Reprogramming transcription during the differentiation of precursor CD4+ T cells into effector Th1 and Th2 cells.
about
Isoprenoids determine Th1/Th2 fate in pathogenic T cells, providing a mechanism of modulation of autoimmunity by atorvastatin.Peroxisome proliferator-activated receptor (PPAR)alpha expression in T cells mediates gender differences in development of T cell-mediated autoimmunity.Neither T-helper type 2 nor Foxp3+ regulatory T cells are necessary for therapeutic benefit of atorvastatin in treatment of central nervous system autoimmunity.Weak TCR stimulation induces a calcium signal that triggers IL-4 synthesis, stronger TCR stimulation induces MAP kinases that control IFN-gamma production.Pitavastatin regulates helper T-cell differentiation and ameliorates autoimmune myocarditis in mice.
P2860
Reprogramming transcription during the differentiation of precursor CD4+ T cells into effector Th1 and Th2 cells.
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Reprogramming transcription du ...... to effector Th1 and Th2 cells.
@ast
Reprogramming transcription du ...... to effector Th1 and Th2 cells.
@en
type
label
Reprogramming transcription du ...... to effector Th1 and Th2 cells.
@ast
Reprogramming transcription du ...... to effector Th1 and Th2 cells.
@en
prefLabel
Reprogramming transcription du ...... to effector Th1 and Th2 cells.
@ast
Reprogramming transcription du ...... to effector Th1 and Th2 cells.
@en
P1476
Reprogramming transcription du ...... to effector Th1 and Th2 cells.
@en
P2093
P356
10.1016/S1286-4579(99)80013-6
P577
1999-01-01T00:00:00Z