Extinction training after cocaine self-administration induces glutamatergic plasticity to inhibit cocaine seeking.
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A prolyl-isomerase mediates dopamine-dependent plasticity and cocaine motor sensitizationModafinil blocks reinstatement of extinguished opiate-seeking in rats: mediation by a glutamate mechanismUsing glutamate homeostasis as a target for treating addictive disordersVirogenetic and optogenetic mechanisms to define potential therapeutic targets in psychiatric disordersRapid, transient synaptic plasticity in addictionRole of nucleus accumbens glutamatergic plasticity in drug addictionAnisomycin in the medial prefrontal cortex reduces reconsolidation of cocaine-associated memories in the rat self-administration modelPotential role of N-acetylcysteine in the management of substance use disordersExtinction-dependent alterations in corticostriatal mGluR2/3 and mGluR7 receptors following chronic methamphetamine self-administration in ratsThe metabotropic glutamate 5 receptor modulates extinction and reinstatement of methamphetamine-seeking in miceStress-induced sensitization to cocaine: actin cytoskeleton remodeling within mesocorticolimbic nucleiThe Plasticity of Extinction: Contribution of the Prefrontal Cortex in Treating Addiction through Inhibitory LearningImbalances in prefrontal cortex CC-Homer1 versus CC-Homer2 expression promote cocaine preference.NrCAM-regulating neural systems and addiction-related behaviors.Long-lasting impairment of mGluR5-activated intracellular pathways in the striatum after withdrawal of cocaine self-administrationSynaptic mechanisms underlying persistent cocaine cravingAKAP signaling in reinstated cocaine seeking revealed by iTRAQ proteomic analysisImaging glutamate homeostasis in cocaine addiction with the metabotropic glutamate receptor 5 positron emission tomography radiotracer [(11)C]ABP688 and magnetic resonance spectroscopyIntegrating synaptic plasticity and striatal circuit function in addiction.The Bermuda Triangle of cocaine-induced neuroadaptations.Pharmacotherapeutics directed at deficiencies associated with cocaine dependence: focus on dopamine, norepinephrine and glutamate.The neurobiology of modafinil as an enhancer of cognitive performance and a potential treatment for substance use disorders.Dorsal striatum mediation of cocaine-seeking after withdrawal from short or long daily access cocaine self-administration in rats.mGluR5 positive allosteric modulation enhances extinction learning following cocaine self-administration.Chronic N-acetylcysteine during abstinence or extinction after cocaine self-administration produces enduring reductions in drug seekingHypocretin 1/orexin A in the ventral tegmental area enhances dopamine responses to cocaine and promotes cocaine self-administration.Role of the ventral tegmental area in methamphetamine extinction: AMPA receptor-mediated neuroplasticity.Reduced LTP and LTD in prefrontal cortex synapses in the nucleus accumbens after heroin self-administrationThe central amygdala nucleus is critical for incubation of methamphetamine craving.Ceftriaxone prevents the induction of cocaine sensitization and produces enduring attenuation of cue- and cocaine-primed reinstatement of cocaine-seekingNew medications for drug addiction hiding in glutamatergic neuroplasticity.Measuring the incentive value of escalating doses of heroin in heroin-dependent Fischer rats during acute spontaneous withdrawal.Chronic modafinil effects on drug-seeking following methamphetamine self-administration in rats.Gene-based and pathway-based genome-wide association study of alcohol dependence.Integrins modulate relapse to cocaine-seeking.Ethanol and nicotine interaction within the posterior ventral tegmental area in male and female alcohol-preferring rats: evidence of synergy and differential gene activation in the nucleus accumbens shellRole of mGluR5 neurotransmission in reinstated cocaine-seekingHomer2 within the nucleus accumbens core bidirectionally regulates alcohol intake by both P and Wistar ratsCalcium-permeable AMPA receptors in the VTA and nucleus accumbens after cocaine exposure: when, how, and why?Current perspectives on the neurobiology of drug addiction: a focus on genetics and factors regulating gene expression.
P2860
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P2860
Extinction training after cocaine self-administration induces glutamatergic plasticity to inhibit cocaine seeking.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Extinction training after coca ...... ty to inhibit cocaine seeking.
@ast
Extinction training after coca ...... ty to inhibit cocaine seeking.
@en
type
label
Extinction training after coca ...... ty to inhibit cocaine seeking.
@ast
Extinction training after coca ...... ty to inhibit cocaine seeking.
@en
prefLabel
Extinction training after coca ...... ty to inhibit cocaine seeking.
@ast
Extinction training after coca ...... ty to inhibit cocaine seeking.
@en
P2093
P2860
P1476
Extinction training after coca ...... ty to inhibit cocaine seeking.
@en
P2093
Lori A Knackstedt
Marek Schwendt
Matthias Klugmann
Peter W Kalivas
Ryan Lalumiere
P2860
P304
P356
10.1523/JNEUROSCI.1244-10.2010
P407
P577
2010-06-01T00:00:00Z