplastic development and progression.Dynamics of cell transformation in culture and its significance for tumor development in animals.Phenotypic selection as the biological mode of epigenetic conversion and reversion in cell transformation.

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P2860

Physiological induction and reversal of focus formation and tumorigenicity in NIH 3T3 cells.

http://www.wikidata.org/entity/Q33927878

description

1990 nî lūn-bûn

@nan

1990 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1990 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

1990年の論文

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1990年論文

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1990年論文

@zh-hant

1990年論文

@zh-hk

1990年論文

@zh-mo

1990年論文

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1990年论文

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name

Physiological induction and re ...... morigenicity in NIH 3T3 cells.

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Physiological induction and re ...... morigenicity in NIH 3T3 cells.

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P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Physiological induction and re ...... morigenicity in NIH 3T3 cells.

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P2093

A L Rubin

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H Rubin

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P Arnstein

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P2860

Murine sarcoma and leukemia viruses: assay using clonal lines of contact-inhibited mouse cells

Evidence for the progressive and adaptive nature of spontaneous transformation in the NIH 3T3 cell line.

Relation of spontaneous transformation in cell culture to adaptive growth and clonal heterogeneity.

On the nature of enduring modifications induced in cells and organisms.

Cellular biochemistry of the stepwise development of cancer with chemicals: G. H. A. Clowes memorial lecture.

Emergence of neoplastic transformants spontaneously or after exposure to N-methyl-N'-nitro-N-nitrosoguanidine in populations of rat liver epithelial cells cultured under selective and nonselective conditions.

Heritable variations in growth potential and morphology within a clone of Balb/3T3 cells and their relation to tumor formation.

Improved medium and culture conditions for clonal growth with minimal serum protein and for enhanced serum-free survival of Swiss 3T3 cells.

Suppression of transformation by and growth adaptation to low concentrations of glutamine in NIH-3T3 cells.

Quantitative and qualitative studies of chemical transformation of cloned C3H mouse embryo cells sensitive to postconfluence inhibition of cell division.

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