Null mutations at the p66 and bradykinin 2 receptor loci induce divergent phenotypes in the diabetic kidney.
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The Role of SIRT1 in Diabetic Kidney Disease.AT1R blockade in adverse milieus: role of SMRT and corepressor complexes.p66Shc: A novel biomarker of tubular oxidative injury in patients with diabetic nephropathy.Vitamin D receptor deficit induces activation of renin angiotensin system via SIRT1 modulation in podocytes.
P2860
Null mutations at the p66 and bradykinin 2 receptor loci induce divergent phenotypes in the diabetic kidney.
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2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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name
Null mutations at the p66 and ...... otypes in the diabetic kidney.
@ast
Null mutations at the p66 and ...... otypes in the diabetic kidney.
@en
type
label
Null mutations at the p66 and ...... otypes in the diabetic kidney.
@ast
Null mutations at the p66 and ...... otypes in the diabetic kidney.
@en
prefLabel
Null mutations at the p66 and ...... otypes in the diabetic kidney.
@ast
Null mutations at the p66 and ...... otypes in the diabetic kidney.
@en
P2093
P2860
P50
P1476
Null mutations at the p66 and ...... otypes in the diabetic kidney.
@en
P2093
Ashwani Malhotra
Cyril Kuriakose
Francesca Peruzzi
Himanshu Vashistha
Hyung-Suk Kim
Krzysztof Reiss
Leonard G Meggs
Luis Delvalle
Marco Giorgio
P2860
P304
P356
10.1152/AJPRENAL.00246.2012
P577
2012-09-26T00:00:00Z