Estrogen receptor silencing induces epithelial to mesenchymal transition in human breast cancer cells. - Wikidata - wikimedia - TriplyDB

Estrogen receptor silencing induces epithelial to mesenchymal transition in human breast cancer cells.

Estrogen receptor silencing induces epithelial to mesenchymal transition in human breast cancer cells.

http://www.wikidata.org/entity/Q33945493

about

Long chain fatty Acyl-CoA synthetase 4 is a biomarker for and mediator of hormone resistance in human breast cancer EMT in Breast Carcinoma-A Review Minimal residual disease in breast cancer: an overview of circulating and disseminated tumour cells Epithelial-Mesenchymal Transition (EMT) and Regulation of EMT Factors by Steroid Nuclear Receptors in Breast Cancer: A Review and in Silico Investigation The network of pluripotency, epithelial-mesenchymal transition, and prognosis of breast cancer Extracellular alkaline pH leads to increased metastatic potential of estrogen receptor silenced endocrine resistant breast cancer cells The impact of low-dose carcinogens and environmental disruptors on tissue invasion and metastasis Differential effect of growth factors on invasion and proliferation of endocrine resistant breast cancer cells Condition-specific target prediction from motifs and expression.Estradiol promotes pentose phosphate pathway addiction and cell survival via reactivation of Akt in mTORC1 hyperactive cells.Assessment of tracer 99mTc(V)-DMSA uptake as a measure of tumor cell proliferation in vitro.Improving the design of the agarose spot assay for eukaryotic cell chemotaxis.Clinical instability of breast cancer markers is reflected in long-term in vitro estrogen deprivation studies Feed-forward transcriptional programming by nuclear receptors: regulatory principles and therapeutic implications Vangl1 and Vangl2: planar cell polarity components with a developing role in cancer.PAC down-regulates estrogen receptor alpha and suppresses epithelial-to-mesenchymal transition in breast cancer cells.Proteomic characterization of microdissected breast tissue environment provides a protein-level overview of malignant transformation Blockade of voltage-gated sodium channels inhibits invasion of endocrine-resistant breast cancer cells Characterization of mesenchymal cells beneath cornification of the fetal epithelium and epidermis at the face: an immunohistochemical study using human fetal specimens Annexin-A1 and caldesmon are associated with resistance to tamoxifen in estrogen receptor positive recurrent breast cancer.Luminal long non-coding RNAs regulated by estrogen receptor alpha in a ligand-independent manner show functional roles in breast cancer Multi-organ Site Metastatic Reactivation Mediated by Non-canonical Discoidin Domain Receptor 1 Signaling.Clinical implications for loss or diminution of expression of Raf-1 kinase inhibitory protein and its phosphorylated form in ductal breast cancer Genome-wide activity of unliganded estrogen receptor-α in breast cancer cells.Endometriosis risk alleles at 1p36.12 act through inverse regulation of CDC42 and LINC00339.Gene Expression Meta-Analysis of Potential Metastatic Breast Cancer Markers.Resistance to Targeted Therapies in Breast Cancer.Fatty acid metabolism in breast cancer subtypes.Chronic oxidative stress causes estrogen-independent aggressive phenotype, and epigenetic inactivation of estrogen receptor alpha in MCF-7 breast cancer cells.Increased tumor cell expression of Axl is a marker of aggressive features in breast cancer among African women.The GRHL2/ZEB Feedback Loop-A Key Axis in the Regulation of EMT in Breast Cancer.Electrospun fibrous scaffolds promote breast cancer cell alignment and epithelial-mesenchymal transition.Bone morphogenetic proteins, breast cancer, and bone metastases: striking the right balance.Molecular characterization of circulating tumor cells from patients with metastatic breast cancer reflects evolutionary changes in gene expression under the pressure of systemic therapy.Statistically controlled identification of differentially expressed genes in one-to-one cell line comparisons of the CMAP database for drug repositioning.Vimentin, zeb1 and Sip1 are up-regulated in triple-negative and basal-like breast cancers: association with an aggressive tumour phenotype.Overcoming Resistance to Endocrine Therapy in Breast Cancer: New Approaches to a Nagging Problem.Cellular inhibitor of apoptosis protein 2 promotes the epithelial-mesenchymal transition in triple-negative breast cancer cells through activation of the AKT signaling pathway.Suppression of cancer stemness by upregulating Ligand-of-Numb protein X1 in colorectal carcinoma.Notch3 Maintains Luminal Phenotype and Suppresses Tumorigenesis and Metastasis of Breast Cancer via Trans-Activating Estrogen Receptor-α.

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P2860

Estrogen receptor silencing induces epithelial to mesenchymal transition in human breast cancer cells.

http://www.wikidata.org/entity/Q33945493

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2011 nî lūn-bûn

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GATA3 inhibits breast cancer metastasis through the reversal of epithelial-mesenchymal transition

Regulation of ERBB2 by oestrogen receptor-PAX2 determines response to tamoxifen

MicroRNA-221/222 confers tamoxifen resistance in breast cancer by targeting p27Kip1

ProSplicer: a database of putative alternative splicing information derived from protein, mRNA and expressed sequence tag sequence data

The E-cadherin/catenin complex: an important gatekeeper in breast cancer tumorigenesis and malignant progression.

FGFR1 amplification drives endocrine therapy resistance and is a therapeutic target in breast cancer

A new mathematical model for relative quantification in real-time RT-PCR

Twist, a master regulator of morphogenesis, plays an essential role in tumor metastasis

MTA3, a Mi-2/NuRD complex subunit, regulates an invasive growth pathway in breast cancer

Transitions between epithelial and mesenchymal states: acquisition of malignant and stem cell traits

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