Activin A, a product of fetal Leydig cells, is a unique paracrine regulator of Sertoli cell proliferation and fetal testis cord expansion. - Wikidata - wikimedia - TriplyDB

Activin A, a product of fetal Leydig cells, is a unique paracrine regulator of Sertoli cell proliferation and fetal testis cord expansion.

Activin A, a product of fetal Leydig cells, is a unique paracrine regulator of Sertoli cell proliferation and fetal testis cord expansion.

http://www.wikidata.org/entity/Q33934865

about

How to make a gonad: cellular mechanisms governing formation of the testes and ovaries GATA4 regulates Sertoli cell function and fertility in adult male mice Signaling through the TGF beta-activin receptors ALK4/5/7 regulates testis formation and male germ cell development Overexpression of follistatin in the mouse epididymis disrupts fluid resorption and sperm transit in testicular excurrent ducts Loss of smad4 in Sertoli and Leydig cells leads to testicular dysgenesis and hemorrhagic tumor formation in mice Regulation of male sex determination: genital ridge formation and Sry activation in mice.A miR-590/Acvr2a/Rad51b axis regulates DNA damage repair during mESC proliferation hESC expansion and stemness are independent of connexin forty-three-mediated intercellular communication between hESCs and hASC feeder cells Adenomatous polyposis coli (APC) is essential for maintaining the integrity of the seminiferous epithelium.Testicular somatic cells, not gonocytes, are the major source of functional activin A during testis morphogenesis.Combined loss of the GATA4 and GATA6 transcription factors in male mice disrupts testicular development and confers adrenal-like function in the testes.GATA4 is a critical regulator of gonadectomy-induced adrenocortical tumorigenesis in mice.Sertoli Cell Wt1 Regulates Peritubular Myoid Cell and Fetal Leydig Cell Differentiation during Fetal Testis Development.Functional changes in mRNA expression and alternative pre-mRNA splicing associated with the effects of nutrition on apoptosis and spermatogenesis in the adult testis.Overexpression of PRL7D1 in Leydig Cells Causes Male Reproductive Dysfunction in Mice.Follistatin-like 3 (FSTL3) mediated silencing of transforming growth factor β (TGFβ) signaling is essential for testicular aging and regulating testis size Nodal/activin signaling promotes male germ cell fate and suppresses female programming in somatic cells.TGF-β superfamily: how does it regulate testis development.Smads and cell fate: distinct roles in specification, development, and tumorigenesis in the testis.Testis formation in the fetal mouse: dynamic and complex de novo tubulogenesis.Perspectives in pediatric pathology, chapter 1. Normal development of testicular structures: from the bipotential gonad to the fetal testis.Testis Cord Maintenance in Mouse Embryos: Genes and Signaling.Activins in reproductive biology and beyond.Development, function and fate of fetal Leydig cells.Activins and Inhibins: Roles in Development, Physiology, and Disease.Activin and follistatin interactions in the male reproductive tract: activin expression and morphological abnormalities in mice lacking follistatin 288.The TGF-β Family in the Reproductive Tract.Selective deletion of Smad4 in postnatal germ cells does not affect spermatogenesis or fertility in mice.Adult testicular dysgenesis of Inhba conditional knockout mice may also be caused by disruption of cross-talk between Leydig cells and germ cells.Regulation of development of rat stem and progenitor Leydig cells by activin.Effects of TGFbeta2 on wild-type and Tgfbr3 knockout mouse fetal testis.Receptors and signaling pathways involved in proliferation and differentiation of Sertoli cells.Reply to Sun et al.: Roles of adult Leydig cell-derived activin A remain to be determined.Roles of CD34+ cells and ALK5 signaling in the reconstruction of seminiferous tubule-like structures in 3-D re-aggregate culture of dissociated cells from neonatal mouse testes.Genetic ablation of androgen receptor signaling in fetal Leydig cell lineage affects Leydig cell functions in adult testis.Feasible etiology of vanishing testis regarding disturbance of testicular development: histopathological and immunohistochemical evaluation of testicular nubbins.

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P2860

Activin A, a product of fetal Leydig cells, is a unique paracrine regulator of Sertoli cell proliferation and fetal testis cord expansion.

http://www.wikidata.org/entity/Q33934865

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010年の論文

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2010年論文

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2010年論文

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2010年论文

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Proceedings of the National Academy of Sciences of the United States of America

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Denise R Archambeault

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Humphrey Hung-Chang Yao

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P2860

Betaglycan binds inhibin and can mediate functional antagonism of activin signalling

The question of declining sperm density revisited: an analysis of 101 studies published 1934-1996

Expression and localization of inhibin alpha, inhibin/activin betaA and betaB and the activin type II and inhibin beta-glycan receptors in the developing human testis

Requirement of Bmpr1a for Müllerian duct regression during male sexual development

Intraovarian activins are required for female fertility

Testicular dysgenesis syndrome: an increasingly common developmental disorder with environmental aspects

Morphometric analysis of testis cord formation in Sox9-EGFP mice.

Alpha-inhibin is a tumour-suppressor gene with gonadal specificity in mice.

Activins as regulators of branching morphogenesis.

In situ analysis of fetal, prepuberal and adult XX----XY chimaeric mouse testes: Sertoli cells are predominantly, but not exclusively, XY.

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10526-10531

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