Lack of both bradykinin B1 and B2 receptors enhances nephropathy, neuropathy, and bone mineral loss in Akita diabetic mice - Wikidata - wikimedia - TriplyDB

Lack of both bradykinin B1 and B2 receptors enhances nephropathy, neuropathy, and bone mineral loss in Akita diabetic mice

Lack of both bradykinin B1 and B2 receptors enhances nephropathy, neuropathy, and bone mineral loss in Akita diabetic mice

http://www.wikidata.org/entity/Q33933352

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Vascular endothelial dysfunction and pharmacological treatment The kallikrein-kinin system in diabetic nephropathy The kallikrein-kinin system and oxidative stress Role of plasma kallikrein in diabetes and metabolism Alternative pathways in the development of diabetic retinopathy: the renin-angiotensin and kallikrein-kinin systems Altered glucose homeostasis and hepatic function in obese mice deficient for both kinin receptor genes Kinin B1 receptor enhances the oxidative stress in a rat model of insulin resistance: outcome in hypertension, allodynia and metabolic complications Balance between the two kinin receptors in the progression of experimental focal and segmental glomerulosclerosis in mice Plasma bradykinin and early diabetic nephropathy lesions in type 1 diabetes mellitus Classical Renin-Angiotensin system in kidney physiology Prevention of diabetic nephropathy in Ins2(+/)⁻(AkitaJ) mice by the mitochondria-targeted therapy MitoQ.Inflammation in the pathogenesis of microvascular complications in diabetes.Kinin B2 receptor does not exert renoprotective effects on mice with glycerol-induced rhabdomyolysis.Global renal gene expression profiling analysis in B2-kinin receptor null mice: impact of diabetes Downregulation of kinin B1 receptor function by B2 receptor heterodimerization and signaling.Genetic modulation of diabetic nephropathy among mouse strains with Ins2 Akita mutation.Osteoporosis in diabetes mellitus: Possible cellular and molecular mechanisms.Knockout of the TauT gene predisposes C57BL/6 mice to streptozotocin-induced diabetic nephropathy.SOD1, but not SOD3, deficiency accelerates diabetic renal injury in C57BL/6-Ins2(Akita) diabetic mice Local bone interaction between renin-angiotensin system and kallikrein-kinin system in diabetic rat.Semaphorin3a promotes advanced diabetic nephropathy The identification of gene expression profiles associated with progression of human diabetic neuropathy Integrin α1/Akita double-knockout mice on a Balb/c background develop advanced features of human diabetic nephropathy.Effects of Alpha-Lipoic Acid on Oxidative Stress and Kinin Receptor Expression in Obese Zucker Diabetic Fatty Rats Mechanisms of bradykinin-induced expression of connective tissue growth factor and nephrin in podocytes.The kallikrein-kinin system as a regulator of cardiovascular and renal function.Null mutations at the p66 and bradykinin 2 receptor loci induce divergent phenotypes in the diabetic kidney.IL-4 and IL-13 inhibit IL-1β and TNF-α induced kinin B1 and B2 receptors through a STAT6-dependent mechanism.Kidney tubules: intertubular, vascular, and glomerular cross-talk.Protective role of AT(2) and B(1) receptors in kinin B(2)-receptor-knockout mice with myocardial infarction Mouse models of diabetic neuropathy.VEGF and podocytes in diabetic nephropathy.Targeting the 'Janus face' of the B2-bradykinin receptor.Non-canonical signalling and roles of the vasoactive peptides angiotensins and kinins.Bradykinin receptors and EphB2/EphrinB2 pathway in response to high glucose-induced osteoblast dysfunction and hyperglycemia-induced bone deterioration in mice.A new perspective on mechanisms governing skeletal complications in type 1 diabetes.Bradykinin regulates osteoblast differentiation by Akt/ERK/NFκB signaling axis.Antihypertensive and renoprotective effect of the kinin pathway activated by potassium in a model of salt sensitivity following overload proteinuria.Kinin B1 Receptor Deletion Affects Bone Healing in Type 1 Diabetic Mice.Vascular Kinin B1 and B2 Receptors Determine Endothelial Dysfunction through Neuronal Nitric Oxide Synthase.

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Lack of both bradykinin B1 and B2 receptors enhances nephropathy, neuropathy, and bone mineral loss in Akita diabetic mice

http://www.wikidata.org/entity/Q33933352

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2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Lack of both bradykinin B1 and ...... al loss in Akita diabetic mice

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Lack of both bradykinin B1 and ...... al loss in Akita diabetic mice

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Lack of both bradykinin B1 and ...... al loss in Akita diabetic mice

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Lack of both bradykinin B1 and ...... al loss in Akita diabetic mice

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Lack of both bradykinin B1 and ...... al loss in Akita diabetic mice

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Proceedings of the National Academy of Sciences of the United States of America

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Lack of both bradykinin B1 and ...... al loss in Akita diabetic mice

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Adil M H Gasim

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Carey Backus

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Hirofumi Tomita

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Hyung-Suk Kim

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J Charles Jennette

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John M Hayes

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Kelli A Sullivan

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Kunjie Hua

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Masao Kakoki

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Oliver Smithies

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P2860

cAMP controls oxygen metabolism in mammalian cells

Reaction conditions affecting the relationship between thiobarbituric acid reactivity and lipid peroxides in human plasma

Regulation of cardiac bradykinin B1- and B2-receptor mRNA in experimental ischemic, diabetic, and pressure-overload-induced cardiomyopathy.

The effect of bradykinin on the oxidative state of rats with acute hyperglycaemia.

Importance of quantitative genetic variations in the etiology of hypertension.

An insertion/deletion polymorphism in the angiotensin I-converting enzyme gene accounting for half the variance of serum enzyme levels.

Hepatic expression of mature transforming growth factor beta 1 in transgenic mice results in multiple tissue lesions.

Senescence-associated phenotypes in Akita diabetic mice are enhanced by absence of bradykinin B2 receptors.

Control of cytochrome c oxidase activity by nitric oxide.

Bradykinin B1 and B2 receptors both have protective roles in renal ischemia/reperfusion injury.

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10190-10195

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20479236

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