Chronic N-acetylcysteine during abstinence or extinction after cocaine self-administration produces enduring reductions in drug seeking - Wikidata - wikimedia - TriplyDB

Chronic N-acetylcysteine during abstinence or extinction after cocaine self-administration produces enduring reductions in drug seeking

Chronic N-acetylcysteine during abstinence or extinction after cocaine self-administration produces enduring reductions in drug seeking

http://www.wikidata.org/entity/Q34868089

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Future pharmacological treatments for substance use disorders Oxidative stress and cerebral endothelial cells: regulation of the blood-brain-barrier and antioxidant based interventions Thinking outside the cleft to understand synaptic activity: contribution of the cystine-glutamate antiporter (System xc-) to normal and pathological glutamatergic signaling Astrocytic dysfunction and addiction: consequences of impaired glutamate homeostasis Rapid, transient synaptic plasticity in addiction Glutamate Transporter GLT-1 as a Therapeutic Target for Substance Use Disorders Potential role of N-acetylcysteine in the management of substance use disorders Behavioral History of Withdrawal Influences Regulation of Cocaine Seeking by Glutamate Re-Uptake Morphine induces redox-based changes in global DNA methylation and retrotransposon transcription by inhibition of excitatory amino acid transporter type 3-mediated cysteine uptake.N-acetylcysteine in the treatment of psychiatric disorders: current status and future prospects.Achieving cannabis cessation -- evaluating N-acetylcysteine treatment (ACCENT): design and implementation of a multi-site, randomized controlled study in the National Institute on Drug Abuse Clinical Trials Network.An open-label pilot trial of N-acetylcysteine and varenicline in adult cigarette smokers.Redox-based epigenetic status in drug addiction: a potential contributor to gene priming and a mechanistic rationale for metabolic intervention.A double-blind placebo-controlled trial of N-acetylcysteine in the treatment of cocaine dependence Ceftriaxone and cefazolin attenuate the cue-primed reinstatement of alcohol-seeking Ceftriaxone prevents the induction of cocaine sensitization and produces enduring attenuation of cue- and cocaine-primed reinstatement of cocaine-seeking Chronic modafinil effects on drug-seeking following methamphetamine self-administration in rats.Nicotine self-administration and reinstatement of nicotine-seeking in male and female rats.Glutathione and redox signaling in substance abuse.Effect of fenfluramine on reinstatement of food seeking in female and male rats: implications for the predictive validity of the reinstatement model.A double-blind randomized controlled trial of N-acetylcysteine in cannabis-dependent adolescents.The effects of N-Acetylcysteine on frontostriatal resting-state functional connectivity, withdrawal symptoms and smoking abstinence: A double-blind, placebo-controlled fMRI pilot study.Genetic variation in the alpha synuclein gene (SNCA) is associated with BOLD response to alcohol cues.Ceftriaxone attenuates cocaine relapse after abstinence through modulation of nucleus accumbens AMPA subunit expression.Enhanced extinction of cocaine seeking in brain-derived neurotrophic factor Val66Met knock-in mice S-Glutathionylation and Redox Protein Signaling in Drug Addiction.N-acetylcysteine decreased nicotine self-administration and cue-induced reinstatement of nicotine seeking in rats: comparison with the effects of N-acetylcysteine on food responding and food seeking The Nucleus Accumbens: Mechanisms of Addiction across Drug Classes Reflect the Importance of Glutamate Homeostasis The reinstatement model of drug relapse: recent neurobiological findings, emerging research topics, and translational research.Dysregulation of dopamine and glutamate release in the prefrontal cortex and nucleus accumbens following methamphetamine self-administration and during reinstatement in rats.Acquisition, Maintenance and Relapse-Like Alcohol Drinking: Lessons from the UChB Rat Line.Emerging roles of system [Formula: see text] antiporter and its inhibition in CNS disorders.Recent developments in the behavioural and pharmacological enhancement of extinction of drug seeking.Glutamate Transport: A New Bench to Bedside Mechanism for Treating Drug Abuse Chronic N-acetylcysteine after cocaine self-administration produces enduring reductions in drug-seeking.Toward early estimation and treatment of addiction vulnerability: radial arm maze and N-acetyl cysteine before cocaine sensitization or nicotine self-administration in neonatal ventral hippocampal lesion rats.Glial and neuroinflammatory targets for treating substance use disorders.Effect of N-acetylcysteine on motivation, seeking and relapse to ethanol self-administration.N-acetylcysteine amide (AD4) reduces cocaine-induced reinstatement.N-Acetylcysteine reduces cocaine-cue attentional bias and differentially alters cocaine self-administration based on dosing order.

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P2860

Chronic N-acetylcysteine during abstinence or extinction after cocaine self-administration produces enduring reductions in drug seeking

http://www.wikidata.org/entity/Q34868089

description

2011 nî lūn-bûn

@nan

2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Chronic N-acetylcysteine durin ...... ing reductions in drug seeking

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Chronic N-acetylcysteine durin ...... ing reductions in drug seeking

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Chronic N-acetylcysteine durin ...... ing reductions in drug seeking

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Chronic N-acetylcysteine durin ...... ing reductions in drug seeking

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Chronic N-acetylcysteine durin ...... ing reductions in drug seeking

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Chronic N-acetylcysteine durin ...... ing reductions in drug seeking

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Chronic N-acetylcysteine durin ...... ing reductions in drug seeking

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Journal of Pharmacology and Experimental Therapeutics

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Chronic N-acetylcysteine durin ...... ing reductions in drug seeking

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Peter W Kalivas

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Phong H Do

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Ronald E See

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P2860

Fos protein expression and cocaine-seeking behavior in rats after exposure to a cocaine self-administration environment

Forced Abstinence Model of Relapse to Study Pharmacological Treatments of Substance Use Disorder

Neural mechanisms of extinction learning and retrieval

The reinstatement model of drug relapse: history, methodology and major findings

Reversing cocaine-induced synaptic potentiation provides enduring protection from relapse.

Metabotropic glutamate receptor 5 (mGluR5) antagonists attenuate cocaine priming- and cue-induced reinstatement of cocaine seeking

Anti-relapse medications: preclinical models for drug addiction treatment.

Extinction training after cocaine self-administration induces glutamatergic plasticity to inhibit cocaine seeking.

The origin and neuronal function of in vivo nonsynaptic glutamate.

Context, ambiguity, and unlearning: sources of relapse after behavioral extinction.

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Khaled Moussawi

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